MCP-1 overexpressed in tuberous sclerosis lesions acts as a paracrine factor for tumor development.
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Mesenchymal-epithelial interactions involving epiregulin in tuberous sclerosis complex hamartomasLymphangioleiomyomatosis and TSC2-/- cellsThe natural history of lymphangioleiomyomatosis: markers of severity, rate of progression and prognosis.Involvement of lymphatics in lymphangioleiomyomatosisGene expression analysis of tuberous sclerosis complex cortical tubers reveals increased expression of adhesion and inflammatory factors.Chemokine-enhanced chemotaxis of lymphangioleiomyomatosis cells with mutations in the tumor suppressor TSC2 gene.Acral lesions in tuberous sclerosis complex: insights into pathogenesisThe role of chemokines in migration of metastatic-like lymphangioleiomyomatosis cells.Lymphangioleiomyomatosis (LAM): molecular insights lead to targeted therapies.Human TSC2-null fibroblast-like cells induce hair follicle neogenesis and hamartoma morphogenesis.Lymphatic involvement in lymphangioleiomyomatosisCooperative benefit for the combination of rapamycin and imatinib in tuberous sclerosis complex neoplasia.Nipple Angiofibromas with Loss of TSC2 Are Associated with Tuberous Sclerosis Complex.Recruitment of the inflammatory subset of monocytes to sites of ischemia induces angiogenesis in a monocyte chemoattractant protein-1-dependent fashion.Application of angiogenesis to clinical dermatology.EPIDEMIOLOGY, PATHOGENESIS and DIAGNOSIS of LYMPHANGIOLEIOMYOMATOSIS.Feasibility of immunotherapy for lymphangioleiomyomatosis.Sun exposure causes somatic second-hit mutations and angiofibroma development in tuberous sclerosis complexA current viewpoint of lymphangioleiomyomatosis supporting immunotherapeutic treatment options.Resveratrol and its synthetic derivatives exert opposite effects on mesothelial cell-dependent angiogenesis via modulating secretion of VEGF and IL-8/CXCL8.Tsc2 disruption in mesenchymal progenitors results in tumors with vascular anomalies overexpressing Lgals3.Cystic Lung Disease in Genetic Syndromes with Deficient Tumor Suppressor Gene Function.The LAM Lung Cell and Its Human Cell Models.Aberrant SYK Kinase Signaling Is Essential for Tumorigenesis Induced by TSC2 Inactivation.The functional polymorphism in monocyte chemoattractant protein-1 gene increases susceptibility to gastric cancer.
P2860
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P2860
MCP-1 overexpressed in tuberous sclerosis lesions acts as a paracrine factor for tumor development.
description
2005 nî lūn-bûn
@nan
2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
MCP-1 overexpressed in tuberou ...... e factor for tumor development
@nl
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@ast
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@en
type
label
MCP-1 overexpressed in tuberou ...... e factor for tumor development
@nl
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@ast
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@en
prefLabel
MCP-1 overexpressed in tuberou ...... e factor for tumor development
@nl
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@ast
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@en
P2093
P2860
P356
P1476
MCP-1 overexpressed in tuberou ...... factor for tumor development.
@en
P2093
Christopher Fuller
Fumiko Takeuchi
Gustavo Pacheco-Rodriguez
Ji-an Wang
Shaowei Li
Thomas N Darling
P2860
P304
P356
10.1084/JEM.20042469
P407
P577
2005-08-29T00:00:00Z