Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors - Wikidata - thesis-toulmin - TriplyDB

Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

http://www.wikidata.org/entity/Q24796326

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Cardiotonic Steroids as Modulators of Neuroinflammation NMDA Receptor Function During Senescence: Implication on Cognitive Performance The neuron-astrocyte-microglia triad in normal brain ageing and in a model of neuroinflammation in the rat hippocampus Sustained hippocampal IL-1beta overexpression impairs contextual and spatial memory in transgenic mice.Behavioral and monoamine perturbations in adult male mice with chronic inflammation induced by repeated peripheral lipopolysaccharide administration.IL1R2, CCR2, and CXCR4 May Form Heteroreceptor Complexes with NMDAR and D2R: Relevance for Schizophrenia.Rat brain docosahexaenoic acid metabolism is not altered by a 6-day intracerebral ventricular infusion of bacterial lipopolysaccharide.Susceptibility to Calcium Dysregulation during Brain Aging.Interleukin-1 mediates Alzheimer and Lewy body pathologies.Acetate supplementation attenuates lipopolysaccharide-induced neuroinflammation Spermine reverses lipopolysaccharide-induced memory deficit in mice.Insulin improves memory and reduces chronic neuroinflammation in the hippocampus of young but not aged brains Methemoglobin is an endogenous toll-like receptor 4 ligand-relevance to subarachnoid hemorrhage.TNF-α protein synthesis inhibitor restores neuronal function and reverses cognitive deficits induced by chronic neuroinflammation.Effects of nanoparticle zinc oxide on spatial cognition and synaptic plasticity in mice with depressive-like behaviors Lipopolysaccharide induces delayed FosB/DeltaFosB immunostaining within the mouse extended amygdala, hippocampus and hypothalamus, that parallel the expression of depressive-like behavior.Mutations within the selectivity filter of the NMDA receptor-channel influence voltage dependent block by 5-hydroxytryptamine.Tumor necrosis factor-α synthesis inhibitor 3,6'-dithiothalidomide attenuates markers of inflammation, Alzheimer pathology and behavioral deficits in animal models of neuroinflammation and Alzheimer's disease Chronic NMDA administration increases neuroinflammatory markers in rat frontal cortex: cross-talk between excitotoxicity and neuroinflammation.Differential effects of duration and age on the consequences of neuroinflammation in the hippocampus Riluzole partially rescues age-associated, but not LPS-induced, loss of glutamate transporters and spatial memory.Critical appraisal of the long-term impact of memantine in treatment of moderate to severe Alzheimer's disease.Time-Dependent Compensatory Responses to Chronic Neuroinflammation in Hippocampus and Brainstem: The Potential Role of Glutamate Neurotransmission Age and duration of inflammatory environment differentially affect the neuroimmune response and catecholaminergic neurons in the midbrain and brainstem.Neuroinflammation and the plasticity-related immediate-early gene Arc.Oxidative stress and β-amyloid protein in Alzheimer's disease.Telomere shortening and Alzheimer's disease.Chronic brain inflammation causes a reduction in GluN2A and GluN2B subunits of NMDA receptors and an increase in the phosphorylation of mitogen-activated protein kinases in the hippocampus.Construction and implantation of a microinfusion system for sustained delivery of neuroactive agents.Bacterial lipopolysaccharide induces a dose-dependent activation of neuroglia and loss of basal forebrain cholinergic cells in the rat brain.Anti-inflammatory property of the cannabinoid agonist WIN-55212-2 in a rodent model of chronic brain inflammation.Stimulation of Toll-like receptor 9 by chronic intraventricular unmethylated cytosine-guanine DNA infusion causes neuroinflammation and impaired spatial memory.Initiating Mechanisms of Surgery-induced Memory Decline: The Role of HMGB1.Molecular and cellular characterization of the age-related neuroinflammatory processes occurring in normal rat hippocampus: potential relation with the loss of somatostatin GABAergic neurons.Involvement of interleukin-1 in lipopolysaccaride-induced microglial activation and learning and memory deficits.Unique Contribution of Haptoglobin and Haptoglobin Genotype in Aneurysmal Subarachnoid Hemorrhage.Sleep-Aids Derived from Natural Products.

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P2860

Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

http://www.wikidata.org/entity/Q24796326

description

2004 nî lūn-bûn

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2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2004 թվականի հուլիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年论文

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name

Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

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Journal of Neuroinflammation

P1476

Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

@en

P2093

Beatrice Hauss-Wegrzyniak

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Gary L Wenk

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Susanna Rosi

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Victor Ramirez-Amaya

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P2860

Protein measurement with the Folin phenol reagent

Neuropathological stageing of Alzheimer-related changes

Inflammation and Alzheimer's disease

Heteromeric NMDA receptors: molecular and functional distinction of subtypes

Glutamate receptor function in learning and memory

Direct evidence for tumor necrosis factor-induced mitochondrial reactive oxygen intermediates and their involvement in cytotoxicity

The neuropharmacological basis for the use of memantine in the treatment of Alzheimer's disease.

Alternative excitotoxic hypotheses.

Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

Altered glutamatergic transmission in neurological disorders: from high extracellular glutamate to excessive synaptic efficacy.

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