The hepatitis C virus 3'-untranslated region or a poly(A) tract promote efficient translation subsequent to the initiation phase.
about
MicroRNA-122 dependent binding of Ago2 protein to hepatitis C virus RNA is associated with enhanced RNA stability and translation stimulationThe 3' proximal translational enhancer of Turnip crinkle virus binds to 60S ribosomal subunitsmicroRNA-122 stimulates translation of hepatitis C virus RNAAnimal virus schemes for translation dominanceThe Hepatitis C Virus RNA 3'-Untranslated Region Strongly Enhances Translation Directed by the Internal Ribosome Entry SitePoly(A)-binding protein is differentially required for translation mediated by viral internal ribosome entry sitesStudying Hepatitis C Virus: Making the Best of a Bad VirusThe major form of hepatitis C virus alternate reading frame protein is suppressed by core protein expressionA long-range RNA-RNA interaction between the 5' and 3' ends of the HCV genomeZebrafish as a potential model organism for drug test against hepatitis C virusThe inhibitory effects of anacardic acid on hepatitis C virus life cycleXRN1 stalling in the 5' UTR of Hepatitis C virus and Bovine Viral Diarrhea virus is associated with dysregulated host mRNA stabilityCharacterization of nonprimate hepacivirus and construction of a functional molecular clonePoly(A)-binding protein modulates mRNA susceptibility to cap-dependent miRNA-mediated repression.An internal ribosome entry site directs translation of the 3'-gene from Pelargonium flower break virus genomic RNA: implications for infectivity.The 3'-terminal hexamer sequence of classical swine fever virus RNA plays a role in negatively regulating the IRES-mediated translation.Induction of viral, 7-methyl-guanosine cap-independent translation and oncolysis by mitogen-activated protein kinase-interacting kinase-mediated effects on the serine/arginine-rich protein kinase.Hepatitis C virus NS5A binds to the mRNA cap-binding eukaryotic translation initiation 4F (eIF4F) complex and up-regulates host translation initiation machinery through eIF4E-binding protein 1 inactivation.Y-Box Binding Protein 1 Stabilizes Hepatitis C Virus NS5A via Phosphorylation-Mediated Interaction with NS5A To Regulate Viral Propagation.Activation of cap-independent translation by variant eukaryotic initiation factor 4G in vivo.A miRNA-responsive cell-free translation system facilitates isolation of hepatitis C virus miRNP complexes.Hepatitis C virus 3'UTR regulates viral translation through direct interactions with the host translation machinery.The landscape of viral expression and host gene fusion and adaptation in human cancerThe 3' end of Turnip crinkle virus contains a highly interactive structure including a translational enhancer that is disrupted by binding to the RNA-dependent RNA polymerase.Hepatitis C viral protein translation: mechanisms and implications in developing antivirals.The role and clinical implications of microRNAs in hepatocellular carcinoma.Unmasking the information encoded as structural motifs of viral RNA genomes: a potential antiviral target.NMR study of RNA structures in the 3´-end of the Hepatitis C Virus genome.A researcher's guide to the galaxy of IRESs.Mutations in HCV non-structural genes do not contribute to resistance to nitazoxanide in replicon-containing cells.The functional RNA domain 5BSL3.2 within the NS5B coding sequence influences hepatitis C virus IRES-mediated translation.Influence of the hepatitis C virus 3'-untranslated region on IRES-dependent and cap-dependent translation initiation.Hepatitis C virus internal ribosome entry site-mediated translation is stimulated by cis-acting RNA elements and trans-acting viral factors.Functional characterization of fingers subdomain-specific monoclonal antibodies inhibiting the hepatitis C virus RNA-dependent RNA polymerase.A ferritin-responsive internal ribosome entry site regulates folate metabolism.The double-stranded RNA binding protein 76:NF45 heterodimer inhibits translation initiation at the rhinovirus type 2 internal ribosome entry site.In-cell SHAPE uncovers dynamic interactions between the untranslated regions of the foot-and-mouth disease virus RNA.microRNAs stimulate translation initiation mediated by HCV-like IRESes.Effects of short RNA structural analogues against hepatitis C virus genotypes 2, 3 and 4 in replicon cells.Hepatitis C Virus Evasion from RIG-I-Dependent Hepatic Innate Immunity.
P2860
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P2860
The hepatitis C virus 3'-untranslated region or a poly(A) tract promote efficient translation subsequent to the initiation phase.
description
2006 nî lūn-bûn
@nan
2006 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մարտին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
The hepatitis C virus 3'-untra ...... equent to the initiation phase
@nl
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@ast
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@en
type
label
The hepatitis C virus 3'-untra ...... equent to the initiation phase
@nl
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@ast
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@en
prefLabel
The hepatitis C virus 3'-untra ...... equent to the initiation phase
@nl
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@ast
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@en
P2093
P2860
P356
P1476
The hepatitis C virus 3'-untra ...... quent to the initiation phase.
@en
P2093
Matthias Gromeier
Robert W Walters
Shelton S Bradrick
P2860
P304
P356
10.1093/NAR/GKL019
P407
P577
2006-03-01T00:00:00Z