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Crawling with Virus: Translational Insights from a Neonatal Mouse Model on the Pathogenesis of Respiratory Syncytial Virus in InfantsRadical-containing ultrafine particulate matter initiates epithelial-to-mesenchymal transitions in airway epithelial cellsLimited type I interferons and plasmacytoid dendritic cells during neonatal respiratory syncytial virus infection permit immunopathogenesis upon reinfection.Deficiency of the two-pore-domain potassium channel TREK-1 promotes hyperoxia-induced lung injury.Exposure to combustion generated environmentally persistent free radicals enhances severity of influenza virus infectionEnvironmentally persistent free radicals induce airway hyperresponsiveness in neonatal rat lungs.Impaired gamma delta T cell-derived IL-17A and inflammasome activation during early respiratory syncytial virus infection in infantsSerotonin 5-HT₂ receptor activation prevents allergic asthma in a mouse modelRespiratory Syncytial Virus Disease Is Mediated by Age-Variable IL-33.Exposure to Deepwater Horizon Crude Oil Burnoff Particulate Matter Induces Pulmonary Inflammation and Alters Adaptive Immune ResponseBuilding a better neonatal mouse model to understand infant respiratory syncytial virus disease.Regulation of inflammatory biomarkers by intravenous methylprednisolone in pediatric ARDS patients: Results from a double-blind, placebo-controlled randomized pilot trial.IL-4Rα on CD4+ T cells plays a pathogenic role in respiratory syncytial virus reinfection in mice infected initially as neonatesMaternal exposure to combustion generated PM inhibits pulmonary Th1 maturation and concomitantly enhances postnatal asthma development in offspring.Exposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice.Chronic alcohol induces M2 polarization enhancing pulmonary disease caused by exposure to particulate air pollutionGfi1-Foxo1 axis controls the fidelity of effector gene expression and developmental maturation of thymocytes.Regulatory T cells and IL10 suppress pulmonary host defense during early-life exposure to radical containing combustion derived ultrafine particulate matter.IL-4Rα on dendritic cells in neonates and Th2 immunopathology in respiratory syncytial virus infection.Early-life exposure to combustion-derived particulate matter causes pulmonary immunosuppression.Type I Interferon Potentiates IgA Immunity to Respiratory Syncytial Virus Infection During InfancyNew mouse model of pulmonary hypertension induced by respiratory syncytial virus bronchiolitisSignaling networks in immunometabolismHelper T cell differentiationHomeostasis and transitional activation of regulatory T cells require c-Myc
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-2691-501X
@en
name
Jordy Saravia
@ast
Jordy Saravia
@en
Jordy Saravia
@es
Jordy Saravia
@nl
type
label
Jordy Saravia
@ast
Jordy Saravia
@en
Jordy Saravia
@es
Jordy Saravia
@nl
prefLabel
Jordy Saravia
@ast
Jordy Saravia
@en
Jordy Saravia
@es
Jordy Saravia
@nl
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P1153
37036166500
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0000-0002-2691-501X