An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype - Wikidata - thesis-toulmin - TriplyDB

An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype

An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype

http://www.wikidata.org/entity/Q27339197

about

Transcription factors OVOL1 and OVOL2 induce the mesenchymal to epithelial transition in human cancer JARID1B is a luminal lineage-driving oncogene in breast cancer Sirtuins and Cancer: Role in the Epithelial-Mesenchymal Transition Current status and perspectives in atomic force microscopy-based identification of cellular transformation Splicing Regulation: A Molecular Device to Enhance Cancer Cell Adaptation Epithelial cell polarity determinant CRB3 in cancer development Misregulation of pre-mRNA alternative splicing in cancer Epithelial-mesenchymal transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compounds Molecular mechanisms of epithelial-mesenchymal transition Defective control of pre-messenger RNA splicing in human disease Epithelial-Mesenchymal Plasticity: A Central Regulator of Cancer Progression The role of splicing factors in deregulation of alternative splicing during oncogenesis and tumor progression Role of ABL family kinases in cancer: from leukaemia to solid tumours Genomic mapping of phosphorothioates reveals partial modification of short consensus sequences In vivo quantification of the structural changes of collagens in a melanoma microenvironment with second and third harmonic generation microscopy.The alternatively-included 11a sequence modifies the effects of Mena on actin cytoskeletal organization and cell behavior Identification of potential new treatment response markers and therapeutic targets using a Gaussian process-based method in lapatinib insensitive breast cancer models.Regulated splicing of the α6 integrin cytoplasmic domain determines the fate of breast cancer stem cells.Increased macroH2A1.1 expression correlates with poor survival of triple-negative breast cancer patients N-cadherin/FGFR promotes metastasis through epithelial-to-mesenchymal transition and stem/progenitor cell-like properties.Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing.Distinct Transcriptional and Alternative Splicing Signatures of Decidual CD4+ T Cells in Early Human Pregnancy.Evolution at protein ends: major contribution of alternative transcription initiation and termination to the transcriptome and proteome diversity in mammals.Systems biology approaches to understanding Epithelial Mesenchymal Transition (EMT) in mucosal remodeling and signaling in asthma Alternative splicing regulates distinct subcellular localization of Epithelial splicing regulatory protein 1 (Esrp1) isoforms.The role of mRNA splicing in prostate cancer.Muscleblind-like 1 (Mbnl1) regulates pre-mRNA alternative splicing during terminal erythropoiesis Proteomic analysis reveals CACN-1 is a component of the spliceosome in Caenorhabditis elegans.Cellular migration and invasion uncoupled: increased migration is not an inexorable consequence of epithelial-to-mesenchymal transition CD44 upregulation in E-cadherin-negative esophageal cancers results in cell invasion Identification of genetic variants associated with alternative splicing using sQTLseekeR.RNA-Seq and human complex diseases: recent accomplishments and future perspectives.Bioinformatic approaches to augment study of epithelial-to-mesenchymal transition in lung cancer Sleeping giants: emerging roles for the fat cadherins in health and disease.Mapping Splicing Quantitative Trait Loci in RNA-Seq.Variants affecting exon skipping contribute to complex traits.Human genomics. The human transcriptome across tissues and individuals.The role of epithelial plasticity in prostate cancer dissemination and treatment resistance Building robust transcriptomes with master splicing factors WNT5A inhibits metastasis and alters splicing of Cd44 in breast cancer cells.

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P2860

An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype

http://www.wikidata.org/entity/Q27339197

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

An EMT-driven alternative spli ...... d modulates cellular phenotype

@ast

An EMT-driven alternative spli ...... d modulates cellular phenotype

@en

An EMT-driven alternative spli ...... d modulates cellular phenotype

@nl

type

label

An EMT-driven alternative spli ...... d modulates cellular phenotype

@ast

An EMT-driven alternative spli ...... d modulates cellular phenotype

@en

An EMT-driven alternative spli ...... d modulates cellular phenotype

@nl

prefLabel

An EMT-driven alternative spli ...... d modulates cellular phenotype

@ast

An EMT-driven alternative spli ...... d modulates cellular phenotype

@en

An EMT-driven alternative spli ...... d modulates cellular phenotype

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JOURNAL.PGEN.1002218

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An EMT-driven alternative spli ...... d modulates cellular phenotype

@en

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Albert W Cheng

http://www.w3.org/2001/XMLSchema#string

Christopher B Burge

http://www.w3.org/2001/XMLSchema#string

Frank B Gertler

http://www.w3.org/2001/XMLSchema#string

Irina M Shapiro

http://www.w3.org/2001/XMLSchema#string

John S Condeelis

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Maja H Oktay

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Nicholas C Flytzanis

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P2860

AceView: a comprehensive cDNA-supported gene and transcripts annotation

Mapping and quantifying mammalian transcriptomes by RNA-Seq

The mammalian Scribble polarity protein regulates epithelial cell adhesion and migration through E-cadherin

ESRP1 and ESRP2 are epithelial cell-type-specific regulators of FGFR2 splicing

Failure of MBNL1-dependent post-natal splicing transitions in myotonic dystrophy

Regulation of cell-cell adhesion by the cadherin-catenin complex

An RNA code for the FOX2 splicing regulator revealed by mapping RNA-protein interactions in stem cells

The epithelial-mesenchymal transition generates cells with properties of stem cells

Mesenchyme Forkhead 1 (FOXC2) plays a key role in metastasis and is associated with aggressive basal-like breast cancers

Host microenvironment in breast cancer development: epithelial-mesenchymal transition in breast cancer development

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e1002218

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930935

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10.1371/JOURNAL.PGEN.1002218

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8

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7

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Michele Balsamo

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2011-08-18T00:00:00Z

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51604597

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21876675

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3158048

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