Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice
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Molecular characterization of mutant Arabidopsis plants with reduced plasma membrane proton pump activityThe neural substrates of rapid-onset Dystonia-Parkinsonism.Mutation I810N in the alpha3 isoform of Na+,K+-ATPase causes impairments in the sodium pump and hyperexcitability in the CNS.ATP1A2 Mutations in Migraine: Seeing through the Facets of an Ion Pump onto the Neurobiology of DiseaseEffects of Age on Na(+),K(+)-ATPase Expression in Human and Rodent Skeletal MuscleInsights into the Pathology of the α3 Na(+)/K(+)-ATPase Ion Pump in Neurological Disorders; Lessons from Animal ModelsInsights into the Pathology of the α2-Na(+)/K(+)-ATPase in Neurological Disorders; Lessons from Animal Models.Cognitive deficits caused by a disease-mutation in the α3 Na(+)/K(+)-ATPase isoform.Transgenic rescue of phenotypic deficits in a mouse model of alternating hemiplegia of childhood.Alternating hemiplegia of childhood-related neural and behavioural phenotypes in Na+,K+-ATPase α3 missense mutant mice.Glutamate-system defects behind psychiatric manifestations in a familial hemiplegic migraine type 2 disease-mutation mouse model.Learning-Induced Gene Expression in the Hippocampus Reveals a Role of Neuron -Astrocyte Metabolic Coupling in Long Term MemoryAstrocytes: biology and pathologyComprehensive Application of Time-of-flight Secondary Ion Mass Spectrometry (TOF-SIMS) for Ionic Imaging and Bio-energetic Analysis of Club Drug-induced Cognitive DeficiencyDeficits in social behavioral tests in a mouse model of alternating hemiplegia of childhood.Altered glutamate protein co-expression network topology linked to spine loss in the auditory cortex of schizophrenia.Targeted mutations in the Na,K-ATPase α 2 isoform confer ouabain resistance and result in abnormal behavior in mice.Prenatal oxycodone exposure impairs spatial learning and/or memory in rats.Mouse plasmacytoma-expressed transcript 1 knock out induced 5-HT disruption results in a lack of cognitive deficits and an anxiety phenotype complicated by hypoactivity and defensiveness.The beta3 subunit of the Na+,K+-ATPase mediates variable nociceptive sensitivity in the formalin testChronic activation of corticotropin-releasing factor type 2 receptors reveals a key role for 5-HT1A receptor responsiveness in mediating behavioral and serotonergic responses to stressful challenge.Genetic reduction of the α1 subunit of Na/K-ATPase corrects multiple hippocampal phenotypes in Angelman syndrome.Efficient disruption of Zebrafish genes using a Gal4-containing gene trapIncreased susceptibility to cortical spreading depression in the mouse model of familial hemiplegic migraine type 2.Alcohol-related brain damage in humans.Quantitative proteomic analysis of mitochondria in aging PS-1 transgenic mice.Na+/K+-ATPase is present in scrapie-associated fibrils, modulates PrP misfolding in vitro and links PrP function and dysfunction.Serotonin-mediated modulation of Na+/K+ pump current in rat hippocampal CA1 pyramidal neurons.Characterization of Atp1a3 mutant mice as a model of rapid-onset dystonia with parkinsonism.Abnormal partitioning of hexokinase 1 suggests disruption of a glutamate transport protein complex in schizophreniaDistinct neurological disorders with ATP1A3 mutationsIdentification of ATP1A3 mutations by exome sequencing as the cause of alternating hemiplegia of childhood in Japanese patients.Glial-specific gene alterations associated with manic behaviorsAlternative approaches to modeling hereditary dystonias.Aberrant Purkinje cell activity is the cause of dystonia in a shRNA-based mouse model of Rapid Onset Dystonia-Parkinsonism.An α2-Na/K ATPase/α-adducin complex in astrocytes triggers non-cell autonomous neurodegenerationA specific and essential role for Na,K-ATPase α3 in neurons co-expressing α1 and α3.α3Na+/K+-ATPase deficiency causes brain ventricle dilation and abrupt embryonic motility in zebrafish.CRF enhancement of GIRK channel-mediated transmission in dopamine neurons.Reciprocal modulation of function between the D1 and D2 dopamine receptors and the Na+,K+-ATPase
P2860
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P2860
Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice
description
2007 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2007
@ast
im Januar 2007 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2007/01/17)
@sk
vědecký článek publikovaný v roce 2007
@cs
wetenschappelijk artikel (gepubliceerd op 2007/01/17)
@nl
наукова стаття, опублікована в січні 2007
@uk
مقالة علمية (نشرت في 17-1-2007)
@ar
name
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@ast
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@en
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@nl
type
label
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@ast
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@en
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@nl
prefLabel
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@ast
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@en
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@nl
P2093
P921
P3181
P1476
Deficiency in Na,K-ATPase alph ...... activity, and anxiety in mice
@en
P2093
Amy E Moseley
Charles V Vorhees
Cynthia S Bohanan
Jerry B Lingrel
Jon C Neumann
Michael M Behbehani
Tori L Schaefer
P304
P3181
P356
10.1523/JNEUROSCI.4464-06.2007
P407
P577
2007-01-01T00:00:00Z