Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus - Wikidata - thesis-toulmin - TriplyDB

Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

http://www.wikidata.org/entity/Q28276729

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Cohabitation in the Intestine: Interactions among Helminth Parasites, Bacterial Microbiota, and Host Immunity Understanding the role of antibodies in murine infections with Heligmosomoides (polygyrus) bakeri: 35 years ago, now and 35 years ahead Macrophage proliferation, provenance, and plasticity in macroparasite infection Immunity to gastrointestinal nematodes: mechanisms and myths Intestinal Epithelial Cell-Intrinsic Deletion of Setd7 Identifies Role for Developmental Pathways in Immunity to Helminth Infection Extracellular Vesicles from a Helminth Parasite Suppress Macrophage Activation and Constitute an Effective Vaccine for Protective Immunity Helminths in the gastrointestinal tract as modulators of immunity and pathology.Cell-intrinsic lysosomal lipolysis is essential for alternative activation of macrophages MyD88 signaling inhibits protective immunity to the gastrointestinal helminth parasite Heligmosomoides polygyrus.Intestinal epithelial tuft cells initiate type 2 mucosal immunity to helminth parasites.Secretion of protective antigens by tissue-stage nematode larvae revealed by proteomic analysis and vaccination-induced sterile immunity.Mucosal immune responses following intestinal nematode infection Systemic impact of intestinal helminth infections.IFNγ and IL-12 Restrict Th2 Responses during Helminth/Plasmodium Co-Infection and Promote IFNγ from Th2 Cells Parasite Proximity Drives the Expansion of Regulatory T Cells in Peyer's Patches following Intestinal Helminth Infection.Migratory CD103+ dendritic cells suppress helminth-driven type 2 immunity through constitutive expression of IL-12 T-cell-intrinsic Tif1α/Trim24 regulates IL-1R expression on TH2 cells and TH2 cell-mediated airway allergy.Chronic Gastrointestinal Nematode Infection Mutes Immune Responses to Mycobacterial Infection Distal to the Gut.Novel O-linked methylated glycan antigens decorate secreted immunodominant glycoproteins from the intestinal nematode Heligmosomoides polygyrus.Interleukin-13 Receptor α1-Dependent Responses in the Intestine Are Critical to Parasite Clearance.Commensal-pathogen interactions in the intestinal tract: lactobacilli promote infection with, and are promoted by, helminth parasites.The immunology and genetics of resistance of sheep to Teladorsagia circumcincta.Trichinella spiralis immunomodulation: an interactive multifactorial process.TPL-2 restricts Ccl24-dependent immunity to Heligmosomoides polygyrus.Enteric Helminths Promote Salmonella Coinfection by Altering the Intestinal Metabolome.Immunological Functions of the Omentum.Zoonotic intestinal helminths interact with the canine immune system by modulating T cell responses and preventing dendritic cell maturation.Interactions between fibroblastic reticular cells and B cells promote mesenteric lymph node lymphangiogenesis.Toxoplasma Co-infection Prevents Th2 Differentiation and Leads to a Helminth-Specific Th1 Response.PPAR-γ promotes type 2 immune responses in allergy and nematode infection.The role of rare innate immune cells in Type 2 immune activation against parasitic helminths.Aging parasites produce offspring with poor fitness prospects.Inflammatory arthritis and systemic bone loss are attenuated by gastrointestinal helminth parasites.Primary Heligmosomoides polygyrus bakeri infection induces myeloid-derived suppressor cells that suppress CD4+ Th2 responses and promote chronic infection.Type I interferon restricts type 2 immunopathology through the regulation of group 2 innate lymphoid cells.Production and analysis of immunomodulatory excretory-secretory products from the mouse gastrointestinal nematode Heligmosomoides polygyrus bakeri.Epithelial-Cell-Derived Phospholipase A2 Group 1B Is an Endogenous Anthelmintic.Metabolic Reprogramming Mediated by the mTORC2-IRF4 Signaling Axis Is Essential for Macrophage Alternative Activation.Effects of protein malnutrition on tolerance to helminth infection.Epidermal Growth Factor Receptor Expression Licenses Type-2 Helper T Cells to Function in a T Cell Receptor-Independent Fashion.

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

http://www.wikidata.org/entity/Q28276729

description

2012 nî lūn-bûn

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2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年论文

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name

Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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type

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Seminars in Immunopathology

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Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus

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Kara J Filbey

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Lisa A Reynolds

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Rick M Maizels

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P2860

Enterocolitis and colon cancer in interleukin-10-deficient mice are associated with aberrant cytokine production and CD4(+) TH1-like responses

Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity

IL25 elicits a multipotent progenitor cell population that promotes T(H)2 cytokine responses

Constitutive ablation of dendritic cells breaks self-tolerance of CD4 T cells and results in spontaneous fatal autoimmunity

Specific microbiota direct the differentiation of IL-17-producing T-helper cells in the mucosa of the small intestine

Identification of an interleukin (IL)-25-dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion

Inhibition of autoimmune type 1 diabetes by gastrointestinal helminth infection

Trichuris suis therapy in Crohn's disease

Nematode modulation of inflammatory bowel disease

Foxp3 programs the development and function of CD4+CD25+ regulatory T cells

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1025619490

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23053394

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