about
Heart failure with preserved ejection fraction: emerging drug strategiesTranslating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulatesUsing proteomics to uncover extracellular matrix interactions during cardiac remodeling.Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodelingCitrate synthase is a novel in vivo matrix metalloproteinase-9 substrate that regulates mitochondrial function in the postmyocardial infarction left ventricle.P. gingivalis lipopolysaccharide intensifies inflammation post-myocardial infarction through matrix metalloproteinase-9Evaluation of crosslinked and non-crosslinked biologic prostheses for abdominal hernia repair.Secreted protein acidic and rich in cysteine facilitates age-related cardiac inflammation and macrophage M1 polarization.Plasma Glycoproteomics Reveals Sepsis Outcomes Linked to Distinct Proteins in Common Pathways.A Novel Collagen Matricryptin Reduces Left Ventricular Dilation Post-Myocardial Infarction by Promoting Scar Formation and Angiogenesis.Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction.Opposing aging-related shift of excitatory dopamine D1 and inhibitory D3 receptor protein expression in striatum and spinal cordMatrix metalloproteinase-9-dependent mechanisms of reduced contractility and increased stiffness in the aging heartCirculating Porphyromonas gingivalis lipopolysaccharide resets cardiac homeostasis in mice through a matrix metalloproteinase-9-dependent mechanism.Texas 3-step decellularization protocol: looking at the cardiac extracellular matrix.Women are different: the role of coupling factor 6 in blood pressure regulationAge and SPARC change the extracellular matrix composition of the left ventricle.Cardiac wound healing post-myocardial infarction: a novel method to target extracellular matrix remodeling in the left ventricleOsteopontin is proteolytically processed by matrix metalloproteinase 9.Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.Extracellular matrix proteomics in cardiac ischemia/reperfusion: the search is on.Effect of crosslinking on the performance of a collagen-derived biomaterial as an implant for soft tissue repair: a rodent model.Osteopontin: A major player on hypertension-induced vascular remodelingThe Prospective Cardioprotective Effects of DPP-4 inhibition in the ischemic myocardium
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description
investigador
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researcher
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wetenschapper
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name
Lisandra E de Castro Brás
@en
Lisandra E de Castro Brás
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type
label
Lisandra E de Castro Brás
@en
Lisandra E de Castro Brás
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prefLabel
Lisandra E de Castro Brás
@en
Lisandra E de Castro Brás
@nl
P31
P496
0000-0001-9014-8858