Block of the background K(+) channel TASK-1 contributes to arrhythmogenic effects of platelet-activating factor
about
sameAs
p11, an annexin II subunit, an auxiliary protein associated with the background K+ channel, TASK-1Regulation of two-pore-domain (K2P) potassium leak channels by the tyrosine kinase inhibitor genisteinSelective block of the human 2-P domain potassium channel, TASK-3, and the native leak potassium current, IKSO, by zincBiophysical, pharmacological, and functional characteristics of cloned and native mammalian two-pore domain K+ channelsEmerging roles for two-pore-domain potassium channels and their potential therapeutic impact.TASK-1 current is inhibited by phosphorylation during human and canine chronic atrial fibrillation.TASK1 (K(2P)3.1) K(+) channel inhibition by endothelin-1 is mediated through Rho kinase-dependent phosphorylationAbility to induce atrial fibrillation in the peri-operative period is associated with phosphorylation-dependent inhibition of TWIK protein-related acid-sensitive potassium channel 1 (TASK-1)Neuronal two-pore-domain potassium channels and their regulation by G protein-coupled receptors.PPARalpha-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy.Molecular Basis of Functional Myocardial Potassium Channel Diversity.Two-pore potassium channels in the cardiovascular system.PITX2 Modulates Atrial Membrane Potential and the Antiarrhythmic Effects of Sodium-Channel Blockers.Platelet activating factor in heart failure: potential role in disease progression and novel target for therapy.Cardiac potassium channel subtypes: new roles in repolarization and arrhythmia.The role of acid-sensitive two-pore domain potassium channels in cardiac electrophysiology: focus on arrhythmias.Novel ion channel targets in atrial fibrillation.Investigation of the role of TASK-2 channels in rat pulmonary arteries; pharmacological and functional studies following RNA interference procedures.Identification of a region in the TASK3 two pore domain potassium channel that is critical for its blockade by methanandamide.Functional and molecular identification of pH-sensitive K+ channels in murine urinary bladder smooth muscle.Functional role of TASK-1 in the heart: studies in TASK-1-deficient mice show prolonged cardiac repolarization and reduced heart rate variability.Tandem-pore domain potassium channels are functionally expressed in retinal (Müller) glial cells.K+-dependent cerebellar granule neuron apoptosis. Role of task leak K+ channels.G-CSF therapy reduces myocardial repolarization reserve in the presence of increased arteriogenesis, angiogenesis and connexin 43 expression in an experimental model of pacing-induced heart failure.
P2860
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P2860
Block of the background K(+) channel TASK-1 contributes to arrhythmogenic effects of platelet-activating factor
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Block of the background K(+) c ...... of platelet-activating factor
@ast
Block of the background K(+) c ...... of platelet-activating factor
@en
Block of the background K(+) c ...... of platelet-activating factor
@nl
type
label
Block of the background K(+) c ...... of platelet-activating factor
@ast
Block of the background K(+) c ...... of platelet-activating factor
@en
Block of the background K(+) c ...... of platelet-activating factor
@nl
prefLabel
Block of the background K(+) c ...... of platelet-activating factor
@ast
Block of the background K(+) c ...... of platelet-activating factor
@en
Block of the background K(+) c ...... of platelet-activating factor
@nl
P2093
P2860
P3181
P1476
Block of the background K(+) c ...... of platelet-activating factor
@en
P2093
Andrea Barbuti
Richard B Robinson
Satoshi Ishii
Steven J Feinmark
Takao Shimizu
P2860
P304
P3181
P356
10.1152/AJPHEART.00956.2001
P407
P577
2002-06-01T00:00:00Z