EBNA1-specific T cells from patients with multiple sclerosis cross react with myelin antigens and co-produce IFN-gamma and IL-2
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CD8+ T-Cell Deficiency, Epstein-Barr Virus Infection, Vitamin D Deficiency, and Steps to Autoimmunity: A Unifying HypothesisImmune modulation during latent herpesvirus infectionEpstein-Barr virus infection is not a characteristic feature of multiple sclerosis brainB Cells and Autoantibodies in Multiple SclerosisEpstein-Barr virus in systemic lupus erythematosus, rheumatoid arthritis and multiple sclerosis—association and causationEnvironmental risk factors for multiple sclerosis: a review with a focus on molecular mechanismsThe orally available, synthetic ether lipid edelfosine inhibits T cell proliferation and induces a type I interferon responseEpstein-Barr virus stimulates torque teno virus replication: a possible relationship to multiple sclerosisThe long and evolving relationship between viruses and multiple sclerosisEpstein-Barr virus and multiple sclerosis: potential opportunities for immunotherapyEpstein-Barr virus latent infection and BAFF expression in B cells in the multiple sclerosis brain: implications for viral persistence and intrathecal B-cell activation.Elevated Epstein-Barr virus-encoded nuclear antigen-1 immune responses predict conversion to multiple sclerosisHumoral immune response to EBV in multiple sclerosis is associated with disease activity on MRI.Temporal relationship between environmental influenza A and Epstein-Barr viral infections and high multiple sclerosis relapse occurrence.Antiviral immune responses: triggers of or triggered by autoimmunity?Exhausted cytotoxic control of Epstein-Barr virus in human lupus.Environmental risk factors in multiple sclerosis.Gammaherpesvirus latency accentuates EAE pathogenesis: relevance to Epstein-Barr virus and multiple sclerosis.Polymorphisms in the IL2, IL2RA and IL2RB genes in multiple sclerosis risk.Tracing environmental markers of autoimmunity: introducing the infectome.Increased CD8+ T cell response to Epstein-Barr virus lytic antigens in the active phase of multiple sclerosisIntrathecal antibody production against Epstein-Barr and other neurotropic viruses in pediatric and adult onset multiple sclerosis.High-throughput sequencing of TCR repertoires in multiple sclerosis reveals intrathecal enrichment of EBV-reactive CD8+ T cells.Epstein-Barr virus: the path from association to causality for a ubiquitous human pathogen.Autoantigen cross-reactive environmental antigen can trigger multiple sclerosis-like disease.The initiation and prevention of multiple sclerosis.Identifying Patient-Specific Epstein-Barr Nuclear Antigen-1 Genetic Variation and Potential Autoreactive Targets Relevant to Multiple Sclerosis Pathogenesis.A meta-analysis of interaction between Epstein-Barr virus and HLA-DRB1*1501 on risk of multiple sclerosisIncreased CD8+ T cell responses to apoptotic T cell-associated antigens in multiple sclerosis.The essential role of Epstein-Barr virus in the pathogenesis of multiple sclerosisTherapeutic vaccination against the rhesus lymphocryptovirus EBNA-1 homologue, rhEBNA-1, elicits T cell responses to novel epitopes in rhesus macaques.Is the risk of multiple sclerosis related to the 'biography' of the immune system?Polyreactive monoclonal autoantibodies in multiple sclerosis: functional selection from phage display library and characterization by deep sequencing analysis.Deficient EBV-specific B- and T-cell response in patients with chronic fatigue syndrome.Fighting Viral Infections and Virus-Driven Tumors with Cytotoxic CD4+ T Cells.99th Dahlem conference on infection, inflammation and chronic inflammatory disorders: triggering of autoimmune diseases by infections.99th Dahlem conference on infection, inflammation and chronic inflammatory disorders: Epstein-Barr virus and multiple sclerosis: epidemiological evidence.Multiple sclerosis etiology: beyond genes and environment.Role of CD4+ cytotoxic T lymphocytes in the control of viral diseases and cancer.Transient immunosuppression: a bridge between infection and the atypical autoimmunity of Guillain-Barré syndrome?
P2860
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P2860
EBNA1-specific T cells from patients with multiple sclerosis cross react with myelin antigens and co-produce IFN-gamma and IL-2
description
2008 nî lūn-bûn
@nan
2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@ast
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@en
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@nl
type
label
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@ast
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@en
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@nl
prefLabel
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@ast
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@en
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@nl
P2093
P2860
P50
P356
P1476
EBNA1-specific T cells from pa ...... co-produce IFN-gamma and IL-2
@en
P2093
Andreas Lutterotti
Björn Tackenberg
Christian Münz
Susanne Roberts
P2860
P304
P356
10.1084/JEM.20072397
P407
P577
2008-07-28T00:00:00Z