Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT - Wikidata - thesis-toulmin - TriplyDB

Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

http://www.wikidata.org/entity/Q28285463

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Modulation of Potassium Channel Activity in the Balance of ROS and ATP Production by Durum Wheat Mitochondria-An Amazing Defense Tool Against Hyperosmotic Stress Role of the MPTP in conditioning the heart - translatability and mechanism Circadian influences on myocardial infarction Increased propensity for cell death in diabetic human heart is mediated by mitochondrial-dependent pathways Cross talk between mitochondria and NADPH oxidases Protein S-nitrosylation and cardioprotection KCNMA1 encoded cardiac BK channels afford protection against ischemia-reperfusion injury.Mitochondrial cyclophilin-D as a critical mediator of ischaemic preconditioning.Cardioprotective PKG-independent NO signaling at reperfusion The effect of combined glutamate receptor blockade in the NTS on the hypoxic ventilatory response in awake rats differs from the effect of individual glutamate receptor blockade.Pharmacological protection of retinal pigmented epithelial cells by sulindac involves PPAR-α.Epsilon PKC increases brain mitochondrial SIRT1 protein levels via heat shock protein 90 following ischemic preconditioning in rats.Redox regulation of the mitochondrial K(ATP) channel in cardioprotection.Mediterranean diet and cardioprotection: the role of nitrite, polyunsaturated fatty acids, and polyphenols.Protein kinase C-epsilon activation induces mitochondrial dysfunction and fragmentation in renal proximal tubules A novel class of mitochondria-targeted soft electrophiles modifies mitochondrial proteins and inhibits mitochondrial metabolism in breast cancer cells through redox mechanisms.Extracellular signalling molecules in the ischaemic/reperfused heart - druggable and translatable for cardioprotection?Mitochondrial reactive oxygen species at the heart of the matter: new therapeutic approaches for cardiovascular diseases.RETRACTED: Glycogen synthase kinase 3β transfers cytoprotective signaling through connexin 43 onto mitochondrial ATP-sensitive K+ channels.Sevoflurane Preconditioning Reduces Intestinal Ischemia-Reperfusion Injury: Role of Protein Kinase C and Mitochondrial ATP-Sensitive Potassium Channel.Biological role of connexin intercellular channels and hemichannels Levosimendan Administration in Limb Ischemia: Multicomponent Signaling Serving Kidney Protection.Roles of the nitric oxide signaling pathway in cardiac ischemic preconditioning against myocardial ischemia-reperfusion injury.Essential role of nitric oxide in acute ischemic preconditioning: S-nitros(yl)ation versus sGC/cGMP/PKG signaling?Contribution of mitochondrial oxidative stress to hypertension.Vascular endothelial cells mediate mechanical stimulation-induced enhancement of endothelin hyperalgesia via activation of P2X2/3 receptors on nociceptors.Priming adult stem cells by hypoxic pretreatments for applications in regenerative medicine.Cellular and molecular neurobiology of brain preconditioning.Mitochondrial reactive oxygen species: which ROS signals cardioprotection?Nox2 as a potential target of mitochondrial superoxide and its role in endothelial oxidative stress Role of mitochondrial oxidative stress in hypertension.Cardioprotective signaling to mitochondria Loss of cardioprotection with ageing.Ryanodine receptor phosphorylation by oxidized CaMKII contributes to the cardiotoxic effects of cardiac glycosides.Nox2-induced production of mitochondrial superoxide in angiotensin II-mediated endothelial oxidative stress and hypertension.Interaction of mitochondrial potassium channels with the permeability transition pore.Signal transduction to the permeability transition pore.Mechanism of cardioprotection by early ischemic preconditioning Physiology of potassium channels in the inner membrane of mitochondria.In the eye of the storm: mitochondrial damage during heart and brain ischaemia.

P2860

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P2860

Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

http://www.wikidata.org/entity/Q28285463

description

2008 nî lūn-bûn

@nan

2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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AJPHEART.01189.2007

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American Journal of Physiology - Heart and Circulatory Physiology

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Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

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Alexandre D T Costa

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P2860

Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon protein kinase C translocation

Preconditioning protects ischemic rabbit heart by protein kinase C activation

Mitochondrial PKC epsilon and mitochondrial ATP-sensitive K+ channel copurify and coreconstitute to form a functioning signaling module in proteoliposomes

A protein kinase C translocation inhibitor as an isozyme-selective antagonist of cardiac function

The mitochondrial permeability transition pore and its role in cell death

Enzymatic properties of the inner and outer membranes of rat liver mitochondria.

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria.

Peptide modulators of protein-protein interactions in intracellular signaling.

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

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