about
RNA destabilization by the granulocyte colony-stimulating factor stem-loop destabilizing element involves a single stem-loop that promotes deadenylation.Experimental strategies for microRNA target identificationThe miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1Significance of PELP1/HDAC2/miR-200 regulatory network in EMT and metastasis of breast cancerA double-negative feedback loop between ZEB1-SIP1 and the microRNA-200 family regulates epithelial-mesenchymal transitionInferring microRNA and transcription factor regulatory networks in heterogeneous data.Inferring condition-specific miRNA activity from matched miRNA and mRNA expression data.Identifying direct miRNA-mRNA causal regulatory relationships in heterogeneous data.Exploring complex miRNA-mRNA interactions with Bayesian networks by splitting-averaging strategy.Specificity protein 1 (Sp1) maintains basal epithelial expression of the miR-200 family: implications for epithelial-mesenchymal transitionRegulated post-transcriptional RNA cleavage diversifies the eukaryotic transcriptome.A novel role for the Pol I transcription factor UBTF in maintaining genome stability through the regulation of highly transcribed Pol II genes.Chromatinized protein kinase C-θ directly regulates inducible genes in epithelial to mesenchymal transition and breast cancer stem cellsGlobal analysis of the mammalian RNA degradome reveals widespread miRNA-dependent and miRNA-independent endonucleolytic cleavage.Mutant p53 drives invasion in breast tumors through up-regulation of miR-155.A multi-protein complex containing cold shock domain (Y-box) and polypyrimidine tract binding proteins forms on the vascular endothelial growth factor mRNA. Potential role in mRNA stabilization.Genome-wide identification of miR-200 targets reveals a regulatory network controlling cell invasionIdentifying functional miRNA-mRNA regulatory modules with correspondence latent dirichlet allocation.Induction of miR-21 by retinoic acid in estrogen receptor-positive breast carcinoma cells: biological correlates and molecular targets.Inferring microRNA-mRNA causal regulatory relationships from expression data.Discovering functional microRNA-mRNA regulatory modules in heterogeneous data.Reversal and prevention of arsenic-induced human bronchial epithelial cell malignant transformation by microRNA-200b.Assessing the gene regulatory properties of Argonaute-bound small RNAs of diverse genomic origin.Identification of an enhancer that increases miR-200b~200a~429 gene expression in breast cancer cells.p53 Represses the Oncogenic Sno-MiR-28 Derived from a SnoRNAIntegration of microRNA signatures of distinct mammary epithelial cell types with their gene expression and epigenetic portraitsA cytokine mRNA-destabilizing element that is structurally and functionally distinct from A+U-rich elements.MicroRNA networks regulated by all-trans retinoic acid and Lapatinib control the growth, survival and motility of breast cancer cellsDown-regulation of the miRNA-200 family at the invasive front of colorectal cancers with degraded basement membrane indicates EMT is involved in cancer progression.The receptor for interleukin 3 is selectively induced in human endothelial cells by tumor necrosis factor alpha and potentiates interleukin 8 secretion and neutrophil transmigrationHoxb8 regulates expression of microRNAs to control cell death and differentiation.MicroRNAs as regulators of epithelial-mesenchymal transition.The role of microRNAs in metastasis and epithelial-mesenchymal transition.microRNAs and EMT in mammary cells and breast cancer.IsomiRs--the overlooked repertoire in the dynamic microRNAome.Understanding principles of miRNA target recognition and function through integrated biological and bioinformatics approaches.Direct transcriptional regulation by nuclear microRNAs.Network-Based Approaches to Understand the Roles of miR-200 and Other microRNAs in Cancer.MicroRNA-194 Promotes Prostate Cancer Metastasis by Inhibiting SOCS2.A ZEB1-miR-375-YAP1 pathway regulates epithelial plasticity in prostate cancer.
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description
researcher ORCID ID = 0000-0003-1294-0692
@en
wetenschapper
@nl
name
Gregory J Goodall
@ast
Gregory J Goodall
@en
Gregory J Goodall
@nl
type
label
Gregory J Goodall
@ast
Gregory J Goodall
@en
Gregory J Goodall
@nl
altLabel
Gregory Goodall
@en
prefLabel
Gregory J Goodall
@ast
Gregory J Goodall
@en
Gregory J Goodall
@nl
P21
P2456
P31
P496
0000-0003-1294-0692