about
Germline mutations in DIS3L2 cause the Perlman syndrome of overgrowth and Wilms tumor susceptibilityAddition of poly(A) and poly(A)-rich tails during RNA degradation in the cytoplasm of human cellsDis3-like 1: a novel exoribonuclease associated with the human exosomeStructural plasticity and in vivo activity of Cas1 from the type I-F CRISPR-Cas systemQuorum Sensing Controls Adaptive Immunity through the Regulation of Multiple CRISPR-Cas SystemsGlobal analysis of the nuclear processing of transcripts with unspliced U12-type introns by the exosome.The Cpf1 CRISPR-Cas protein expands genome-editing tools.Degenerate target sites mediate rapid primed CRISPR adaptation.CRISPRDetect: A flexible algorithm to define CRISPR arrays.The role of CRISPR-Cas systems in virulence of pathogenic bacteriaThe human exosome and disease.Interference-driven spacer acquisition is dominant over naive and primed adaptation in a native CRISPR-Cas system.CRISPR-Cas systems preferentially target the leading regions of MOBF conjugative plasmids.Priming in the Type I-F CRISPR-Cas system triggers strand-independent spacer acquisition, bi-directionally from the primed protospacer.Inactivation of CRISPR-Cas systems by anti-CRISPR proteins in diverse bacterial species.Ethylene regulates fast apoplastic acidification and expansin A transcription during submergence-induced petiole elongation in Rumex palustris.Spacer capture and integration by a type I-F Cas1-Cas2-3 CRISPR adaptation complex.CRISPR-Cas-Mediated Phage Resistance Enhances Horizontal Gene Transfer by Transduction.Analysis of protein-RNA interactions in CRISPR proteins and effector complexes by UV-induced cross-linking and mass spectrometry.Shooting the messenger: RNA-targetting CRISPR-Cas systems.Bioinformatic evidence of widespread priming in type I and II CRISPR-Cas systemsKeeping crispr in check: diverse mechanisms of phage-encoded anti-crisprs.The human exosome and diseaseType I-F CRISPR-Cas resistance against virulent phages results in abortive infection and provides population-level immunityEditor's cut: DNA cleavage by CRISPR RNA-guided nucleases Cas9 and Cas12aDifferent genetic and morphological outcomes for phages targeted by single or multiple CRISPR-Cas spacersGood guide, bad guide: spacer sequence-dependent cleavage efficiency of Cas12a
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description
investigador
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researcher
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wetenschapper
@nl
name
Raymond H J Staals
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Raymond H J Staals
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type
label
Raymond H J Staals
@en
Raymond H J Staals
@nl
prefLabel
Raymond H J Staals
@en
Raymond H J Staals
@nl
P31
P496
0000-0002-5741-9457