B7-H1 blockade increases survival of dysfunctional CD8(+) T cells and confers protection against Leishmania donovani infections
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Modulation of Innate Immune Mechanisms to Enhance Leishmania Vaccine-Induced Immunity: Role of Coinhibitory MoleculesThe Contribution of Immune Evasive Mechanisms to Parasite Persistence in Visceral LeishmaniasisPost-Genomics and Vaccine Improvement for LeishmaniaImmunoregulation in human American leishmaniasis: balancing pathology and protectionThe Combined Deficiency of Immunoproteasome Subunits Affects Both the Magnitude and Quality of Pathogen- and Genetic Vaccination-Induced CD8+ T Cell Responses to the Human Protozoan Parasite Trypanosoma cruziDynamic imaging of experimental Leishmania donovani-induced hepatic granulomas detects Kupffer cell-restricted antigen presentation to antigen-specific CD8 T cellsCD4+ Recent Thymic Emigrants Are Recruited into Granulomas during Leishmania donovani Infection but Have Limited Capacity for Cytokine ProductionLessons from other diseases: granulomatous inflammation in leishmaniasisRegulation of immunity during visceral Leishmania infectionIdentifying vaccine targets for anti-leishmanial vaccine developmentA third generation vaccine for human visceral leishmaniasis and post kala azar dermal leishmaniasis: First-in-human trial of ChAd63-KH.CD8 cells of patients with diffuse cutaneous leishmaniasis display functional exhaustion: the latter is reversed, in vitro, by TLR2 agonists.Immune regulation during chronic visceral leishmaniasis.Immunotherapy and targeted therapies in treatment of visceral leishmaniasis: current status and future prospects.Murine visceral leishmaniasis: IgM and polyclonal B-cell activation lead to disease exacerbation.Dual immune modulatory effect of vitamin A in human visceral leishmaniasis.IL-10-producing Th1 cells and disease progression are regulated by distinct CD11c⁺ cell populations during visceral leishmaniasisImmunobiology of visceral leishmaniasis.Role of CD8(+) T cells in protection against Leishmania donovani infection in healed Visceral Leishmaniasis individuals.Impairment of T cell function in parasitic infections.Differential Regulation of the Immune Response in the Spleen and Liver of Mice Infected with Leishmania donovani.Apoptosis mediated leishmanicidal activity of Azadirachta indica bioactive fractions is accompanied by Th1 immunostimulatory potential and therapeutic cure in vivoImmunogenicity evaluation of a rationally designed polytope construct encoding HLA-A*0201 restricted epitopes derived from Leishmania major related proteins in HLA-A2/DR1 transgenic mice: steps toward polytope vaccineIRF-5-Mediated Inflammation Limits CD8+ T Cell Expansion by Inducing HIF-1α and Impairing Dendritic Cell Functions during Leishmania InfectionTherapeutic vaccination with recombinant adenovirus reduces splenic parasite burden in experimental visceral leishmaniasis.CD8(+) T cells in leishmania infections: friends or foes?Splenic CD4+ T Cells in Progressive Visceral Leishmaniasis Show a Mixed Effector-Regulatory Phenotype and Impair Macrophage Effector Function through Inhibitory Receptor Expression.Leishmanicidal Activity of Piper nigrum Bioactive Fractions is Interceded via Apoptosis In Vitro and Substantiated by Th1 Immunostimulatory Potential In Vivo.Hyperlipidemia offers protection against Leishmania donovani infection: role of membrane cholesterol.Live attenuated Leishmania donovani p27 gene knockout parasites are nonpathogenic and elicit long-term protective immunity in BALB/c mice.Leishmania-encoded orthologs of macrophage migration inhibitory factor regulate host immunity to promote parasite persistence.Parasite-derived arginase influences secondary anti-Leishmania immunity by regulating programmed cell death-1-mediated CD4+ T cell exhaustion.CD8 T cell exhaustion in human visceral leishmaniasis.Programmed death 1-mediated T cell exhaustion during visceral leishmaniasis impairs phagocyte function.Structure-guided development of a high-affinity human Programmed Cell Death-1: Implications for tumor immunotherapy.The PD-1/PD-L1 (B7-H1) pathway in chronic infection-induced cytotoxic T lymphocyte exhaustion.Inhibitory receptors on lymphocytes: insights from infectionsVaccine Development Against Leishmania donovani.T cell exhaustion in protozoan disease.Imaging host-Leishmania interactions: significance in visceral leishmaniasis.
P2860
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P2860
B7-H1 blockade increases survival of dysfunctional CD8(+) T cells and confers protection against Leishmania donovani infections
description
2009 nî lūn-bûn
@nan
2009 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
B7-H1 blockade increases survi ...... Leishmania donovani infections
@ast
B7-H1 blockade increases survi ...... Leishmania donovani infections
@en
B7-H1 blockade increases survi ...... Leishmania donovani infections
@nl
type
label
B7-H1 blockade increases survi ...... Leishmania donovani infections
@ast
B7-H1 blockade increases survi ...... Leishmania donovani infections
@en
B7-H1 blockade increases survi ...... Leishmania donovani infections
@nl
prefLabel
B7-H1 blockade increases survi ...... Leishmania donovani infections
@ast
B7-H1 blockade increases survi ...... Leishmania donovani infections
@en
B7-H1 blockade increases survi ...... Leishmania donovani infections
@nl
P2093
P2860
P3181
P1433
P1476
B7-H1 blockade increases survi ...... Leishmania donovani infections
@en
P2093
Simona Stäger
Susana Rodriguez
Trupti Joshi
Vladimir Perovic
P2860
P304
P3181
P356
10.1371/JOURNAL.PPAT.1000431
P407
P50
P577
2009-05-01T00:00:00Z