The Nalp3 inflammasome is essential for the development of silicosis - Wikidata - thesis-toulmin - TriplyDB

The Nalp3 inflammasome is essential for the development of silicosis

The Nalp3 inflammasome is essential for the development of silicosis

http://www.wikidata.org/entity/Q24645580

about

Type I interferon and pattern recognition receptor signaling following particulate matter inhalation Mechanisms of carbon nanotube-induced toxicity: focus on oxidative stress Advances in mechanisms and signaling pathways of carbon nanotube toxicity Sintered indium-tin oxide particles induce pro-inflammatory responses in vitro, in part through inflammasome activation Blood gene expression profiling detects silica exposure and toxicity Transcriptomics analysis of lungs and peripheral blood of silica-exposed rats Particle length-dependent titanium dioxide nanomaterials toxicity and bioactivity Reactive oxygen species-independent activation of the IL-1beta inflammasome in cells from patients with chronic granulomatous disease Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)Inflammasomes: too big to miss Function of Nod-like receptors in microbial recognition and host defense Neisseria gonorrhoeae activates the proteinase cathepsin B to mediate the signaling activities of the NLRP3 and ASC-containing inflammasome Fibrosis biomarkers in workers exposed to MWCNTs A review of UHMWPE wear-induced osteolysis: the role for early detection of the immune response.The complex cascade of cellular events governing inflammasome activation and IL-1β processing in response to inhaled particles Lung inflammation caused by inhaled toxicants: a review Inflammasome, IL-1 and inflammation in ozone-induced lung injury Silica, Silicosis, and Autoimmunity Reactive oxygen species and mitochondria: A nexus of cellular homeostasis The Role of Mitochondrial DNA in Mediating Alveolar Epithelial Cell Apoptosis and Pulmonary Fibrosis TRPM2 Channel-Mediated ROS-Sensitive Ca(2+) Signaling Mechanisms in Immune Cells Innate Immunity and Biomaterials at the Nexus: Friends or Foes Reactive oxygen species at the crossroads of inflammasome and inflammation Targeting NADPH oxidases for the treatment of cancer and inflammation NOD-Like Receptors: Master Regulators of Inflammation and Cancer Molecular mechanisms of inflammasome activation during microbial infections Asbestos-induced cellular and molecular alteration of immunocompetent cells and their relationship with chronic inflammation and carcinogenesis An Update on PYRIN Domain-Containing Pattern Recognition Receptors: From Immunity to Pathology Beyond the NLRP3 inflammasome: autoinflammatory diseases reach adolescence Oxidative stress and pulmonary fibrosis Control of innate and adaptive immunity by the inflammasome The inflammasome: an emerging therapeutic oncotarget for cancer prevention The cell biology of inflammasomes: Mechanisms of inflammasome activation and regulation The NLRP3 inflammasome in pathogenic particle and fibre-associated lung inflammation and diseases International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease Initiation and perpetuation of NLRP3 inflammasome activation and assembly Intracellular innate immunity in gouty arthritis: role of NALP3 inflammasome Silica-induced NLRP3 inflammasome activation in vitro and in rat lungs Interlaboratory evaluation of in vitro cytotoxicity and inflammatory responses to engineered nanomaterials: the NIEHS Nano GO Consortium The dendritic cell response to classic, emerging, and homeostatic danger signals. Implications for autoimmunity

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P2860

The Nalp3 inflammasome is essential for the development of silicosis

http://www.wikidata.org/entity/Q24645580

description

2008 nî lūn-bûn

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2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2008 թվականի հուլիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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The Nalp3 inflammasome is essential for the development of silicosis

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The Nalp3 inflammasome is essential for the development of silicosis

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The Nalp3 inflammasome is essential for the development of silicosis

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The Nalp3 inflammasome is essential for the development of silicosis

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The Nalp3 inflammasome is essential for the development of silicosis

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The Nalp3 inflammasome is essential for the development of silicosis

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The Nalp3 inflammasome is essential for the development of silicosis

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PNAS.0803933105

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Proceedings of the National Academy of Sciences of the United States of America

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The Nalp3 inflammasome is essential for the development of silicosis

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P2093

A Brent Carter

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Fayyaz S Sutterwala

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Jeffrey J Sadler

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Linda A Tephly

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Paul B Rothman

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Richard A Flavell

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Shankar S Iyer

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Suzanne L Cassel

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P2860

Mechanisms in the Pathogenesis of Asbestosis and Silicosis

Relative release of interleukin-1beta and interleukin-1 receptor antagonist by alveolar macrophages. A study in asbestos-induced lung disease, sarcoidosis, and idiopathic pulmonary fibrosis

Enhanced IL-1 beta and tumor necrosis factor-alpha release and messenger RNA expression in macrophages from idiopathic pulmonary fibrosis or after asbestos exposure

Polymorphisms of the IL-1 gene complex in coal miners with silicosis

Asbestosis and silicosis

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Active caspase-1 is a regulator of unconventional protein secretion

Role of the caspase-1 inflammasome in Salmonella typhimurium pathogenesis

Gout-associated uric acid crystals activate the NALP3 inflammasome

Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

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9035-9040

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26

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105

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Stephanie Eisenbarth

Oscar R Colegio

Oscar R Colegio

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2008-06-24T00:00:00Z

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5277814

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