Golgi export of the Kir2.1 channel is driven by a trafficking signal located within its tertiary structure
about
Protein assemblies of sodium and inward rectifier potassium channels control cardiac excitability and arrhythmogenesisIon channel macromolecular complexes in cardiomyocytes: roles in sudden cardiac deathAlternative splicing regulates kv3.1 polarized targeting to adjust maximal spiking frequency.Basolateral sorting of chloride channel 2 is mediated by interactions between a dileucine motif and the clathrin adaptor AP-1.Rare mutations in renal sodium and potassium transporter genes exhibit impaired transport function.Regulation of potassium channel trafficking in the distal nephron.Molecular mechanism of constitutive endocytosis of Acid-sensing ion channel 1a and its protective function in acidosis-induced neuronal death.Sorting signals that mediate traffic of chitin synthase III between the TGN/endosomes and to the plasma membrane in yeast.Potential use of potassium efflux-deficient yeast for studying trafficking signals and potassium channel functions.The B7-1 cytoplasmic tail enhances intracellular transport and mammalian cell surface display of chimeric proteins in the absence of a linear ER export motif.Expression of three topologically distinct membrane proteins elicits unique stress response pathways in the yeast Saccharomyces cerevisiaeIncompatibility between a pair of residues from the pre-M1 linker and Cys-loop blocks surface expression of the glycine receptorBasolateral sorting of the coxsackie and adenovirus receptor through interaction of a canonical YXXPhi motif with the clathrin adaptors AP-1A and AP-1BCdo Regulates Surface Expression of Kir2.1 K+ Channel in Myoblast DifferentiationRole of C-terminal membrane-proximal basic residues in cell surface trafficking of HIV coreceptor GPR15 proteinThe clathrin adaptor AP-1A mediates basolateral polarityA Common Signal Patch Drives AP-1 Protein-dependent Golgi Export of Inwardly Rectifying Potassium ChannelsESCRT regulates surface expression of the Kir2.1 potassium channel.Golgi complex-plasma membrane trafficking directed by an autonomous, tribasic Golgi export signalThe role of protein-protein interactions in the intracellular traffic of the potassium channels TASK-1 and TASK-3.Endoplasmic reticulum-associated degradation of the renal potassium channel, ROMK, leads to type II Bartter syndrome.A New Splice Variant of Large Conductance Ca2+-activated K+ (BK) Channel α Subunit Alters Human Chondrocyte Function.The CaVβ Subunit Protects the I-II Loop of the Voltage-gated Calcium Channel CaV2.2 from Proteasomal Degradation but Not Oligoubiquitination.HIV-1 Vpu Antagonizes CD317/Tetherin by Adaptor Protein-1-Mediated Exclusion from Virus Assembly SitesInhibiting the clathrin-mediated endocytosis pathway rescues K(IR)2.1 downregulation by pentamidine.A novel GTP-binding protein-adaptor protein complex responsible for export of Vangl2 from the trans Golgi networkCeapins inhibit ATF6α signaling by selectively preventing transport of ATF6α to the Golgi apparatus during ER stress.Determinants for Arabidopsis peptide transporter targeting to the tonoplast or plasma membrane.The Putative Drp1 Inhibitor mdivi-1 Is a Reversible Mitochondrial Complex I Inhibitor that Modulates Reactive Oxygen Species.Inward Rectifier Potassium Channels (Kir2.x) and Caveolin-3 Domain-Specific Interaction: Implications for Purkinje Cell-Dependent Ventricular Arrhythmias.An Overview of Protein Secretion in Yeast and Animal Cells.Andersen's syndrome mutants produce a knockdown of inwardly rectifying K+ channel in mouse skeletal muscle in vivo.A novel bipartite UNC-101/AP-1 μ1 binding signal mediates KVS-4/Kv2.1 somatodendritic distribution in Caenorhabditis elegans.Cardiac Kir2.1 and NaV1.5 Channels Traffic Together to the Sarcolemma to Control Excitability.A Novel KCNJ2 Mutation Identified in an Autistic Proband Affects the Single Channel Properties of Kir2.1.Investigating Potassium Channels in Budding Yeast: A Genetic SandboxGolgin-97 Targets Ectopically Expressed Inward Rectifying Potassium Channel, Kir2.1, to the -Golgi Network in COS-7 CellsBrugada syndrome trafficking-defective Nav1.5 channels can trap cardiac Kir2.1/2.2 channels
P2860
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P2860
Golgi export of the Kir2.1 channel is driven by a trafficking signal located within its tertiary structure
description
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2011
@ast
im Juni 2011 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2011/06/24)
@sk
vědecký článek publikovaný v roce 2011
@cs
wetenschappelijk artikel (gepubliceerd op 2011/06/24)
@nl
наукова стаття, опублікована в червні 2011
@uk
مقالة علمية (نشرت في 24-6-2011)
@ar
name
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@ast
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@en
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@nl
type
label
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@ast
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@en
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@nl
prefLabel
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@ast
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@en
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@nl
P2093
P2860
P3181
P1433
P1476
Golgi export of the Kir2.1 cha ...... within its tertiary structure
@en
P2093
Bernardo Ortega
Bo-Young Kim
Brian M. Hagen
Donghui Ma
Paul A. Welling
Tarvinder Kaur Taneja
W. Jonathan Lederer
P2860
P304
P3181
P356
10.1016/J.CELL.2011.06.007
P407
P577
2011-06-24T00:00:00Z