Neutrophils mediate blood-spinal cord barrier disruption in demyelinating neuroinflammatory diseases - Wikidata - thesis-toulmin - TriplyDB

Neutrophils mediate blood-spinal cord barrier disruption in demyelinating neuroinflammatory diseases

Neutrophils mediate blood-spinal cord barrier disruption in demyelinating neuroinflammatory diseases

http://www.wikidata.org/entity/Q28244425

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Pivotal roles of GM-CSF in autoimmunity and inflammation GM-CSF: An immune modulatory cytokine that can suppress autoimmunity Multifaceted interactions between adaptive immunity and the central nervous system.Myeloid cell transmigration across the CNS vasculature triggers IL-1β-driven neuroinflammation during autoimmune encephalomyelitis in mice.Sevoflurane preconditioning ameliorates neuronal deficits by inhibiting microglial MMP-9 expression after spinal cord ischemia/reperfusion in rats Neutrophil contribution to spinal cord injury and repair.Anti-inflammatory mechanisms of IFN-γ studied in experimental autoimmune encephalomyelitis reveal neutrophils as a potential target in multiple sclerosis.Infiltration of CCR2+Ly6Chigh Proinflammatory Monocytes and Neutrophils into the Central Nervous System Is Modulated by Nicotinic Acetylcholine Receptors in a Model of Multiple Sclerosis Restrained Th17 response and myeloid cell infiltration into the central nervous system by human decidua-derived mesenchymal stem cells during experimental autoimmune encephalomyelitis.Inhibition of Myeloperoxidase at the Peak of Experimental Autoimmune Encephalomyelitis Restores Blood-Brain-Barrier Integrity and Ameliorates Disease Severity.Neutrophils in animal models of autoimmune disease Peripheral Inflammatory Markers Contributing to Comorbidities in Autism.Microglia Versus Myeloid Cell Nomenclature during Brain Inflammation Plasticity of neutrophils reveals modulatory capacity.Propitious Therapeutic Modulators to Prevent Blood-Spinal Cord Barrier Disruption in Spinal Cord Injury.The contribution of neutrophils to CNS autoimmunity.Role of the JAK/STAT signaling pathway in regulation of innate immunity in neuroinflammatory diseases.The Diverse Biological Functions of Neutrophils, Beyond the Defense Against Infections.Inflammasome activation in multiple sclerosis and experimental autoimmune encephalomyelitis (EAE).Myeloperoxidase: A new player in autoimmunity.The IL-1β phenomena in neuroinflammatory diseases.New Insights into the Role of IL-1β in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis.Endocannabinod Signal Dysregulation in Autism Spectrum Disorders: A Correlation Link between Inflammatory State and Neuro-Immune Alterations.Endothelial Wnt/β-catenin signaling reduces immune cell infiltration in multiple sclerosis.IL-12Rβ2 has a protective role in relapsing-remitting experimental autoimmune encephalomyelitis.Interleukin-36γ is expressed by neutrophils and can activate microglia, but has no role in experimental autoimmune encephalomyelitis.Neuroendothelial NMDA receptors as therapeutic targets in experimental autoimmune encephalomyelitis.Gestational bisphenol-A exposure lowers the threshold for autoimmunity in a model of multiple sclerosis.Caveolin1 Is Required for Th1 Cell Infiltration, but Not Tight Junction Remodeling, at the Blood-Brain Barrier in Autoimmune Neuroinflammation.GM-CSF as a therapeutic target in autoimmune diseases.IL-1β enables CNS access to CCR2hi monocytes and the generation of pathogenic cells through GM-CSF released by CNS endothelial cells.Neutrophil infiltration to the brain is platelet-dependent, and is reversed by blockade of platelet GPIbα.Combination of cuprizone and experimental autoimmune encephalomyelitis to study inflammatory brain lesion formation and progression.Promotion of experimental autoimmune encephalomyelitis upon neutrophil granulocytes' stimulation with formyl-methionyl-leucyl-phenylalanine (fMLP) peptide.Mechanisms for lesion localization in neuromyelitis optica spectrum disorders.Matrix Metalloproteinase-8 Inhibition Prevents Disruption of Blood-Spinal Cord Barrier and Attenuates Inflammation in Rat Model of Spinal Cord Injury.Induced CNS expression of CXCL1 augments neurologic disease in a murine model of multiple sclerosis via enhanced neutrophil recruitment.Diversity of innate immune cell subsets across spatial and temporal scales in an EAE mouse model.Potential immunotherapies for traumatic brain and spinal cord injury.The Immunometabolomic Interface Receptor Hydroxycarboxylic Acid Receptor 2 Mediates the Therapeutic Effects of Dimethyl Fumarate in Autoantibody-Induced Skin Inflammation

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Neutrophils mediate blood-spinal cord barrier disruption in demyelinating neuroinflammatory diseases

http://www.wikidata.org/entity/Q28244425

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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Neutrophils mediate blood-spin ...... ing neuroinflammatory diseases

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Neutrophils mediate blood-spin ...... ing neuroinflammatory diseases

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Journal of Immunology

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Neutrophils mediate blood-spin ...... ing neuroinflammatory diseases

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Alexandre Paré

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Alexandre Prat

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Benoit Aubé

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Daniel Côté

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Hania Kébir

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Jorge I Alvarez

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Marc-André Lécuyer

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Roser Gorina

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Yves De Koninck

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2014-09-01T00:00:00Z

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