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Concepts of papillomavirus entry into host cellsHuman papillomavirus type 16 entry: retrograde cell surface transport along actin-rich protrusionsLarge scale RNAi reveals the requirement of nuclear envelope breakdown for nuclear import of human papillomavirusesEntry of human papillomavirus type 16 by actin-dependent, clathrin- and lipid raft-independent endocytosisViral Genome Tethering to Host Cell Chromatin: Cause and ConsequencesVirus entry by endocytosisA central region in the minor capsid protein of papillomaviruses facilitates viral genome tethering and membrane penetration for mitotic nuclear entry.Single-cell analysis of population context advances RNAi screening at multiple levels.Kallikrein-8 Proteolytically Processes Human Papillomaviruses in the Extracellular Space To Facilitate Entry into Host CellsHeparin increases the infectivity of Human Papillomavirus type 16 independent of cell surface proteoglycans and induces L1 epitope exposureThe evolving field of human papillomavirus receptor research: a review of binding and entry.Human papillomavirus types 16, 18, and 31 share similar endocytic requirements for entry.Come in and take your coat off - how host cells provide endocytosis for virus entry.Analysis of virus entry and cellular membrane dynamics by single particle tracking.Principles of polyoma- and papillomavirus uncoating.Systematic analysis of endocytosis by cellular perturbations.Early herpes simplex virus type 1 infection is dependent on regulated Rac1/Cdc42 signalling in epithelial MDCKII cells.Fluorescently Labeled Human Papillomavirus Pseudovirions for Use in Virus Entry Experiments.Simian Virus 40 depends on ER protein folding and quality control factors for entry into host cells.Herpes simplex virus type 1 exhibits a tropism for basal entry in polarized epithelial cells.TATA-binding protein and TBP-associated factors during herpes simplex virus type 1 infection: localization at viral DNA replication sites.Viruses and cancer: molecular relations and perspectives.Extracellular conformational changes in the capsid of human papillomaviruses contribute to asynchronous uptake into host cells.Glycans Controlling Virus Infections: Meeting Report on the 1st International Symposium on Glycovirology Schöntal, Germany, 02⁻04 May 2018Actin dynamics in host-pathogen interactionProphylactic Antiviral Activity of Sulfated Glycomimetic Oligomers and PolymersInterview-Mario Schelhaas
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description
researcher ORCID ID = 0000-0002-6735-583X
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wetenschapper
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name
Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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Mario Schelhaas
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0000-0002-6735-583X