Recurrent epimutations activate gene body promoters in primary glioblastoma - Wikidata - thesis-toulmin - TriplyDB

Recurrent epimutations activate gene body promoters in primary glioblastoma

Recurrent epimutations activate gene body promoters in primary glioblastoma

http://www.wikidata.org/entity/Q28385236

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Next-Generation Sequencing Approaches in Cancer: Where Have They Brought Us and Where Will They Take Us?The complexity of epigenetic diseases Common features of chromatin in aging and cancer: cause or coincidence?Endogenous retroviral promoter exaptation in human cancer Relation between Established Glioma Risk Variants and DNA Methylation in the Tumor HIGD1A Regulates Oxygen Consumption, ROS Production, and AMPK Activity during Glucose Deprivation to Modulate Cell Survival and Tumor Growth Comparative DNA methylome analysis of endometrial carcinoma reveals complex and distinct deregulation of cancer promoters and enhancers.DNA Methylation and Somatic Mutations Converge on the Cell Cycle and Define Similar Evolutionary Histories in Brain Tumors Prognostic quality of activating TERT promoter mutations in glioblastoma: interaction with the rs2853669 polymorphism and patient age at diagnosis Homeobox genes gain trimethylation of histone H3 lysine 4 in glioblastoma tissue CpG location and methylation level are crucial factors for the early detection of oral squamous cell carcinoma in brushing samples using bisulfite sequencing of a 13-gene panel.ChIP-seq in studying epigenetic mechanisms of disease and promoting precision medicine: progresses and future directions.DNA methylation in adult diffuse gliomas.Cancer. The transcription factor GABP selectively binds and activates the mutant TERT promoter in cancer.An update on the epigenetics of glioblastomas.Distal CpG islands can serve as alternative promoters to transcribe genes with silenced proximal promoters.The DNA methylation landscape of human melanoma.Orphan CpG islands define a novel class of highly active enhancers.The Role of DNA Methylation in Cancer.An integrated -omics analysis of the epigenetic landscape of gene expression in human blood cells.

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Recurrent epimutations activate gene body promoters in primary glioblastoma

http://www.wikidata.org/entity/Q28385236

description

2014 nî lūn-bûn

@nan

2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Recurrent epimutations activate gene body promoters in primary glioblastoma

@ast

Recurrent epimutations activate gene body promoters in primary glioblastoma

@en

Recurrent epimutations activate gene body promoters in primary glioblastoma

@nl

type

label

Recurrent epimutations activate gene body promoters in primary glioblastoma

@ast

Recurrent epimutations activate gene body promoters in primary glioblastoma

@en

Recurrent epimutations activate gene body promoters in primary glioblastoma

@nl

prefLabel

Recurrent epimutations activate gene body promoters in primary glioblastoma

@ast

Recurrent epimutations activate gene body promoters in primary glioblastoma

@en

Recurrent epimutations activate gene body promoters in primary glioblastoma

@nl

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Genome Research

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Recurrent epimutations activate gene body promoters in primary glioblastoma

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Aaron Diaz

http://www.w3.org/2001/XMLSchema#string

Adam B Olshen

http://www.w3.org/2001/XMLSchema#string

Ashley E Graham

http://www.w3.org/2001/XMLSchema#string

Brett E Johnson

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Ivan Smirnov

http://www.w3.org/2001/XMLSchema#string

Joseph F Costello

http://www.w3.org/2001/XMLSchema#string

Pamela L Paris

http://www.w3.org/2001/XMLSchema#string

Raman P Nagarajan

http://www.w3.org/2001/XMLSchema#string

Robert J A Bell

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Shaun D Fouse

http://www.w3.org/2001/XMLSchema#string

P2860

Model-based analysis of ChIP-Seq (MACS)

An integrated encyclopedia of DNA elements in the human genome

Genome-wide association study of glioma and meta-analysis

MAGE-A tumor antigens target p53 transactivation function through histone deacetylase recruitment and confer resistance to chemotherapeutic agents

Telomerase modulates Wnt signalling by association with target gene chromatin

hEST2, the putative human telomerase catalytic subunit gene, is up-regulated in tumor cells and during immortalization

Bisulfite genomic sequencing: systematic investigation of critical experimental parameters

Identification of a CpG island methylator phenotype that defines a distinct subgroup of glioma

Genome-scale DNA methylation maps of pluripotent and differentiated cells

Variants in the CDKN2B and RTEL1 regions are associated with high-grade glioma susceptibility

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761-774

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scholarly article

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2818413

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10.1101/GR.164707.113

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5

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24

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Vasavi Sundaram

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2014-04-07T00:00:00Z

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261443651

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24709822

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4009606

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