Endoplasmic Reticulum Stress Is a Determinant of Retrovirus-Induced Spongiform Neurodegeneration
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Cytoplasmic vacuolization in cell death and survivalHuman cytomegalovirus infection activates and regulates the unfolded protein response.West Nile Virus-Induced Neuroinflammation: Glial Infection and Capsid Protein-Mediated NeurovirulenceWest Nile Virus Infection Activates the Unfolded Protein Response, Leading to CHOP Induction and ApoptosisTumor progression and the different faces of the PERK kinaseThe ubiquitin-proteasome system in spongiform degenerative disordersEndoplasmic reticulum stress stimulates p53 expression through NF-κB activationSex-specific hippocampus-dependent cognitive deficits and increased neuronal autophagy in DEspR haploinsufficiency in miceThe SARS Coronavirus 3a protein causes endoplasmic reticulum stress and induces ligand-independent downregulation of the type 1 interferon receptor.Familial CJD associated PrP mutants within transmembrane region induced Ctm-PrP retention in ER and triggered apoptosis by ER stress in SH-SY5Y cells.Role of the host cell's unfolded protein response in arenavirus infection.Reverse genetics approaches to combat pathogenic arenavirusesGene expression profiling of microglia infected by a highly neurovirulent murine leukemia virus: implications for neuropathogenesisResistance of mRNA translation to acute endoplasmic reticulum stress-inducing agents in herpes simplex virus type 1-infected cells requires multiple virus-encoded functions.Endoplasmic reticulum stress and neurodegeneration in rats neonatally infected with borna disease virus.Modulation of the unfolded protein response by the severe acute respiratory syndrome coronavirus spike protein.Differences in scrapie-induced pathology of the retina and brain in transgenic mice that express hamster prion protein in neurons, astrocytes, or multiple cell typesOligodendrocytes are a major target of the toxicity of spongiogenic murine retroviruses.Disparate regions of envelope protein regulate syncytium formation versus spongiform encephalopathy in neurological disease induced by murine leukemia virus TR.The unfolded protein response is required for dendrite morphogenesis.Prion protein expression differences in microglia and astroglia influence scrapie-induced neurodegeneration in the retina and brain of transgenic miceThe other transmissible spongiform encephalopathies.Expression of murine leukemia virus envelope protein is sufficient for the induction of apoptosisUp-regulation of a cellular protein at the translational level by a retrovirus.Endoplasmic reticulum stress in disease pathogenesisNeurovirulence of polytropic murine retrovirus is influenced by two separate regions on opposite sides of the envelope protein receptor binding domainRole of Erk1/2 activation in prion disease pathogenesis: absence of CCR1 leads to increased Erk1/2 activation and accelerated disease progression.The degree of folding instability of the envelope protein of a neurovirulent murine retrovirus correlates with the severity of the neurological disease.Effects of acute and chronic murine norovirus infections on immune responses and recovery from Friend retrovirus infection.Mink epithelial cell killing by pathogenic murine leukemia viruses involves endoplasmic reticulum stress.Separate sequences in a murine retroviral envelope protein mediate neuropathogenesis by complementary mechanisms with differing requirements for tumor necrosis factor alpha.Inflammation in neurodegenerative diseases.Unfolded protein response in hepatitis C virus infection.Cellular unfolded protein response against viruses used in gene therapyThe expanding roles of endoplasmic reticulum stress in virus replication and pathogenesis.Splicing of Friend Murine Leukemia Virus env-mRNA Enhances Its Ability to Form Polysomes.African swine fever virus inhibits induction of the stress-induced proapoptotic transcription factor CHOP/GADD153.Activation of transcription factor Nrf-2 and its downstream targets in response to moloney murine leukemia virus ts1-induced thiol depletion and oxidative stress in astrocytes.SAT: a late NS protein of porcine parvovirus.Endoplasmic reticulum (ER) stress induced by a neurovirulent mouse retrovirus is associated with prolonged BiP binding and retention of a viral protein in the ER.
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P2860
Endoplasmic Reticulum Stress Is a Determinant of Retrovirus-Induced Spongiform Neurodegeneration
description
2003 nî lūn-bûn
@nan
2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@ast
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@en
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@nl
type
label
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@ast
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@en
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@nl
prefLabel
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@ast
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@en
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@nl
P2093
P2860
P1433
P1476
Endoplasmic Reticulum Stress I ...... d Spongiform Neurodegeneration
@en
P2093
Audrey H Baker
Cedar Johnson-Fowler
Derek E Dimcheff
John L Portis
Srdjan Askovic
P2860
P304
P356
10.1128/JVI.77.23.12617-12629.2003
P577
2003-12-01T00:00:00Z