Gene 33/Mig-6, a transcriptionally inducible adapter protein that binds GTP-Cdc42 and activates SAPK/JNK. A potential marker transcript for chronic pathologic conditions, such as diabetic nephropathy. Possible role in the response to persistent stre
about
Chk1 phosphorylates the tumour suppressor Mig-6, regulating the activation of EGF signallingActivated Cdc42-associated kinase 1 is a component of EGF receptor signaling complex and regulates EGF receptor degradationWound healing and inflammation genes revealed by array analysis of 'macrophageless' PU.1 null miceFeedback inhibition by RALT controls signal output by the ErbB networkMig-6 is a negative regulator of the epidermal growth factor receptor signalExpression of RALT, a feedback inhibitor of ErbB receptors, is subjected to an integrated transcriptional and post-translational controlLoss of RALT/MIG-6 expression in ERBB2-amplified breast carcinomas enhances ErbB-2 oncogenic potency and favors resistance to HerceptinHypoxia promotes the differentiated human articular chondrocyte phenotype through SOX9-dependent and -independent pathwaysGene 33/Mig6 inhibits hexavalent chromium-induced DNA damage and cell transformation in human lung epithelial cellsThe pro-hypertrophic basic helix-loop-helix protein p8 is degraded by the ubiquitin/proteasome system in a protein kinase B/Akt- and glycogen synthase kinase-3-dependent manner, whereas endothelin induction of p8 mRNA and renal mesangial cell hypertMig-6 is required for appropriate lung development and to ensure normal adult lung homeostasisMig6 is a negative regulator of EGF receptor-mediated skin morphogenesis and tumor formationActivation of the nonreceptor protein tyrosine kinase Ack by multiple extracellular stimuli.The genomic response of skeletal muscle to methylprednisolone using microarrays: tailoring data mining to the structure of the pharmacogenomic time series.The synergistic effect of Mig-6 and Pten ablation on endometrial cancer development and progression.ERBB receptor feedback inhibitor 1 regulation of estrogen receptor activity is critical for uterine implantation in mice.Accelerated and increased joint damage in young mice with global inactivation of mitogen-inducible gene 6 after ligament and meniscus injury.Mig-6 controls EGFR trafficking and suppresses gliomagenesisTargeted disruption of Mig-6 in the mouse genome leads to early onset degenerative joint disease.Genome wide expression profiling reveals suppression of host defence responses during colonisation by Neisseria meningitides but not N. lactamica.Mig6 haploinsufficiency protects mice against streptozotocin-induced diabetesCancer-type regulation of MIG-6 expression by inhibitors of methylation and histone deacetylation.Decreased Mitogen Inducible Gene 6 (MIG-6) Associated with Symptom Severity in Children with Autism.Mig-6 plays a critical role in the regulation of cholesterol homeostasis and bile acid synthesis.13-acetoxysarcocrassolide induces apoptosis on human gastric carcinoma cells through mitochondria-related apoptotic pathways: p38/JNK activation and PI3K/AKT suppressionAmelioration of hypercholesterolemia by an EGFR tyrosine kinase inhibitor in mice with liver-specific knockout of Mig-6.Mig-6 suppresses endometrial cancer associated with Pten deficiency and ERK activation.Mig-6 gene knockout induces neointimal hyperplasia in the vascular smooth muscle cell.Gene expression modifications in Wharton's Jelly mesenchymal stem cells promoted by prolonged in vitro culturingmiR-200 expression regulates epithelial-to-mesenchymal transition in bladder cancer cells and reverses resistance to epidermal growth factor receptor therapy.The regulation of embryo implantation and endometrial decidualization by progesterone receptor signaling.Loss of MIG6 Accelerates Initiation and Progression of Mutant Epidermal Growth Factor Receptor-Driven Lung Adenocarcinoma.Induction of apoptosis by sinulariolide from soft coral through mitochondrial-related and p38MAPK pathways on human bladder carcinoma cellsMitogen-inducible gene 6 triggers apoptosis and exacerbates ER stress-induced β-cell death.Structure and mechanism of activity-based inhibition of the EGF receptor by Mig6Tumor necrosis factor (TNF)-alpha persistently activates nuclear factor-kappaB signaling through the type 2 TNF receptor in chromaffin cells: implications for long-term regulation of neuropeptide gene expression in inflammationA glimpse of various pathogenetic mechanisms of diabetic nephropathy.Mechanical strain activates a program of genes functionally involved in paracrine signaling of angiogenesis.Mig-6 modulates uterine steroid hormone responsiveness and exhibits altered expression in endometrial diseaseFatty Liver and Insulin Resistance in the Liver-Specific Knockout Mice of Mitogen Inducible Gene-6.
