Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
about
Lead and cognitive function in adults: a questions and answers approach to a review of the evidence for cause, treatment, and preventionPublic health and economic consequences of methyl mercury toxicity to the developing brainFetal environment and schizophrenia.Prenatal lead exposure and schizophrenia: a plausible neurobiologic connectionThe National Children's Study: a critical national investmentUrbanization and traffic related exposures as risk factors for schizophreniaInteractions of lifetime lead exposure and stress: behavioral, neurochemical and HPA axis effectsAlterations in glucocorticoid negative feedback following maternal Pb, prenatal stress and the combination: a potential biological unifying mechanism for their corresponding disease profilesThe protean toxicities of lead: new chapters in a familiar storyBlood lead concentration and thyroid function during pregnancy: results from the Yugoslavia Prospective Study of Environmental Lead ExposureAnimal models of gene-environment interaction in schizophrenia: A dimensional perspective.Chronic exposure of mutant DISC1 mice to lead produces sex-dependent abnormalities consistent with schizophrenia and related mental disorders: a gene-environment interaction study.Early-life lead exposure recapitulates the selective loss of parvalbumin-positive GABAergic interneurons and subcortical dopamine system hyperactivity present in schizophrenia.Is lead exposure in early life an environmental risk factor for Schizophrenia? Neurobiological connections and testable hypotheses.The environment and susceptibility to schizophrenia.A classification of sociomedical health indicators: perspectives for health administrators and health planners.The consequence of errors. From memory molecules to the criminal chromosome, erroneous conclusions continue to blight scientific research.A review of research trends in physiological abnormalities in autism spectrum disorders: immune dysregulation, inflammation, oxidative stress, mitochondrial dysfunction and environmental toxicant exposures.Effect of socioeconomic status and parents' education at birth on risk of schizophrenia in offspring.Tetrachloroethylene exposure and risk of schizophrenia: offspring of dry cleaners in a population birth cohort, preliminary findings.Sex-based differences in gene expression in hippocampus following postnatal lead exposure.Dysregulation of BDNF-TrkB signaling in developing hippocampal neurons by Pb(2+): implications for an environmental basis of neurodevelopmental disordersEarly-life metal exposure and schizophrenia: A proof-of-concept study using novel tooth-matrix biomarkers.Low-level human equivalent gestational lead exposure produces sex-specific motor and coordination abnormalities and late-onset obesity in year-old miceThe environment in pediatric practice: a study of New York pediatricians' attitudes, beliefs, and practices towards children's environmental health.Prenatal exposure to lead, delta-aminolevulinic acid, and schizophrenia: further evidence.In Vitro Effects of Lead on Gene Expression in Neural Stem Cells and Associations between Up-regulated Genes and Cognitive Scores in Children.Environmental exposure to lead and mercury in Mexican children: a real health problem.Lead neurotoxicity: exploring the potential impact of lead substitution in zinc-finger proteins on mental health.Thermodynamics of Pb(ii) and Zn(ii) binding to MT-3, a neurologically important metallothionein.Social disorganization of neighborhoods and incidence of psychotic disorders: a 7-year first-contact incidence study.RNA-Seq of Human Neural Progenitor Cells Exposed to Lead (Pb) Reveals Transcriptome Dynamics, Splicing Alterations and Disease Risk Associations.Comparison of blood lead and blood and plasma δ-aminolevulinic acid concentrations as biomarkers for lead poisoning in cattle.Prenatal lead exposure and schizophrenia: further evidence and more neurobiological connections.Time-dependent changes in lead and delta-aminolevulinic acid after subchronic lead exposure in rats.Sensorimotor gating deficits in "two-hit" models of schizophrenia risk factors.Oxidative stress and neurological disorders in relation to blood lead levels in children.Interactions of chronic lead exposure and intermittent stress: consequences for brain catecholamine systems and associated behaviors and HPA axis function.Choosing the types of biological sample to collect in longitudinal birth cohort studiesEnvironmental Risk Factors for Schizophrenia
P2860
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P2860
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
description
2004 nî lūn-bûn
@nan
2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia
@nl
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@ast
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@en
type
label
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia
@nl
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@ast
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@en
prefLabel
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia
@nl
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@ast
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@en
P2093
P2860
P356
P1476
Prenatal lead exposure, delta-aminolevulinic acid, and schizophrenia.
@en
P2093
Alan S Brown
Catherine Schaefer
Ezra S Susser
Joseph Graziano
Manisha Desai
Mark G A Opler
Pamela Factor-Litvak
P2860
P304
P356
10.1289/EHP.6777
P407
P577
2004-04-01T00:00:00Z