about
Resveratrol prevents oxidative stress and inhibition of Na(+)K(+)-ATPase activity induced by transient global cerebral ischemia in ratsArginine administration decreases cerebral cortex acetylcholinesterase and serum butyrylcholinesterase probably by oxidative stress inductionProline induces alterations on nucleotide hydrolysis in synaptosomes from cerebral cortex of ratsOvariectomy increases Na+, K+-ATPase, acetylcholinesterase and catalase in rat hippocampusThe role of oxidative damage in the neuropathology of organic acidurias: insights from animal studies.Effects of L-2-hydroxyglutaric acid on various parameters of the glutamatergic system in cerebral cortex of rats.In vitro effects of D-2-hydroxyglutaric acid on glutamate binding, uptake and release in cerebral cortex of rats.Creatine prevents the inhibition of energy metabolism and lipid peroxidation in rats subjected to GAA administration.Na+, K+ ATPase activity is reduced in amygdala of rats with chronic stress-induced anxiety-like behavior.Chronic variable stress induces oxidative stress and decreases butyrylcholinesterase activity in blood of rats.Guanidino compounds inhibit acetylcholinesterase and butyrylcholinesterase activities: effect neuroprotector of vitamins E plus C.Proline impairs energy metabolism in cerebral cortex of young rats.N-acetylaspartic acid impairs enzymatic antioxidant defenses and enhances hydrogen peroxide concentration in rat brain.Hyperhomocysteinemia reduces glutamate uptake in parietal cortex of rats.Homocysteine decreases extracellular nucleotide hydrolysis in rat platelets.Methylphenidate treatment increases Na(+), K (+)-ATPase activity in the cerebrum of young and adult rats.Exercise effects on activities of Na(+),K(+)-ATPase, acetylcholinesterase and adenine nucleotides hydrolysis in ovariectomized rats.Protective effect of antioxidants on cerebrum oxidative damage caused by arginine on pyruvate kinase activity.Tyrosine administration decreases glutathione and stimulates lipid and protein oxidation in rat cerebral cortex.Acute and chronic hypermethioninemia alter Na+ K+-ATPase activity in rat hippocampus: prevention by antioxidants.Reduction of large neutral amino acid levels in plasma and brain of hyperleucinemic rats.Nitric oxide synthase inhibition by L-NAME prevents the decrease of Na+,K+-ATPase activity in midbrain of rats subjected to arginine administration.Inhibition of rat brain Na+, K+-ATPase activity induced by homocysteine is probably mediated by oxidative stress.Inhibition of Na+,K+-ATPase activity from rat hippocampus by proline.Inhibition of the mitochondrial respiratory chain complex activities in rat cerebral cortex by methylmalonic acid.Experimental hyperphenylalaninemia provokes oxidative stress in rat brain.On the mechanism of the inhibition of Na(+), K(+)-ATPase activity caused by homocysteine.Inhibition of Na+, K+-ATPase activity by the metabolites accumulating in homocystinuria.Arginine administration inhibits hippocampal Na(+),K(+)-ATPase activity and impairs retention of an inhibitory avoidance task in rats.Pretreatment with vitamins E and C prevent the impairment of memory caused by homocysteine administration in rats.Alanine prevents the reduction of pyruvate kinase activity in brain cortex of rats subjected to chemically induced hyperphenylalaninemia.Reduction of Na(+),K(+)-ATPase activity in hippocampus of rats subjected to chemically induced hyperhomocysteinemia.Inhibition of creatine kinase activity in vitro by ethylmalonic acid in cerebral cortex of young rats.Effect of leucine administration on creatine kinase activity in rat brain.Proline induces oxidative stress in cerebral cortex of rats.Kinetic studies on the inhibition of creatine kinase activity by branched-chain alpha-amino acids in the brain cortex of rats.Creatine kinase activity from rat brain is inhibited by branched-chain amino acids in vitro.Ethylmalonic acid inhibits mitochondrial creatine kinase activity from cerebral cortex of young rats in vitro.Brain Na+,K(+)-ATPase inhibition induced by arginine administration is prevented by vitamins E and C.Ascorbic acid prevents water maze behavioral deficits caused by early postnatal methylmalonic acid administration in the rat.
P50
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P50
description
researcher
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wetenschapper
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հետազոտող
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name
Angela T.S. Wyse
@ast
Angela T.S. Wyse
@en
Angela T.S. Wyse
@es
Angela T.S. Wyse
@nl
type
label
Angela T.S. Wyse
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Angela T.S. Wyse
@en
Angela T.S. Wyse
@es
Angela T.S. Wyse
@nl
prefLabel
Angela T.S. Wyse
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Angela T.S. Wyse
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Angela T.S. Wyse
@es
Angela T.S. Wyse
@nl
P106
P1153
7006126130
P31
P496
0000-0001-8769-1147