P2860
Q24314273-13B7B235-FE2F-42D9-8FBB-40CBC790C23EQ24337716-BFCA89A1-86B8-47D3-9054-6DD96080FCBFQ24805609-238EF742-12DF-409F-B823-23EDD9FA7C59Q28183228-EDB44CD7-3018-483B-8DC1-95FE70339E33Q28201153-D8675EA0-D39C-4B02-8736-28BCE2322EF9Q28201580-7BCB41F4-00DD-4AF9-87B1-A3C3DDB93C1BQ28247112-686744A6-DA0D-4B33-B979-C16DFB9B4D0BQ28260898-87D5CEEF-1FDD-49B2-906D-85B91D1C4CFFQ28395858-913841D4-4C92-4577-B918-EFBF74BCDFE1Q28564994-41C76F52-FC87-4295-BD08-AA0294C7035BQ28587589-7238F0D1-0ADD-489D-BF86-3DC51D6DC67BQ28590384-5CBF1A59-5280-4A9C-A3B9-7536C164C472Q30159764-F71962AB-F0A6-414F-9556-7381D542BF44Q30939855-FEDE0461-5032-4AFA-B49E-7707B4CBC71CQ33580898-A540C4EC-14CB-4613-8A33-FBFAE0F0FB3BQ33742326-8AA7A916-F951-4F01-939C-922180B1D573Q33765726-4EDBA2FB-053D-4A22-A955-98DD132934BDQ33859568-303BA1A5-2767-4C0E-BC90-7B10C2290E71Q33920211-49CFB1A9-9A62-4DCB-A99A-10A51E76A731Q34058392-031B4EEF-3E21-4731-87BB-8BB89D215C3AQ34143192-F38F46BF-FB0B-4B82-8943-D86DCA52BBFDQ34305840-43A09646-62F0-4974-8387-83D9C7440C36Q34342656-B369303C-5F82-48CE-B136-1B2C7F7385C2Q34389069-1784094D-9750-49F0-8548-369D6D19249DQ34411580-9738EA32-8422-46C1-A181-513902E968D5Q34655000-EB71742F-232F-4DD1-8F89-18C2E985E224Q34714606-7EB31CC7-6BDD-4189-9371-C24634251700Q34773853-1730D7CC-FFA6-4870-A1A4-8B0D8A43BBF7Q34992991-0F2F8DFF-1D1B-4F81-9871-3D3F0C0DA7B1Q34996775-EB88564C-3CD2-4167-B3A8-53240FB5CF7CQ35667152-3DC7DCF4-EB1F-4E41-BE42-24512DF78214Q36028210-CE57017F-C207-4705-91E6-07C8AD886235Q36482384-A9489851-CABE-4BE5-8431-EC48DDDB549CQ36533418-D8F8CA39-38B7-4248-9C4F-BC7BEF1165C1Q36683851-24AA43CD-14F8-4948-B2E7-DA41FCF3CDE3Q36692101-772AEE88-3431-4009-9265-CE640AFF1AA0Q36979205-90829CDC-7D08-4CBB-BEF1-10F0CA303815Q37018256-42A6F75B-D487-430C-8656-9AB803E97FEBQ37190151-F8C8F94B-BE14-40B1-98D6-08E09C697FC9Q37521565-8D1B2242-859B-409F-9FE7-7CEC594014FD
P2860
Gene 33/Mig-6, a transcriptionally inducible adapter protein that binds GTP-Cdc42 and activates SAPK/JNK. A potential marker transcript for chronic pathologic conditions, such as diabetic nephropathy. Possible role in the response to persistent stre
description
2000 nî lūn-bûn
@nan
2000 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Gene 33/Mig-6, a transcription ...... he response to persistent stre
@en
Gene 33/Mig-6, a transcription ...... he response to persistent stre
@nl
type
label
Gene 33/Mig-6, a transcription ...... he response to persistent stre
@en
Gene 33/Mig-6, a transcription ...... he response to persistent stre
@nl
prefLabel
Gene 33/Mig-6, a transcription ...... he response to persistent stre
@en
Gene 33/Mig-6, a transcription ...... he response to persistent stre
@nl
P2093
P2860
P921
P3181
P356
P1476
Gene 33/Mig-6, a transcription ...... response to persistent stress
@en
P2093
A Makkinje
C L Cadilla
J M Kyriakis
J V Bonventre
P2860
P304
17838-17847
P3181
P356
10.1074/JBC.M909735199
P407
P50
P577
2000-06-01T00:00:00Z