sameAs
BDNF is essential to promote persistence of long-term memory storageDifferent molecular cascades in different sites of the brain control memory consolidationAltered behavioural response to acute stress in mice lacking cellular prion proteinThe interaction between prion protein and laminin modulates memory consolidationBDNF-triggered events in the rat hippocampus are required for both short- and long-term memory formationBDNF activates mTOR to regulate GluR1 expression required for memory formationThe vesicular acetylcholine transporter is required for neuromuscular development and functionPharmacological findings on the biochemical bases of memory processes: a general view.Plastic modifications induced by object recognition memory processingElimination of the vesicular acetylcholine transporter in the forebrain causes hyperactivity and deficits in spatial memory and long-term potentiationMemory reconsolidation and its maintenance depend on L-voltage-dependent calcium channels and CaMKII functions regulating protein turnover in the hippocampus.Posterior parietal cortex and long-term memory: some data from laboratory animals.Retrieval induces reconsolidation of fear extinction memory.Cellular prion protein: on the road for functions.Extinction learning, which consists of the inhibition of retrieval, can be learned without retrievalParticipation of CaMKII in neuronal plasticity and memory formation.Molecular pharmacological dissection of short- and long-term memory.Molecular mechanisms of memory retrieval.Topiramate diminishes fear memory consolidation and extinguishes conditioned fear in ratsPharmacological findings contribute to the understanding of the main physiological mechanisms of memory retrieval.Facilitation of fear extinction by novelty depends on dopamine acting on D1-subtype dopamine receptors in hippocampus.Histamine in the basolateral amygdala promotes inhibitory avoidance learning independently of hippocampus.Role of the hippocampus and amygdala in the extinction of fear-motivated learning.Behavioral tagging of extinction learningA link between the hippocampal and the striatal memory systems of the brain.The connection between the hippocampal and the striatal memory systems of the brain: a review of recent findings.Differential effects of acute diazepam on emotional and neutral memory tasks in acutely hospitalized depressed patients.The role of the entorhinal cortex in extinction: influences of aging.Parallel memory processing by the CA1 region of the dorsal hippocampus and the basolateral amygdala.Memory retrieval of inhibitory avoidance requires histamine H1 receptor activation in the hippocampusBDNF and memory formation and storage.Memory-enhancing treatments reverse the impairment of inhibitory avoidance retention in sepsis-surviving rats.Age-dependent and age-independent human memory persistence is enhanced by delayed posttraining methylphenidate administrationERK1/2 and CaMKII-mediated events in memory formation: is 5HT regulation involved?Major neurotransmitter systems in dorsal hippocampus and basolateral amygdala control social recognition memory.The molecular cascades of long-term potentiation underlie memory consolidation of one-trial avoidance in the CA1 region of the dorsal hippocampus, but not in the basolateral amygdala or the neocortex.Reconsolidation and the fate of consolidated memories.Hippocampal molecular mechanisms involved in the enhancement of fear extinction caused by exposure to novelty.Persistence of long-term memory storage: new insights into its molecular signatures in the hippocampus and related structures.Treatment of fear memories: interactions between extinction and reconsolidation.
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Argentijns neurowetenschapper
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