about
FOXO3a-dependent regulation of Puma in response to cytokine/growth factor withdrawalBH3-only Bcl-2 family member Bim is required for apoptosis of autoreactive thymocytesHobit and Blimp1 instruct a universal transcriptional program of tissue residency in lymphocytesARIH2 is essential for embryogenesis, and its hematopoietic deficiency causes lethal activation of the immune systemChemical chaperone TUDCA prevents apoptosis and improves survival during polymicrobial sepsis in micePlatelet production proceeds independently of the intrinsic and extrinsic apoptosis pathways.Shutdown of an acute T cell immune response to viral infection is mediated by the proapoptotic Bcl-2 homology 3-only protein Bim.Fatal hepatitis mediated by tumor necrosis factor TNFalpha requires caspase-8 and involves the BH3-only proteins Bid and Bim.Cellular inhibitor of apoptosis proteins prevent clearance of hepatitis B virus.NLRP1 inflammasome activation induces pyroptosis of hematopoietic progenitor cells.TB incidence and characteristics in the remote gulf province of Papua New Guinea: a prospective studyIL-6 promotes acute and chronic inflammatory disease in the absence of SOCS3.Prosurvival Bcl-2 family members reveal a distinct apoptotic identity between conventional and plasmacytoid dendritic cellsEliminating hepatitis B by antagonizing cellular inhibitors of apoptosis.Loss of the signaling adaptor TRAF1 causes CD8+ T cell dysregulation during human and murine chronic infectionAnalysis of the apoptotic and therapeutic activities of histone deacetylase inhibitors by using a mouse model of B cell lymphomaT-lymphocyte death during shutdown of an immune response.TNF-alpha is critical for antitumor but not antiviral T cell immunity in mice.CRISPR/Cas9-The ultimate weapon to battle infectious diseases?Apaf-1 and caspase-9 do not act as tumor suppressors in myc-induced lymphomagenesis or mouse embryo fibroblast transformation.Loss of Bim increases T cell production and function in interleukin 7 receptor-deficient miceCellular FLICE-inhibitory protein is required for T cell survival and cyclingCD4 T cells, lymphopenia, and IL-7 in a multistep pathway to autoimmunity.NFκB1 is essential to prevent the development of multiorgan autoimmunity by limiting IL-6 production in follicular B cells.Tumor immune therapy: lessons from infection and implications for cancer - can IL-7 help overcome immune inhibitory networks?Fighting cancers from within: augmenting tumor immunity with cytokine therapy.Changing treatment paradigms for patients with plasma cell myeloma: impact upon immune determinants of infection.Granzyme K-deficient mice show no evidence of impaired antiviral immunity.Vaccines licensed and in clinical trials for the prevention of dengue.A molecular threshold for effector CD8(+) T cell differentiation controlled by transcription factors Blimp-1 and T-bet.Acetylation of the Cd8 Locus by KAT6A Determines Memory T Cell Diversity.CXCR5(+) follicular cytotoxic T cells control viral infection in B cell follicles.RUNX2 Mediates Plasmacytoid Dendritic Cell Egress from the Bone Marrow and Controls Viral Immunity.Hydrodynamic Injection as a Method of Gene Delivery in Mice: A Model of Chronic Hepatitis B Virus Infection.Role of proapoptotic BH3-only proteins in Listeria monocytogenes infection.Severe Malaria Infections Impair Germinal Center Responses by Inhibiting T Follicular Helper Cell Differentiation.Fas regulates neutrophil lifespan during viral and bacterial infection.FADD and caspase-8 are required for cytokine-induced proliferation of hemopoietic progenitor cells.SIDT2 Transports Extracellular dsRNA into the Cytoplasm for Innate Immune Recognition.Bim controls IL-15 availability and limits engagement of multiple BH3-only proteins.
P50
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P50
description
researcher
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wetenschapper
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հետազոտող
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name
Marc Pellegrini
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Marc Pellegrini
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Marc Pellegrini
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Marc Pellegrini
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type
label
Marc Pellegrini
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Marc Pellegrini
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Marc Pellegrini
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Marc Pellegrini
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Pellegrini M
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prefLabel
Marc Pellegrini
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Marc Pellegrini
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Marc Pellegrini
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Marc Pellegrini
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P1153
35901776400
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0000-0003-3627-3126