FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5 - Wikidata - thesis-toulmin - TriplyDB

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

http://www.wikidata.org/entity/Q24645421

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FAK-MAPK-dependent adhesion disassembly downstream of L1 contributes to semaphorin3A-induced collapse Autocrine netrin function inhibits glioma cell motility and promotes focal adhesion formation The UNC-6/Netrin receptors UNC-40/DCC and UNC-5 inhibit growth cone filopodial protrusion via UNC-73/Trio, Rac-like GTPases and UNC-33/CRMP.In the absence of frazzled over-expression of Abelson tyrosine kinase disrupts commissure formation and causes axons to leave the embryonic CNS.Gonad morphogenesis and distal tip cell migration in the Caenorhabditis elegans hermaphrodite.Down syndrome cell adhesion molecule (DSCAM) associates with uncoordinated-5C (UNC5C) in netrin-1-mediated growth cone collapse.Peters'-plus syndrome is a congenital disorder of glycosylation caused by a defect in the beta1,3-glucosyltransferase that modifies thrombospondin type 1 repeats.Netrin-1 suppresses the MEK/ERK pathway and ITGB4 in pancreatic cancer Role of Netrin-1 Beyond the Brain: From Biomarker of Tissue Injury to Therapy for Inflammatory Diseases.The diverse roles and multiple forms of focal adhesion kinase in brain.Synergistic integration of Netrin and ephrin axon guidance signals by spinal motor neurons.CD151 mediates netrin-1-induced angiogenesis through the Src-FAK-Paxillin pathway.Cooperation and crosstalk in axon guidance cue integration: Additivity, synergy, and fine-tuning in combinatorial signaling.Local translation of the Down syndrome cell adhesion molecule (DSCAM) mRNA in the vertebrate central nervous system.DCC mutation update: Congenital mirror movements, isolated agenesis of the corpus callosum, and developmental split brain syndrome.

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P2860

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

http://www.wikidata.org/entity/Q24645421

description

2006 nî lūn-bûn

@nan

2006 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2006年の論文

@ja

2006年論文

@yue

2006年論文

@zh-hant

2006年論文

@zh-hk

2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

name

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@ast

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@en

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@nl

type

label

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@ast

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@en

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@nl

prefLabel

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@ast

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@en

FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@nl

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Journal of Cell Science

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FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

@en

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Claudia Jürgense

http://www.w3.org/2001/XMLSchema#string

Claudia Jürgensen

http://www.w3.org/2001/XMLSchema#string

Jennifer Aurandt

http://www.w3.org/2001/XMLSchema#string

Kun-Liang Guan

http://www.w3.org/2001/XMLSchema#string

Weiquan Li

http://www.w3.org/2001/XMLSchema#string

Yi Rao

http://www.w3.org/2001/XMLSchema#string

P2860

Netrin-integrin signaling in epithelial morphogenesis, axon guidance and vascular patterning

Tyrosine phosphorylation of focal adhesion kinase at sites in the catalytic domain regulates kinase activity: a role for Src family kinases

Deleted in Colorectal Cancer (DCC) encodes a netrin receptor

Multiple signaling mechanisms of the UNC-6/netrin receptors UNC-5 and UNC-40/DCC in vivo

The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation

The DCC gene product induces apoptosis by a mechanism requiring receptor proteolysis

Vertebrate homologues of C. elegans UNC-5 are candidate netrin receptors

Cellular mechanisms of netrin function: long-range and short-range actions

Kinases of the Src family: structure and functions

Plexin-neuropilin-1 complexes form functional semaphorin-3A receptors

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47-55

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scholarly article

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3887181

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10.1242/JCS.02697

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Pt 1

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P478

119

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2006-01-01T00:00:00Z

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7398831

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16371650

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phosphorylation

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2248276

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