Estrogen receptor-alpha knockout mice exhibit resistance to the developmental effects of neonatal diethylstilbestrol exposure on the female reproductive tract
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WNT4 is a key regulator of normal postnatal uterine development and progesterone signaling during embryo implantation and decidualization in the mouse.Endocrine-disrupting chemicals: an Endocrine Society scientific statementNatural variability and the influence of concurrent control values on the detection and interpretation of low-dose or weak endocrine toxicities.Implantation failure: molecular mechanisms and clinical treatmentWNTs in the neonatal mouse uterus: potential regulation of endometrial gland developmentProceedings of the Summit on Environmental Challenges to Reproductive Health and Fertility: executive summaryEnvironmental factors, epigenetics, and developmental origin of reproductive disordersEstrogen suppresses uterine epithelial apoptosis by inducing birc1 expressionThe role of Wnt5a in prostate gland development.CDH1 is essential for endometrial differentiation, gland development, and adult function in the mouse uterusBranching ducts similar to mesonephric ducts or ureteric buds in teratomas originating from mouse embryonic stem cells.Effects of endocrine disrupting chemicals on expression of phospholipid hydroperoxide glutathione peroxidase mRNA in rat testes.Interaction between genetic susceptibility and early-life environmental exposure determines tumor-suppressor-gene penetrance.Physiological effects and mechanisms of action of endocrine disrupting chemicals that alter estrogen signaling.Expression of a dominant negative estrogen receptor alpha variant in transgenic mice accelerates uterine cancer induced by the potent estrogen diethylstilbestrol.Expression of estrogen receptor alpha and beta is decreased in hypospadias.Response of adult mouse uterus to early disruption of estrogen receptor-alpha signaling is influenced by Krüppel-like factor 9Estrogen receptor alpha prevents bladder cancer via INPP4B inhibited akt pathway in vitro and in vivo.Contributions of androgen and estrogen to fetal programming of ovarian dysfunction.Menstrual and reproductive characteristics of women whose mothers were exposed in utero to diethylstilbestrol (DES).Testicular dysgenesis syndrome and the estrogen hypothesis: a quantitative meta-analysis.Diethylstilboestrol exposure does not reduce testosterone production in human fetal testis xenografts.MSX2 promotes vaginal epithelial differentiation and wolffian duct regression and dampens the vaginal response to diethylstilbestrolEpigenomic reprogramming of the developing reproductive tract and disease susceptibility in adulthoodDevelopmental toxicity of estrogenic chemicals on rodents and other species.Estrogen receptor knockout mice: phenotypes in the female reproductive tract.Circulating levels of genistein in the neonate, apart from dose and route, predict future adverse female reproductive outcomesEstrogen receptor-α mediates diethylstilbestrol-induced feminization of the seminal vesicle in male miceThe relationship among HOXA10, estrogen receptor α, progesterone receptor, and progesterone receptor B proteins in rectosigmoid endometriosis: a tissue microarray studyResearch resource: whole-genome estrogen receptor α binding in mouse uterine tissue revealed by ChIP-seq.The development of cervical and vaginal adenosis as a result of diethylstilbestrol exposure in utero.Multigenerational and transgenerational effects of endocrine disrupting chemicals: A role for altered epigenetic regulation?Neonatal diethylstilbestrol exposure alters the metabolic profile of uterine epithelial cells.Developmental exposure to diethylstilbestrol alters uterine gene expression that may be associated with uterine neoplasia later in lifeRapid and estrogen receptor beta mediated actions in the hippocampus mediate some functional effects of estrogen.Fetal and early postnatal environmental exposures and reproductive health effects in the femaleRecruited mast cells in the tumor microenvironment enhance bladder cancer metastasis via modulation of ERβ/CCL2/CCR2 EMT/MMP9 signalsRequirement for estrogen receptor alpha in a mouse model for human papillomavirus-associated cervical cancer.Persistently altered epigenetic marks in the mouse uterus after neonatal estrogen exposure.Targeting estrogen/estrogen receptor alpha enhances Bacillus Calmette-Guérin efficacy in bladder cancer.
P2860
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P2860
Estrogen receptor-alpha knockout mice exhibit resistance to the developmental effects of neonatal diethylstilbestrol exposure on the female reproductive tract
description
2001 nî lūn-bûn
@nan
2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Estrogen receptor-alpha knocko ...... the female reproductive tract
@ast
Estrogen receptor-alpha knocko ...... the female reproductive tract
@en
Estrogen receptor-alpha knocko ...... the female reproductive tract
@nl
type
label
Estrogen receptor-alpha knocko ...... the female reproductive tract
@ast
Estrogen receptor-alpha knocko ...... the female reproductive tract
@en
Estrogen receptor-alpha knocko ...... the female reproductive tract
@nl
prefLabel
Estrogen receptor-alpha knocko ...... the female reproductive tract
@ast
Estrogen receptor-alpha knocko ...... the female reproductive tract
@en
Estrogen receptor-alpha knocko ...... the female reproductive tract
@nl
P2093
P3181
P356
P1476
Estrogen receptor-alpha knocko ...... the female reproductive tract
@en
P2093
P304
P3181
P356
10.1006/DBIO.2001.0413
P407
P577
2001-10-15T00:00:00Z