about
Automated imaging system for fast quantitation of neurons, cell morphology and neurite morphometry in vivo and in vitro.Pomegranate juice exacerbates oxidative stress and nigrostriatal degeneration in Parkinson's disease.Opposing effects of Apoe/Apoa1 double deletion on amyloid-β pathology and cognitive performance in APP mice.Central role for MCP-1/CCL2 in injury-induced inflammation revealed by in vitro, in silico, and clinical studiesImpact of the Sensory Neurons on Melanoma Growth In VivoshRNA targeting α-synuclein prevents neurodegeneration in a Parkinson's disease modelExpression of human E46K-mutated α-synuclein in BAC-transgenic rats replicates early-stage Parkinson's disease features and enhances vulnerability to mitochondrial impairment.Behavioral, neurochemical, and pathologic alterations in bacterial artificial chromosome transgenic G2019S leucine-rich repeated kinase 2 rats.ABCA1 Deficiency Affects Basal Cognitive Deficits and Dendritic Density in Mice.A rapid and sensitive automated image-based approach for in vitro and in vivo characterization of cell morphology and quantification of cell number and neurite architecture.Melatonin and its brain metabolite N(1)-acetyl-5-methoxykynuramine prevent mitochondrial nitric oxide synthase induction in parkinsonian mice.Melatonin treatment potentiates neurodegeneration in a rat rotenone Parkinson's disease model.Phenylpyrrole derivatives as neural and inducible nitric oxide synthase (nNOS and iNOS) inhibitors.Pyrazoles and pyrazolines as neural and inducible nitric oxide synthase (nNOS and iNOS) potential inhibitors (III).Identification of an inducible nitric oxide synthase in diaphragm mitochondria from septic mice: its relation with mitochondrial dysfunction and prevention by melatonin.Melatonin counteracts inducible mitochondrial nitric oxide synthase-dependent mitochondrial dysfunction in skeletal muscle of septic mice.Resveratrol and pinostilbene confer neuroprotection against aging-related deficits through an ERK1/2-dependent mechanism.Mitochondrial Complex I Reversible S-Nitrosation Improves Bioenergetics and Is Protective in Parkinson's Disease.Synthetic alpha-synuclein fibrils cause mitochondrial impairment and selective dopamine neurodegeneration in part via iNOS-mediated nitric oxide production.Protective effects of synthetic kynurenines on 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonism in micePhenothiazine normalizes the NADH/NAD ratio, maintains mitochondrial integrity and protects the nigrostriatal dopamine system in a chronic rotenone model of Parkinson's diseaseBenfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy
P50
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Víctor Tapias
@en
Víctor Tapias
@nl
type
label
Víctor Tapias
@en
Víctor Tapias
@nl
prefLabel
Víctor Tapias
@en
Víctor Tapias
@nl
P31
P496
0000-0002-1783-7320