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Id1, Id2 and Id3 are induced in rat melanotrophs of the pituitary gland by dopamine suppression under continuous stressSimultaneous expression of glutathione, thioredoxin-1, and their reductases in nerve transected hypoglossal motor neurons of ratPancreatitis-associated protein-I and pancreatitis-associated protein-III expression in a rat model of kainic acid-induced seizureUrinary levels of hepatocarcinoma-intestine-pancreas/pancreatitis-associated protein as a diagnostic biomarker in patients with bladder cancer.R-spondin 2 promotes acetylcholine receptor clustering at the neuromuscular junction via Lgr5.N-terminal cleaved pancreatitis-associated protein-III (PAP-III) serves as a scaffold for neurites and promotes neurite outgrowth.Siglec-H is a microglia-specific marker that discriminates microglia from CNS-associated macrophages and CNS-infiltrating monocytes.Pancreatitis-associated protein-III is a novel macrophage chemoattractant implicated in nerve regeneration.Phenylbutazone induces expression of MBNL1 and suppresses formation of MBNL1-CUG RNA foci in a mouse model of myotonic dystrophy.Increase of transcription factor EB (TFEB) and lysosomes in rat DRG neurons and their transportation to the central nerve terminal in dorsal horn after nerve injury.Agonists for G-protein-coupled receptor 84 (GPR84) alter cellular morphology and motility but do not induce pro-inflammatory responses in microglia.Altered expression of neprilysin family members in the pituitary gland of sleep-disturbed rats, an animal model of severe fatigue.Ontogeny and innervation of taste buds in mouse palatal gustatory epithelium.Continuous stress promotes expression of VGF in melanotroph via suppression of dopamine.Chronic stress elicits prolonged activation of alpha-MSH secretion and subsequent degeneration of melanotroph.The formation of argpyrimidine, a methylglyoxal-arginine adduct, in the nucleus of neural cells.The p53-independent nuclear translocation of cyclin G1 in degenerating neurons by ischemic and traumatic insults.Possible ATP release through lysosomal exocytosis from primary sensory neurons.Phospholipid localization implies microglial morphology and function via Cdc42 in vitro.TREM2/DAP12 Signal Elicits Proinflammatory Response in Microglia and Exacerbates Neuropathic Pain.Lack of Fgf18 causes abnormal clustering of motor nerve terminals at the neuromuscular junction with reduced acetylcholine receptor clusters.The absence of somatotroph proliferation during continuous stress is a result of the lack of extracellular signal-regulated kinase 1/2 activation.Induction of pancreatitis-associated protein (PAP) family members in neurons after traumatic brain injury.G-protein-coupled receptor screen reveals a role for chemokine receptor CCR5 in suppressing microglial neurotoxicity.Exposure to diphtheria toxin during the juvenile period impairs both inner and outer hair cells in C57BL/6 mice.Expression analysis of the regenerating gene (Reg) family members Reg-IIIβ and Reg-IIIγ in the mouse during development.A DAP12-dependent signal promotes pro-inflammatory polarization in microglia following nerve injury and exacerbates degeneration of injured neurons.Dual functions of microglia in the formation and refinement of neural circuits during developmentMicroglial TREM2/DAP12 Signaling: A Double-Edged Sword in Neural DiseasesContinuous stress-induced dopamine dysregulation augments PAP-I and PAP-II expression in melanotrophs of the pituitary glandElevated Urinary Levels and Urothelial Expression of Hepatocarcinoma-intestine-pancreas/Pancreatitis-associated Protein in Patients With Interstitial CystitisLocal ventilation system successfully reduced formaldehyde exposure during gross anatomy dissection classesIdentification of Peripherin as a Akt Substrate in NeuronsAnnexin III implicated in the microglial response to motor nerve injuryGPR34 in spinal microglia exacerbates neuropathic pain in miceBranched Sialylated N-glycans Are Accumulated in Brain Synaptosomes and Interact with Siglec-HDual microglia effects on blood brain barrier permeability induced by systemic inflammationNecroptosis of Intestinal Epithelial Cells Induces Type 3 Innate Lymphoid Cell-Dependent Lethal Ileitis
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description
researcher ORCID ID = 0000-0002-4321-8339
@en
name
Hiroyuki Konishi
@ast
Hiroyuki Konishi
@en
Hiroyuki Konishi
@es
Hiroyuki Konishi
@nl
type
label
Hiroyuki Konishi
@ast
Hiroyuki Konishi
@en
Hiroyuki Konishi
@es
Hiroyuki Konishi
@nl
prefLabel
Hiroyuki Konishi
@ast
Hiroyuki Konishi
@en
Hiroyuki Konishi
@es
Hiroyuki Konishi
@nl
P31
P496
0000-0002-4321-8339