S100A1: a regulator of myocardial contractility - Wikidata - thesis-toulmin - TriplyDB

S100A1: a regulator of myocardial contractility

S100A1: a regulator of myocardial contractility

http://www.wikidata.org/entity/Q24555086

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Mechanisms of altered Ca²⁺ handling in heart failure Gene therapy targets in heart failure: the path to translation Extracellular S100A1 protein inhibits apoptosis in ventricular cardiomyocytes via activation of the extracellular signal-regulated protein kinase 1/2 (ERK1/2)Transgenic overexpression of the Ca2+-binding protein S100A1 in the heart leads to increased in vivo myocardial contractile performance Impaired cardiac contractility response to hemodynamic stress in S100A1-deficient mice S100A1 is released from ischemic cardiomyocytes and signals myocardial damage via Toll-like receptor 4 S100A1 in human heart failure: lack of recovery following left ventricular assist device support Cardiac AAV9-S100A1 gene therapy rescues post-ischemic heart failure in a preclinical large animal model S100A1 and calmodulin regulation of ryanodine receptor in striated muscle S100A1 genetically targeted therapy reverses dysfunction of human failing cardiomyocytes.S100A1 in cardiovascular health and disease: closing the gap between basic science and clinical therapy Targeting S100A1 in heart failure S100A1 binds to the calmodulin-binding site of ryanodine receptor and modulates skeletal muscle excitation-contraction coupling S100A1: Structure, Function, and Therapeutic Potential S100A1 enhances the L-type Ca2+ current in embryonic mouse and neonatal rat ventricular cardiomyocytes S100A1: a multifaceted therapeutic target in cardiovascular disease Cardiac adenoviral S100A1 gene delivery rescues failing myocardium MicroRNA-138 regulates hypoxia-induced endothelial cell dysfunction by targeting S100A1 Augmentation of Cav1 channel current and action potential duration after uptake of S100A1 in sympathetic ganglion neurons.The inotropic peptide βARKct improves βAR responsiveness in normal and failing cardiomyocytes through G(βγ)-mediated L-type calcium current disinhibition.Differential effects of S100 proteins A2 and A6 on cardiac Ca(2+) cycling and contractile performance.A biosensor of S100A4 metastasis factor activation: inhibitor screening and cellular activation dynamics.S100A1: a regulator of striated muscle sarcoplasmic reticulum Ca2+ handling, sarcomeric, and mitochondrial function Interplay of troponin- and Myosin-based pathways of calcium activation in skeletal and cardiac muscle: the use of W7 as an inhibitor of thin filament activation.Spatiotemporal expression of matrix metalloproteinases (MMPs) is regulated by the Ca2+-signal transducer S100A4 in the pathogenesis of thoracic aortic aneurysm.Differential gene expression in ovarian carcinoma: identification of potential biomarkers S100A1 as a Potential Diagnostic Biomarker for Assessing Cardiotoxicity and Implications for the Chemotherapy of Certain Cancers.Gene delivery approaches to heart failure treatment.Bioactive nanoparticles improve calcium handling in failing cardiac myocytes.Liquid jet delivery method featuring S100A1 gene therapy in the rodent model following acute myocardial infarction.Gene transfer for congestive heart failure: update 2013.S100A1 DNA-based Inotropic Therapy Protects Against Proarrhythmogenic Ryanodine Receptor 2 Dysfunction.Regulation of Na+/Ca2+ exchange current in the normal and failing heart.S100A1: a novel inotropic regulator of cardiac performance. Transition from molecular physiology to pathophysiological relevance.Gene therapy in heart failure.Influence of doxazosin on biosynthesis of S100A6 and atrial natriuretic factor peptides in the heart of spontaneously hypertensive rats S100A1 transgenic treatment of acute heart failure causes proteomic changes in rats.Designing heart performance by gene transfer.Gene therapy to treat cardiovascular disease.Gene and cytokine therapy for heart failure: molecular mechanisms in the improvement of cardiac function.

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S100A1: a regulator of myocardial contractility

http://www.wikidata.org/entity/Q24555086

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2001 nî lūn-bûn

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2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2001 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2001年の論文

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S100A1: a regulator of myocardial contractility

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S100A1: a regulator of myocardial contractility

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S100A1: a regulator of myocardial contractility

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S100A1: a regulator of myocardial contractility

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S100A1: a regulator of myocardial contractility

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S100A1: a regulator of myocardial contractility

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S100A1: a regulator of myocardial contractility

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Proceedings of the National Academy of Sciences of the United States of America

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S100A1: a regulator of myocardial contractility

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A Remppis

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B Pieske

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F Niroomand

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G L Smith

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H A Katus

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P2860

A simplified system for generating recombinant adenoviruses

Tricine-sodium dodecyl sulfate-polyacrylamide gel electrophoresis for the separation of proteins in the range from 1 to 100 kDa

Functional roles of S100 proteins, calcium-binding proteins of the EF-hand type

Right ventricular upregulation of the Ca(2+) binding protein S100A1 in chronic pulmonary hypertension

S100ao (alpha alpha) protein is mainly located in the heart and striated muscles

Altered expression of the Ca(2+)-binding protein S100A1 in human cardiomyopathy

Ions binding to S100 proteins. I. Calcium- and zinc-binding properties of bovine brain S100 alpha alpha, S100a (alpha beta), and S100b (beta beta) protein: Zn2+ regulates Ca2+ binding on S100b protein

New perspectives on S100 proteins: a multi-functional Ca(2+)-, Zn(2+)- and Cu(2+)-binding protein family.

Three-dimensional engineered heart tissue from neonatal rat cardiac myocytes.

Myofilament calcium regulation in human myocardium

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13889-13894

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10.1073/PNAS.241393598

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