about
Haemophilus ducreyi targets Src family protein tyrosine kinases to inhibit phagocytic signaling.Mutations in the lspA1 and lspA2 genes of Haemophilus ducreyi affect the virulence of this pathogen in an animal model system.Regulatory T cell DNA methyltransferase inhibition accelerates resolution of lung inflammation.Right Ventricular Angiogenesis is an Early Adaptive Response to Chronic Hypoxia-Induced Pulmonary Hypertension.Effect of chronic intermittent hypoxia on triglyceride uptake in different tissues.Therapeutic exercise attenuates neutrophilic lung injury and skeletal muscle wasting.Enhanced resolution of experimental ARDS through IL-4-mediated lung macrophage reprogrammingFlow-cytometric method for simultaneous analysis of mouse lung epithelial, endothelial, and hematopoietic lineage cellsMacrophage A2A adenosinergic receptor modulates oxygen-induced augmentation of murine lung injury.The LspB protein is involved in the secretion of the LspA1 and LspA2 proteins by Haemophilus ducreyi.Regulation of expression of the Haemophilus ducreyi LspB and LspA2 proteins by CpxR.Foxp3+ Regulatory T Cell Expression of Keratinocyte Growth Factor Enhances Lung Epithelial Proliferation.Immunological priming requires regulatory T cells and IL-10-producing macrophages to accelerate resolution from severe lung inflammation.Predicted effects of observed changes in the mRNA and microRNA transcriptome of lung neutrophils during S. pneumoniae pneumonia in mice.Foxp3+ regulatory T cells promote lung epithelial proliferation.A critical role for muscle ring finger-1 in acute lung injury-associated skeletal muscle wasting.Haemophilus ducreyi LspA proteins are tyrosine phosphorylated by macrophage-encoded protein tyrosine kinases.Instillation and Fixation Methods Useful in Mouse Lung Cancer Research.Bronchus-associated Lymphoid Tissue in Kabuki Syndrome with Associated Hyper-IgM Syndrome/Common Variable Immunodeficiency.Correction: Immunological Priming Requires Regulatory T Cells and IL-10–Producing Macrophages To Accelerate Resolution from Severe Lung InflammationTranscriptional analysis of Foxp3+ Tregs and functions of two identified molecules during resolution of ALIAn exophytic, bleeding nodule on the left malar regionEffects of IFN-γ on immune cell kinetics during the resolution of acute lung injury
P50
Q34194693-C3B5046D-D131-4FA7-85C6-BB044E803601Q34931861-9718FA23-439B-4751-83B5-50D4EDEFED14Q35816483-FC5EE089-ED9F-4490-B0F6-7FBE6FCB2219Q36470065-D28F8955-3465-4812-821F-A043C2D0F8B4Q36709586-84127E6B-96C4-42F2-864F-5E6C2C439C1AQ36765186-9867ADD4-DBFC-460C-ADCA-DC21BCA4738BQ36811261-B9A69716-3456-41DF-9498-4ADBBF76679AQ36902409-144A625E-4F7D-4070-BB05-E85843AF1E77Q36999825-AFAF0376-E76C-457D-A732-66F2D655A56EQ37115707-9F39B10F-D9BB-45A3-BC88-037D35CE72CCQ38353976-03D16B3B-2DE5-46F9-AA46-B94512D73742Q38906242-03E04FD0-BE20-4AF0-A5DA-EA63DA19A251Q39001670-1F2BE093-D2E6-4D82-866E-40F787DB118FQ41198010-414D0F55-D527-4E57-9867-0CDF27DB9C1DQ41838208-A1FD7D0F-8DF0-4215-809F-8944236B9EE7Q42085122-3467C5A4-1FB8-4FC0-BF43-B120312C9A83Q42755420-74491219-0551-435B-85A7-59D17E61DEC5Q43114999-89FD23CE-E9E2-4C65-9ACA-6E3A3862B1B0Q49431149-A25A53E1-DC47-4C81-8A23-48811DABF8F8Q58390478-774C4B42-268C-41A2-9D50-0F7D3B4BFED4Q64066385-851D1A6D-BCFC-4FA0-99AF-62F7E73CE281Q84338797-7C0EEB9C-236C-4EFB-94A0-5985BB19D8AFQ89726858-54CA2AD1-3C9B-4E82-8207-7308804103EA
P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Jason R Mock
@ast
Jason R Mock
@en
Jason R Mock
@es
Jason R Mock
@nl
type
label
Jason R Mock
@ast
Jason R Mock
@en
Jason R Mock
@es
Jason R Mock
@nl
prefLabel
Jason R Mock
@ast
Jason R Mock
@en
Jason R Mock
@es
Jason R Mock
@nl
P106
P1153
7005864815
P31
P496
0000-0002-7022-0629