about
Spatial and temporal evolution of neuronal activation, stress and injury in lithium-pilocarpine seizures in adult ratsP2X(7) receptor in the kidneys of diabetic rats submitted to aerobic training or to N-acetylcysteine supplementation [corrected].Variations of ATP and its metabolites in the hippocampus of rats subjected to pilocarpine-induced temporal lobe epilepsyNeuroprotective agents and modulation of temporal lobe epilepsy.Effects of A1 receptor agonist/antagonist on spontaneous seizures in pilocarpine-induced epileptic rats.Hippocampal Proteome of Rats Subjected to the Li-Pilocarpine Epilepsy Model and the Effect of Carisbamate Treatment.Deficit in hippocampal long-term potentiation in monosodium glutamate-treated rats.Role of adenosine in the antiepileptic effects of deep brain stimulation.Glutamate-induced alterations in Ca2+ signaling are modulated by mitochondrial Ca2+ handling capacity in brain slices of R6/1 transgenic mice.Alterations in calcium signaling and a decrease in Bcl-2 expression: possible correlation with apoptosis in aged striatum.Evidence that ATP participates in the pathophysiology of pilocarpine-induced temporal lobe epilepsy: fluorimetric, immunohistochemical, and Western blot studies.Alteration of purinergic P2X4 and P2X7 receptor expression in rats with temporal-lobe epilepsy induced by pilocarpine.Protective effect of the organotelluroxetane RF-07 in pilocarpine-induced status epilepticus.Neurochemical Changes and c-Fos Mapping in the Brain after Carisbamate Treatment of Rats Subjected to Lithium-Pilocarpine-Induced Status Epilepticus.Consequences of pilocarpine-induced status epilepticus in immunodeficient mice.Pilocarpine-Induced Status Epilepticus Increases the Sensitivity of P2X7 and P2Y1 Receptors to Nucleotides at Neural Progenitor Cells of the Juvenile Rodent Hippocampus.Differential neuroprotection by A(1) receptor activation and A(2A) receptor inhibition following pilocarpine-induced status epilepticus.Bone marrow-derived mesenchymal stem cells versus adipose-derived mesenchymal stem cells for peripheral nerve regeneration.The A1 receptor agonist R-Pia reduces the imbalance between cerebral glucose metabolism and blood flow during status epilepticus: could this mechanism be involved with neuroprotection?Altered Proteins in the Hippocampus of Patients with Mesial Temporal Lobe EpilepsyP2X7 receptor and klotho expressions in diabetic nephropathy progression
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Maria José da Silva Fernandes
@ast
Maria José da Silva Fernandes
@en
Maria José da Silva Fernandes
@es
Maria José da Silva Fernandes
@nl
type
label
Maria José da Silva Fernandes
@ast
Maria José da Silva Fernandes
@en
Maria José da Silva Fernandes
@es
Maria José da Silva Fernandes
@nl
prefLabel
Maria José da Silva Fernandes
@ast
Maria José da Silva Fernandes
@en
Maria José da Silva Fernandes
@es
Maria José da Silva Fernandes
@nl
P106
P1153
35614786200
P21
P31
P496
0000-0001-6559-3052