about
On, around, and through: neutrophil-endothelial interactions in innate immunityMitogen-activated protein kinase-activated protein kinase 2 mediates apoptosis during lung vascular permeability by regulating movement of cleaved caspase 3Acute lung injury and acute kidney injury are established by four hours in experimental sepsis and are improved with pre, but not post, sepsis administration of TNF-α antibodies.The role of heparanase in pulmonary cell recruitment in response to an allergic but not non-allergic stimulus.Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.Analysis of Total Human Urinary Glycosaminoglycan Disaccharides by Liquid Chromatography-Tandem Mass SpectrometrySoluble guanylyl cyclase contributes to ventilator-induced lung injury in miceThe pulmonary endothelial glycocalyx regulates neutrophil adhesion and lung injury during experimental sepsis.Heparan Sulfate in the Developing, Healthy, and Injured Lung.Heparanase mediates renal dysfunction during early sepsis in mice.Circulating Heparan Sulfate Fragments Attenuate Histone-Induced Lung Injury Independently of Histone Binding.Adhesion Molecules: Master Controllers of the Circulatory System.Endothelial and Microcirculatory Function and Dysfunction in Sepsis.In vivo measurement of the mouse pulmonary endothelial surface layer.Role of Apoptosis in Amplifying Inflammatory Responses in Lung Diseases.The endothelial glycocalyx: an important regulator of the pulmonary vascular barrier.A model-specific role of microRNA-223 as a mediator of kidney injury during experimental sepsis.Severe Sepsis Outcomes Among Hospitalizations With Inflammatory Bowel Disease.Rtp801 suppression of epithelial mTORC1 augments endotoxin-induced lung inflammation.Introduction to the 57th annual Thomas L. Petty Aspen Lung Conference: Rebuilding the injured lungMore than a biomarker: the systemic consequences of heparan sulfate fragments released during endothelial surface layer degradation (2017 Grover Conference Series).Neutrophil transfer of miR-223 to lung epithelial cells dampens acute lung injury in mice.Chemoenzymatic synthesis of unmodified heparin oligosaccharides: cleavage of p-nitrophenyl glucuronide by alkaline and Smith degradation.Sequestering Damage-associated Molecular Patterns in Critical Illness. A Novel Homeostatic Role for the Erythrocyte.Epithelial Heparan Sulfate Contributes to Alveolar Barrier Function and is Shed During Lung Injury.Challenging Dogma: The Value of Bolus Fluids in the Early Resuscitation of Hyperdynamic SepsisThe Pulmonary Endothelial Glycocalyx in ARDS: A Critical Role for Heparan SulfateThe glycocalyx: a novel diagnostic and therapeutic target in sepsisImages in clinical medicine. Toxic neutrophils in Streptococcus pneumoniae sepsisThe expanding appreciation of heparanase in human diseaseHyperoxia and apoptosis. Too much of a good thing?Electronic Cigarettes Are as Toxic to Skin Flap Survival as Tobacco CigarettesIntravital Microscopy in the Mouse LungCirculating heparin oligosaccharides rapidly target the hippocampus in sepsis, potentially impacting cognitive functionsCirculating heparan sulfate fragments mediate septic cognitive dysfunctionIntravenous fluid resuscitation is associated with septic endothelial glycocalyx degradationStaphylococcus aureus adhesion in endovascular infections is controlled by the ArlRS-MgrA signaling cascadeUsing the Extremes of Human Inflammation to Understand the Transcriptional Control of IL-18Direct Intrabronchial Administration to Improve the Selective Agent Deposition Within the Mouse LungLoss of endothelial sulfatase-1 after experimental sepsis attenuates subsequent pulmonary inflammatory responses
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Eric P Schmidt
@en
Eric P Schmidt
@nl
type
label
Eric P Schmidt
@en
Eric P Schmidt
@nl
prefLabel
Eric P Schmidt
@en
Eric P Schmidt
@nl
P31
P496
0000-0002-7360-0504