Primary afferent tachykinins are required to experience moderate to intense pain
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Selective inflammatory pain insensitivity in the African naked mole-rat (Heterocephalus glaber)Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolaseBreathing: rhythmicity, plasticity, chemosensitivityEffect of ricinoleic acid in acute and subchronic experimental models of inflammationFeed-forward inhibition: a novel cellular mechanism for the analgesic effect of substance P.Substance P-driven feed-forward inhibitory activity in the mammalian spinal cordTRPV1 and SP: key elements for sepsis outcome?Activation of peripheral kappa opioid receptors inhibits capsaicin-induced thermal nociception in rhesus monkeysMice Lacking Serotonin 2C Receptors Have increased Affective Responses to Aversive Stimuli.Decreased substance P and NK1 receptor immunoreactivity and function in the spinal cord dorsal horn of morphine-treated neonatal ratsThe NK1 receptor mediates both the hyperalgesia and the resistance to morphine in mice lacking noradrenalineTRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms.Substance P signaling mediates BMP-dependent heterotopic ossificationStress inhibits hair growth in mice by induction of premature catagen development and deleterious perifollicular inflammatory events via neuropeptide substance P-dependent pathwaysEvidence of a role for NK1 and CGRP receptors in mediating neurogenic vasodilatation in the mouse ear.Substance P: a pioneer amongst neuropeptides.Preferential synaptic relationships between substance P-immunoreactive boutons and neurokinin 1 receptor sites in the rat spinal cordTranscriptional and posttranslational plasticity and the generation of inflammatory painDistinct neurochemical features of acute and persistent pain.Local administration of mu or kappa opioid agonists attenuates capsaicin-induced thermal hyperalgesia via peripheral opioid receptors in rats.Pain and the pathogenesis of biceps tendinopathy.Pathobiology of visceral pain: molecular mechanisms and therapeutic implications. II. Genetic approaches to pain therapy.Neuregulin-1β prevents Ca(2+) overloading and apoptosis through PI3K/Akt activation in cultured dorsal root ganglion neurons with excitotoxicity induced by glutamate.Genetic and pharmacological disruption of neurokinin 1 receptor function decreases anxiety-related behaviors and increases serotonergic function.Tachykinin receptor modulators: novel therapeutics for rheumatoid arthritis.Increased morphine analgesia and reduced side effects in mice lacking the tac1 gene.VGLUT2 expression in primary afferent neurons is essential for normal acute pain and injury-induced heat hypersensitivity.Substance P causes seizures in neurocysticercosis.Substance P: a new era, a new role.Tissue injury regulates serotonin 1D receptor expression: implications for the control of migraine and inflammatory painCharacterization of RO4583298 as a novel potent, dual antagonist with in vivo activity at tachykinin NK₁ and NK₃ receptors.Induction of colitis causes inflammatory responses in fat depots: evidence for substance P pathways in human mesenteric preadipocytesNeurokinin-1 receptor signalling impacts bone marrow repopulation efficiency.αCGRP is essential for algesic exocytotic mobilization of TRPV1 channels in peptidergic nociceptors.Tachykinins and tachykinin receptors in bone.Bone marrow transplantation reveals an essential synergy between neuronal and hemopoietic cell neurokinin production in pulmonary inflammation.Primary afferent and spinal cord expression of gastrin-releasing peptide: message, protein, and antibody concernsDissociation of the opioid receptor mechanisms that control mechanical and heat painThe role of substance P in microvascular responses in murine joint inflammationExpression of gastrin-releasing peptide by excitatory interneurons in the mouse superficial dorsal horn.
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P2860
Primary afferent tachykinins are required to experience moderate to intense pain
description
1998 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մարտին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Nature
@fr
artículu científicu espublizáu en 1998
@ast
scientific journal article
@en
vedecký článok (publikovaný 1998/03/26)
@sk
vědecký článek publikovaný v roce 1998
@cs
wetenschappelijk artikel (gepubliceerd op 1998/03/26)
@nl
наукова стаття, опублікована в березні 1998
@uk
مقالة علمية (نشرت في 26-3-1998)
@ar
name
Primary afferent tachykinins are required to experience moderate to intense pain
@ast
Primary afferent tachykinins are required to experience moderate to intense pain
@en
Primary afferent tachykinins are required to experience moderate to intense pain
@nl
type
label
Primary afferent tachykinins are required to experience moderate to intense pain
@ast
Primary afferent tachykinins are required to experience moderate to intense pain
@en
Primary afferent tachykinins are required to experience moderate to intense pain
@nl
prefLabel
Primary afferent tachykinins are required to experience moderate to intense pain
@ast
Primary afferent tachykinins are required to experience moderate to intense pain
@en
Primary afferent tachykinins are required to experience moderate to intense pain
@nl
P2093
P2860
P3181
P356
P1433
P1476
Primary afferent tachykinins are required to experience moderate to intense pain
@en
P2093
A. I. Basbaum
A. M. Gillespie
C. J. Epstein
E. J. Carlson
P. W. Mantyh
P2860
P2888
P304
P3181
P356
10.1038/32897
P407
P577
1998-03-26T00:00:00Z
P6179
1052310285