about
Syk-mediated translocation of PI3Kdelta to the leading edge controls lamellipodium formation and migration of leukocytesLymphatic function is required prenatally for lung inflation at birth.Critical but overlapping role of FcgammaRIII and FcgammaRIV in activation of murine neutrophils by immobilized immune complexesKinase pathways in chemoattractant-induced degranulation of neutrophils: the role of p38 mitogen-activated protein kinase activated by Src family kinasesThe Src family kinases Hck, Fgr, and Lyn are critical for the generation of the in vivo inflammatory environment without a direct role in leukocyte recruitmentGenetic deficiency of Syk protects mice from autoantibody-induced arthritis.Phospholipase Cγ2 is required for basal but not oestrogen deficiency-induced bone resorption.The cerebral cavernous malformation pathway controls cardiac development via regulation of endocardial MEKK3 signaling and KLF expression.ITAM-based signaling beyond the adaptive immune response.Podoplanin-rich stromal networks induce dendritic cell motility via activation of the C-type lectin receptor CLEC-2.Immunoreceptor-like signaling by beta 2 and beta 3 integrins.Critical role of phospholipase Cgamma2 in integrin and Fc receptor-mediated neutrophil functions and the effector phase of autoimmune arthritis.Phosphoinositide 3-OH kinase regulates integrin-dependent processes in neutrophils by signaling through its effector ARAP3.Platelets mediate lymphovenous hemostasis to maintain blood-lymphatic separation throughout life.G-protein-coupled receptor signaling in Syk-deficient neutrophils and mast cells.Fine-tuning of integrin activation.Importance of Fc Receptor γ-Chain ITAM Tyrosines in Neutrophil Activation and Autoimmune ArthritisHematopoietic or Osteoclast-Specific Deletion of Syk Leads to Increased Bone Mass in Experimental MiceReduced Prenatal Pulmonary Lymphatic Function Is Observed in Clp1 K/K Embryos With Impaired Motor Functions Including Fetal Breathing Movements in Preparation of the Developing Lung for Inflation at BirthFoliate Lymphoid Aggregates as Novel Forms of Serous Lymphocyte Entry Sites of Peritoneal B Cells and High-Grade B Cell LymphomasLymphatic mimicry in maternal endothelial cells promotes placental spiral artery remodelingLymphatic impairment leads to pulmonary tertiary lymphoid organ formation and alveolar damageLymph Flow Induces the Postnatal Formation of Mature and Functional Meningeal Lymphatic Vessels
P50
Q27302372-5F1AF7C9-090B-4EA7-B504-A58CDF50875DQ28505708-60030193-1287-4F45-B518-5CB063AC3D5EQ28594601-B3789D94-D51B-4E20-897E-39905769880BQ28646115-426529FD-0B78-4039-B970-A3157FD02771Q34230548-27590429-F678-44F1-B474-CAA575BCBB0FQ34275410-2555A2D6-E9F3-4DD6-8F7A-6C93D6893F8FQ35697027-7E2B30FC-BE9C-4759-8D79-D9A94F115D09Q36109992-6462A954-B5AB-423A-8FE0-E13AD4FBB37EQ36332906-A301D81B-1C06-4E7C-A432-0E407EBC566AQ36567149-54278F09-E753-407D-B3B5-97255BEA53B3Q36959336-70957AFC-A335-4728-8BA8-22473CADF6EFQ37234322-57239F1A-5A69-4C19-90E2-36682E7455B6Q41545564-E2C8834A-AD93-48F9-BFD1-6FC4548DDCA9Q42779021-D5A3E140-BEA3-48B9-AB0B-37B6886E7647Q44280924-0157A7FF-2CBD-4058-A644-C18A184CF4E8Q48256958-48F6F347-6093-4BAE-85B1-E5A588B0B9E5Q64104748-73F72808-5DC9-468C-9FBF-66BED553B32AQ64264445-5E3480FB-4D1A-4BE5-A30B-52778EAE2FCCQ90643210-FA6CBB73-7FC1-4945-922D-D55E9E659C2FQ91451165-2851A500-0B34-46B0-B471-7A30846A926EQ92636819-BEFA7D59-88A6-4A05-8A0A-528BFECCA3F3Q92843566-2ECA0A02-818F-4EB1-B13E-69C3799BCEE2Q93014186-74E3AEEB-9453-43ED-A173-34A199B551FB
P50
description
researcher ORCID ID = 0000-0002-6304-2369
@en
wetenschapper
@nl
name
Zoltán Jakus
@ast
Zoltán Jakus
@en
Zoltán Jakus
@es
Zoltán Jakus
@nl
type
label
Zoltán Jakus
@ast
Zoltán Jakus
@en
Zoltán Jakus
@es
Zoltán Jakus
@nl
prefLabel
Zoltán Jakus
@ast
Zoltán Jakus
@en
Zoltán Jakus
@es
Zoltán Jakus
@nl
P106
P1153
6507504129
P31
P496
0000-0002-6304-2369