Constitutively active SMAD2/3 facilitates efficient transcription factor-mediated cell conversion

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Constitutively active SMAD2/3 facilitates efficient transcription factor-mediated cell conversion

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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P1476

Constitutively active SMAD2/3 ...... actor-mediated cell conversion

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P2093

Andriana Margariti

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Sophia Kelaini

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Small molecules enable cardiac reprogramming of mouse fibroblasts with a single factor, Oct4.

Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

TGFβ signalling in context

Tgfbeta signal inhibition cooperates in the induction of iPSCs and replaces Sox2 and cMyc

C/EBPα creates elite cells for iPSC reprogramming by upregulating Klf4 and increasing the levels of Lsd1 and Brd4.

A mesenchymal-to-epithelial transition initiates and is required for the nuclear reprogramming of mouse fibroblasts.

The tumor suppressor Smad4/DPC4 and transcriptional adaptor CBP/p300 are coactivators for smad3 in TGF-beta-induced transcriptional activation.

MAPK/ERK signalling mediates VEGF-induced bone marrow stem cell differentiation into endothelial cell.

Genome-wide impact of the BRG1 SWI/SNF chromatin remodeler on the transforming growth factor beta transcriptional program

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scholarly article

editorial

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10.21037/SCI.2018.08.03

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English

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2018-01-01T00:00:00Z

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30221170

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6131529

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