Vitiligo: interplay between oxidative stress and immune system - Wikidata - thesis-toulmin - TriplyDB

Vitiligo: interplay between oxidative stress and immune system

Vitiligo: interplay between oxidative stress and immune system

http://www.wikidata.org/entity/Q26824504

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A central role for inducible heat-shock protein 70 in autoimmune vitiligo Dysfunction of Autophagy: A Possible Mechanism Involved in the Pathogenesis of Vitiligo by Breaking the Redox Balance of Melanocytes Recent advances in understanding vitiligo Methylenetetrahydrofolate reductase (MTHFR): could it be a small piece in vitiligo jigsaw puzzle?Analysis of oxidative stress status, catalase and catechol-O-methyltransferase polymorphisms in Egyptian vitiligo patients CXCL10 is critical for the progression and maintenance of depigmentation in a mouse model of vitiligo.Enhanced bleaching treatment: opportunities for immune-assisted melanocyte suicide in vitiligo.Association of neuropeptide Y (NPY), interleukin-1B (IL1B) genetic variants and correlation of IL1B transcript levels with vitiligo susceptibility Tumor necrosis factor B (TNFB) genetic variants and its increased expression are associated with vitiligo susceptibility Interferon-γ induces senescence in normal human melanocytes.Simvastatin prevents and reverses depigmentation in a mouse model of vitiligo Evaluation of advanced oxidation protein products, prooxidant-antioxidant balance, and total antioxidant capacity in untreated vitiligo patients.Childhood facial vitiligo: how intractable is it?A functional single-nucleotide polymorphism in the ERCC1 gene alters the efficacy of narrowband ultraviolet B therapy in patients with active vitiligo in a Chinese population.FOXP3 is a promising and potential candidate gene in generalised vitiligo susceptibility.AHR promoter variant modulates its transcription and downstream effectors by allele-specific AHR-SP1 interaction functioning as a genetic marker for vitiligo The Effects of Bairesi Complex Prescription (a Uyghur Medicine Prescription) and Its Five Crude Herbal Extracts on Melanogenesis in G-361 Cells Hyperoside protects human primary melanocytes against H2O2-induced oxidative damage.Involvement of interferon-gamma genetic variants and intercellular adhesion molecule-1 in onset and progression of generalized vitiligo Oxidative stress, prooxidants, and antioxidants: the interplay.Decreased glucose-6-phosphate dehydrogenase levels in vitiligo patients: Further evidence of oxidative stress Dysregulated autophagy increased melanocyte sensitivity to H2O2-induced oxidative stress in vitiligo.Novel approaches to vitiligo treatment via modulation of mTOR and NF-κB pathways in human skin melanocytes.The role of tumor necrosis factor-α in the pathogenesis of vitiligo.Association of the 389 C/T polymorphism of the catalase gene with susceptibility to vitiligo: a meta-analysis.Link between PI3K/AKT/PTEN Pathway and NOX Proteinin Diseases.Vitiligo: what's new in the psycho-neuro-endocrine-immune connection and related treatments.Oxidative stress and immune system in vitiligo and thyroid diseases.Increased risk of comorbid rheumatic disorders in vitiligo patients: A nationwide population-based study.miR-196a-2 rs11614913 polymorphism is associated with vitiligo by affecting heterodimeric molecular complexes of Tyr and Tyrp1.Construction of a Comprehensive Protein-Protein Interaction Map for Vitiligo Disease to Identify Key Regulatory Elements: A Systemic Approach.Variants of the melanocortin-1 receptor: do they matter clinically?Effect of narrow band-ultraviolet B on CD4(+) CD25(high) FoxP3(+) T-lymphocytes in the peripheral blood of vitiligo patients.A Lagging Lock: Hair Cycle Abnormalities in Follicular Vitiligo?Vitiligo and alopecia areata: apples and oranges?Genetic polymorphisms in the methylenetetrahydrofolate reductase gene (MTHFR) and risk of vitiligo in Han Chinese populations: a genotype-phenotype correlation study.Levels of oxidative damage and proinflammatory cytokines are enhanced in patients with active vitiligo.Bilobalide protection of normal human melanocytes from hydrogen peroxide-induced oxidative damage via promotion of antioxidase expression and inhibition of endoplasmic reticulum stress.Commentary from the Editorial Board to Vitiligo: interplay between oxidative stress and immune system (Laddha et al.).Role of chemokines and the corresponding receptors in vitiligo: A pilot study.

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Vitiligo: interplay between oxidative stress and immune system

http://www.wikidata.org/entity/Q26824504

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2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013年の論文

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2013年論文

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2013年论文

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Vitiligo: interplay between oxidative stress and immune system

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Vitiligo: interplay between oxidative stress and immune system

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Vitiligo: interplay between oxidative stress and immune system

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Vitiligo: interplay between oxidative stress and immune system

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Experimental Dermatology

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Vitiligo: interplay between oxidative stress and immune system

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A. V. Ramachandran

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Amina R. Gani

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Md Ansarullah

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Mitesh Dwivedi

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Mohmmad S. Mansuri

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Naresh C. Laddha

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Rasheedunnisa Begum

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Sarat Dalai

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P2860

Activation/deactivation of acetylcholinesterase by H2O2: more evidence for oxidative stress in vitiligo

Melanocytorrhagy and apoptosis in vitiligo: connecting jigsaw pieces

Cytotoxic T lymphocyte reactivity to gp100, MelanA/MART-1, and tyrosinase, in HLA-A2-positive vitiligo patients

Increased monoamine oxidase A activity in the epidermis of patients with vitiligo

The transcription factors SOX9 and SOX10 are vitiligo autoantigens in autoimmune polyendocrine syndrome type I.

The melanin-concentrating hormone receptor 1, a novel target of autoantibody responses in vitiligo.

Increased sensitivity of melanocytes to oxidative stress and abnormal expression of tyrosinase-related protein in vitiligo.

In vivo and in vitro evidence for hydrogen peroxide (H2O2) accumulation in the epidermis of patients with vitiligo and its successful removal by a UVB-activated pseudocatalase.

Th17 cells and activated dendritic cells are increased in vitiligo lesions

Role of reactive oxygen species (ROS) in apoptosis induction.

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10.1111/EXD.12103

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23425123

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