Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways - Wikidata - thesis-toulmin - TriplyDB

Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Endometrial Stromal Decidualization Responds Reversibly to Hormone Stimulation and Withdrawal The role of decidual cells in uterine hemostasis, menstruation, inflammation, adverse pregnancy outcomes and abnormal uterine bleeding.A Novel Experimental Model of Colorectal Endometriosis.Effect of simvastatin on monocyte chemoattractant protein-1 expression in endometriosis patients: a randomized controlled trial.Low Body Mass Index in Endometriosis Is Promoted by Hepatic Metabolic Gene Dysregulation in Mice.Serum miR-451a Levels Are Significantly Elevated in Women With Endometriosis and Recapitulated in Baboons ( Papio anubis) With Experimentally-Induced Disease.Analysis of CARD10 and CARD11 somatic mutations in patients with ovarian endometriosis.IL-1β Stimulates Brain-Derived Neurotrophic Factor Production in Eutopic Endometriosis Stromal Cell Cultures: A Model for Cytokine Regulation of Neuroangiogenesis

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

http://www.wikidata.org/entity/Q28083420

description

2015 nî lūn-bûn

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2015 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2015 թվականի հուլիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Seminars in Reproductive Medicine

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Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways

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Maureen Kane

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Neil Sidell

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Robert Taylor

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P2860

Variation in the expression of cellular retinoid binding proteins in human endometrium throughout the menstrual cycle

Natural product agonists of peroxisome proliferator-activated receptor gamma (PPARγ): a review

Pathogenesis and pathophysiology of endometriosis

Association between endometriosis and risk of histological subtypes of ovarian cancer: a pooled analysis of case-control studies

Defining future directions for endometriosis research: workshop report from the 2011 World Congress of Endometriosis In Montpellier, France

Endometriosis: hormone regulation and clinical consequences of chemotaxis and apoptosis

Loss of cellular retinol-binding protein 1 gene expression in microdissected human ovarian cancer

Estrogen directly induces expression of retinoic acid biosynthetic enzymes, compartmentalized between the epithelium and underlying stromal cells in rat uterus

CD36 is a receptor for oxidized low density lipoprotein

CD36 is required for phagocytosis of apoptotic cells by human macrophages that use either a phosphatidylserine receptor or the vitronectin receptor (alpha v beta 3)

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10.1055/S-0035-1554920

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33

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