about
Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infectionsMacrophage: SHIP of ImmunityAltered Natural Killer Cell Function in HIV-Exposed Uninfected InfantsDown regulation of macrophage IFNGR1 exacerbates systemic L. monocytogenes infectionType I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.Macrophages at the fork in the road to health or diseaseMPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP.General parity between trio and pairwise breeding of laboratory mice in static cagingBacterial Manipulation of NK Cell Regulatory Activity Increases Susceptibility to Listeria monocytogenes Infection.Type I IFNs downregulate myeloid cell IFN-γ receptor by inducing recruitment of an early growth response 3/NGFI-A binding protein 1 complex that silences ifngr1 transcription.Rituximab as successful adjunct treatment in a patient with disseminated nontuberculous mycobacterial infection due to acquired anti-interferon-γ autoantibodyA Breakthrough: Macrophage-Directed Cancer Immunotherapy.Editorial overview: Tuning the innate immune response to pathogensThe gut microbiota in infants of obese mothers increases inflammation and susceptibility to NAFLDA Listeria-derived polypeptide promotes in vivo activation of NK cells for antitumor therapyA Batf3/Nlrp3/IL-18 Axis Promotes Natural Killer Cell IL-10 Production during Listeria monocytogenes InfectionCrosstalk between type I and II interferons in regulation of myeloid cell responses during bacterial infectionNK Cell IL-10 Production Requires IL-15 and IL-10 Driven STAT3 ActivationLigand-induced IFNGR1 down-regulation calibrates myeloid cell IFNγ responsivenessAuthor Correction: The gut microbiota in infants of obese mothers increases inflammation and susceptibility to NAFLDIFNγ receptor down-regulation facilitates Legionella survival in alveolar macrophages
P50
Q26849516-67772566-1DC3-4A38-B23C-50EC471C8AAAQ27027100-E386ED6C-E990-42A6-80AF-4316C59D4690Q33598613-F89CDC18-D9F9-4C7D-8EE6-077CBB83F463Q33758400-9BDD82ED-0C77-499A-8DB0-C19B7EA4A936Q34164176-5E6361BB-B17E-4834-8F5A-366FA4707CC1Q35087352-901C87B1-43F8-4131-88AA-96B1EB4F3600Q35175367-84F039CC-57AF-4E1D-A0F8-EFA7691AC36BQ35633052-6EF0A72F-F9FD-4E27-852A-A3138C1334C4Q36050535-48FDCC99-6CE6-4E09-98A9-739D6CEB19A6Q37184202-CAC1BBB1-33B1-45C0-BE26-08B89838AA27Q37604521-1B5F064C-31F3-41BE-8BB2-44890EBFEFDEQ38698154-C4CF7E5F-D3FD-4FDC-9013-79CB3777C486Q57818866-90FF12F9-F26D-49D2-81F1-352013821222Q58115572-4EF09E71-6181-47E3-A48E-077FEB385912Q88366435-94F94557-7982-4AE4-97B6-D1CA6EE8B6D5Q88920913-AC5D5A35-CE28-475F-8EA9-596D24D82FB6Q89052063-F8B29FE0-1F74-4EFD-9057-3E2C85168D83Q90265919-91A64FF7-8370-435B-898B-B17B0B40F549Q90494406-9C3B1CB1-BF4D-4398-89F8-E7BC54111E18Q91528796-660AC075-3EC9-4688-B7A2-95D0144385E6Q91626344-FAB1728E-5339-49FC-93E6-A812A62DCAFF
P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Laurel L Lenz
@en
Laurel L Lenz
@nl
type
label
Laurel L Lenz
@en
Laurel L Lenz
@nl
prefLabel
Laurel L Lenz
@en
Laurel L Lenz
@nl
P31
P496
0000-0002-4933-8913