about
Low-level lead exposure increases systolic arterial pressure and endothelium-derived vasodilator factors in rat aortasAcute lead exposure increases arterial pressure: role of the renin-angiotensin systemIncreased superoxide anion production by interleukin-1beta impairs nitric oxide-mediated relaxation in resistance arteriesToxic effects of mercury on the cardiovascular and central nervous systemsHu antigen R is required for NOX-1 but not NOX-4 regulation by inflammatory stimuli in vascular smooth muscle cells.60-Day chronic exposure to low concentrations of HgCl2 impairs sperm quality: hormonal imbalance and oxidative stress as potential routes for reproductive dysfunction in rats.Apocynin prevents vascular effects caused by chronic exposure to low concentrations of mercuryEndothelial dysfunction of rat coronary arteries after exposure to low concentrations of mercury is dependent on reactive oxygen species.Toll-like receptor 4 upregulation by angiotensin II contributes to hypertension and vascular dysfunction through reactive oxygen species productionHuR mediates the synergistic effects of angiotensin II and IL-1β on vascular COX-2 expression and cell migration.Role of NADPH oxidase and iNOS in vasoconstrictor responses of vessels from hypertensive and normotensive rats.Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin.Role of COX-2-derived PGE2 on vascular stiffness and function in hypertension.Toxic effects of mercury, lead and gadolinium on vascular reactivity.Exercise training and cardiometabolic diseases: focus on the vascular system.New roles for old pathways? A circuitous relationship between reactive oxygen species and cyclo-oxygenase in hypertension.Regulator of calcineurin 1 mediates pathological vascular wall remodelingMolecular physiopathology of obesity-related diseases: multi-organ integration by GRK2.NADPH oxidases and vascular remodeling in cardiovascular diseases.G protein-coupled receptor kinase 2 (GRK2) as an integrative signalling node in the regulation of cardiovascular function and metabolic homeostasis.Altered arachidonic acid metabolism via COX-1 and COX-2 contributes to the endothelial dysfunction of penile arteries from obese Zucker rats.Reproductive dysfunction after mercury exposure at low levels: evidence for a role of glutathione peroxidase (GPx) 1 and GPx4 in male rats.Lysyl Oxidase Induces Vascular Oxidative Stress and Contributes to Arterial Stiffness and Abnormal Elastin Structure in Hypertension: Role of p38MAPK.Effect of pentobarbital on the reactivity of isolated human cerebral arteries.Deficiency of MMP17/MT4-MMP proteolytic activity predisposes to aortic aneurysm in mice.Effect of neuropeptide Y on adrenergic and non-adrenergic, non-cholinergic responses in the rat anococcygeus muscle.Alterations in phenylephrine-induced contractions and the vascular expression of Na+,K+-ATPase in ouabain-induced hypertension.Role of iNOS in the vasodilator responses induced by L-arginine in the middle cerebral artery from normotensive and hypertensive rats.Contribution of the endothelin and renin-angiotensin systems to the vascular changes in rats chronically treated with ouabainc-Src, ERK1/2 and Rho kinase mediate hydrogen peroxide-induced vascular contraction in hypertension: role of TXA2, NAD(P)H oxidase and mitochondria.Differential regulation of Nox1, Nox2 and Nox4 in vascular smooth muscle cells from WKY and SHR.Toll-like receptor 4 contributes to vascular remodelling and endothelial dysfunction in angiotensin II-induced hypertensionMAPK pathway activation by chronic lead-exposure increases vascular reactivity through oxidative stress/cyclooxygenase-2-dependent pathways.Chronic exposure to low doses of mercury impairs sperm quality and induces oxidative stress in rats.Augmented oxidative stress and preserved vasoconstriction induced by hydrogen peroxide in coronary arteries in obesity: role of COX-2.Exposure to low mercury concentration in vivo impairs myocardial contractile function.Chronic HgCl(2) treatment increases vasoconstriction induced by electrical field stimulation: role of adrenergic and nitrergic innervation.The C-terminal module IV of connective tissue growth factor, through EGFR/Nox1 signaling, activates the NF-κB pathway and proinflammatory factors in vascular smooth muscle cells.The role of cyclooxygenase (COX)-2 derived prostanoids on vasoconstrictor responses to phenylephrine is increased by exposure to low mercury concentration.Cell volume and ionic transport systems after cold preservation of coronary endothelial cells.
P50
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P50
description
Forscher
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investigador
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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Mercedes Salaices
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P106
P2038
Mercedes_Salaices
P21
P31
P496
0000-0003-3323-1692