Filamentation by Escherichia coli subverts innate defenses during urinary tract infection - Wikidata - thesis-toulmin - TriplyDB

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

http://www.wikidata.org/entity/Q24676540

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SOS response induces persistence to fluoroquinolones in Escherichia coli Intervening with urinary tract infections using anti-adhesives based on the crystal structure of the FimH-oligomannose-3 complex Adhesive Pili in UTI Pathogenesis and Drug Development Subversion of Host Innate Immunity by Uropathogenic Escherichia coli Urinary tract infections: epidemiology, mechanisms of infection and treatment options Automated image analysis for quantification of filamentous bacteria Fiber formation across the bacterial outer membrane by the chaperone/usher pathway Structure-Based Drug Design and Optimization of Mannoside Bacterial FimH Antagonists Inflammation-Induced Adhesin-Receptor Interaction Provides a Fitness Advantage to Uropathogenic E. coli during Chronic Infection Temperature control of fimbriation circuit switch in uropathogenic Escherichia coli: quantitative analysis via automated model abstraction High-throughput identification of chemical inhibitors of E. coli Group 2 capsule biogenesis as anti-virulence agents Harnessing single cell sorting to identify cell division genes and regulators in bacteria Communal living by bacteria and the pathogenesis of urinary tract infections Urinary Tract Infection: Pathogenesis and Outlook.Morphological plasticity of bacteria-Open questions.Morphological plasticity promotes resistance to phagocyte killing of uropathogenic Escherichia coli Flagellum density regulates Proteus mirabilis swarmer cell motility in viscous environments.Bacteria under the physical constraints of periodic micro-nanofluidic junctions reveal morphological plasticity and dynamic shifting of Min patterns.A man-made ATP-binding protein evolved independent of nature causes abnormal growth in bacterial cells Systems biology approach for mapping the response of human urothelial cells to infection by Enterococcus faecalis.Detection of intracellular bacterial communities in human urinary tract infection.Comparative genomics of the emerging human pathogen Photorhabdus asymbiotica with the insect pathogen Photorhabdus luminescens Polysaccharide capsule and sialic acid-mediated regulation promote biofilm-like intracellular bacterial communities during cystitis.Virulence plasmid harbored by uropathogenic Escherichia coli functions in acute stages of pathogenesis.Metabolic Adaptations of Uropathogenic E. coli in the Urinary Tract.Rapid evolution of phenotypic plasticity and shifting thresholds of genetic assimilation in the nematode Caenorhabditis remanei Transcriptional Alterations of Virulence-Associated Genes in Extended Spectrum Beta-Lactamase (ESBL)-Producing Uropathogenic Escherichia coli during Morphologic Transitions Induced by Ineffective Antibiotics T3SS effector VopL inhibits the host ROS response, promoting the intracellular survival of Vibrio parahaemolyticus.Generation of variants in Listeria monocytogenes continuous-flow biofilms is dependent on radical-induced DNA damage and RecA-mediated repair Bacterial macroscopic rope-like fibers with cytopathic and adhesive properties.Intrauterine growth restriction is a direct consequence of localized maternal uropathogenic Escherichia coli cystitis The role of DNA base excision repair in filamentation in Escherichia coli K-12 adhered to epithelial HEp-2 cells.A murine model of urinary tract infection.You cannot tell a book by looking at the cover: Cryptic complexity in bacterial evolution.Yersinia pestis intracellular parasitism of macrophages from hosts exhibiting high and low severity of plague [Management of biofilm-associated infections: what can we expect from recent research on biofilm lifestyles?].Aberrant community architecture and attenuated persistence of uropathogenic Escherichia coli in the absence of individual IHF subunits.Bacterial differentiation, development, and disease: mechanisms for survival.Uropathogenic Escherichia coli superinfection enhances the severity of mouse bladder infection.Emergence of antibiotic resistance from multinucleated bacterial filaments

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P2860

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

http://www.wikidata.org/entity/Q24676540

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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type

label

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

@en

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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prefLabel

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

@ast

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

@en

Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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Proceedings of the National Academy of Sciences of the United States of America

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Filamentation by Escherichia coli subverts innate defenses during urinary tract infection

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David A Hunstad

http://www.w3.org/2001/XMLSchema#string

Patrick C Seed

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Sheryl S Justice

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P2860

Isolation of mononuclear cells and granulocytes from human blood. Isolation of monuclear cells by one centrifugation, and of granulocytes by combining centrifugation and sedimentation at 1 g

Neutrophil extracellular traps kill bacteria

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Prevention of mucosal Escherichia coli infection by FimH-adhesin-based systemic vaccination

The antimicrobial peptide cathelicidin protects the urinary tract against invasive bacterial infection

Uroplakin Ia is the urothelial receptor for uropathogenic Escherichia coli: evidence from in vitro FimH binding

Cutting edge: functional characterization of the effect of the C3H/HeJ defect in mice that lack an Lpsn gene: in vivo evidence for a dominant negative mutation

Differentiation and developmental pathways of uropathogenic Escherichia coli in urinary tract pathogenesis.

Bacterial SOS checkpoint protein SulA inhibits polymerization of purified FtsZ cell division protein.

Suppression of bladder epithelial cytokine responses by uropathogenic Escherichia coli.

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19884-9

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103

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Scott J. Hultgren

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Escherichia coli

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