Blocking H2A.Z Incorporation via Tip60 Inhibition Promotes Systems Consolidation of Fear Memory in Mice

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Blocking H2A.Z Incorporation via Tip60 Inhibition Promotes Systems Consolidation of Fear Memory in Mice

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scientific article published on 01 September 2018

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wetenschappelijk artikel

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Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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P1476

Blocking H2A.Z Incorporation v ...... idation of Fear Memory in Mice

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Amber B Azam

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Firyal Ramzan

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Klotilda Narkaj

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Histone methylation regulates memory formation.

Histone H2A.Z subunit exchange controls consolidation of recent and remote memory.

Molecular basis and specificity of H2A.Z-H2B recognition and deposition by the histone chaperone YL1

NuA4-dependent acetylation of nucleosomal histones H4 and H2A directly stimulates incorporation of H2A.Z by the SWR1 complex.

CBP histone acetyltransferase activity is a critical component of memory consolidation

Persistence of long-term memory storage requires a late protein synthesis- and BDNF- dependent phase in the hippocampus

Histone acetylation by Trrap-Tip60 modulates loading of repair proteins and repair of DNA double-strand breaks

G9a/GLP histone lysine dimethyltransferase complex activity in the hippocampus and the entorhinal cortex is required for gene activation and silencing during memory consolidation

Maintenance of long-term memory storage is dependent on late posttraining Egr-1 expression

The organization of recent and remote memories

P304

ENEURO.0378-18.2018

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scholarly article

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10.1523/ENEURO.0378-18.2018

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English

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Carl Frank David Steininger

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2018-09-01T00:00:00Z

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30417078

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6223110

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