Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
about
Characterization of mouse short-chain aldehyde reductase (SCALD), an enzyme regulated by sterol regulatory element-binding proteinsHairy-related transcription factors inhibit GATA-dependent cardiac gene expression through a signal-responsive mechanismTargeted disruption of NFATc3, but not NFATc4, reveals an intrinsic defect in calcineurin-mediated cardiac hypertrophic growthRegulation of the stability and transcriptional activity of NFATc4 by ubiquitinationCardiac regulation by phosphoinositide 3-kinases and PTENProtein kinase cascades in the regulation of cardiac hypertrophyActivation of protein synthesis in cardiomyocytes by the hypertrophic agent phenylephrine requires the activation of ERK and involves phosphorylation of tuberous sclerosis complex 2 (TSC2)miR-9 and NFATc3 regulate myocardin in cardiac hypertrophyMechanical stretch up-regulates microRNA-26a and induces human airway smooth muscle hypertrophy by suppressing glycogen synthase kinase-3βMyostatin regulates cardiomyocyte growth through modulation of Akt signalingGenetic analysis of the calcineurin pathway identifies members of the EGR gene family, specifically EGR3, as potential susceptibility candidates in schizophreniaThe GSK-3 family as therapeutic target for myocardial diseasesMolecular basis of physiological heart growth: fundamental concepts and new playersSyndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathwayStabilization of beta-catenin by a Wnt-independent mechanism regulates cardiomyocyte growthThe beta-catenin/T-cell factor/lymphocyte enhancer factor signaling pathway is required for normal and stress-induced cardiac hypertrophyBeta-catenin overexpression reduces myocardial infarct size through differential effects on cardiomyocytes and cardiac fibroblastsCardiac myosin light chain kinase is necessary for myosin regulatory light chain phosphorylation and cardiac performance in vivoHdac2 regulates the cardiac hypertrophic response by modulating Gsk3 beta activityThe pro-hypertrophic basic helix-loop-helix protein p8 is degraded by the ubiquitin/proteasome system in a protein kinase B/Akt- and glycogen synthase kinase-3-dependent manner, whereas endothelin induction of p8 mRNA and renal mesangial cell hypertMolecular role of GATA binding protein 4 (GATA-4) in hyperglycemia-induced reduction of cardiac contractilityCalcineurin initiates smooth muscle differentiation in neural crest stem cellsGSK-3alpha directly regulates beta-adrenergic signaling and the response of the heart to hemodynamic stress in miceInpp5f is a polyphosphoinositide phosphatase that regulates cardiac hypertrophic responsivenessRole of Oxidative Stress in Thyroid Hormone-Induced Cardiomyocyte Hypertrophy and Associated Cardiac Dysfunction: An Undisclosed StoryIn Vivo Phosphoproteomics Analysis Reveals the Cardiac Targets of -Adrenergic Receptor SignalingInsulin Signaling and Heart Failure.Network reconstruction and systems analysis of cardiac myocyte hypertrophy signaling.Lrrc10 is required for early heart development and function in zebrafishGlycogen synthase kinase 3 alpha and 3 beta have distinct functions during cardiogenesis of zebrafish embryo.ERβ selective agonist inhibits angiotensin-induced cardiovascular pathology in female miceCyclin D1 is a bona fide target gene of NFATc1 and is sufficient in the mediation of injury-induced vascular wall remodeling.Toward transcriptional therapies for the failing heart: chemical screens to modulate genesDirect and indirect interactions between calcineurin-NFAT and MEK1-extracellular signal-regulated kinase 1/2 signaling pathways regulate cardiac gene expression and cellular growth.Regulation of PI3-kinase/Akt signaling by muscle-enriched microRNA-486Cardiac hypertrophy is not amplified by deletion of cGMP-dependent protein kinase I in cardiomyocytes.TEAD-1 overexpression in the mouse heart promotes an age-dependent heart dysfunction.Regulation of calcineurin through transcriptional induction of the calcineurin A beta promoter in vitro and in vivo.Suppression of class I and II histone deacetylases blunts pressure-overload cardiac hypertrophy.Glycogen synthase kinase-3beta regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo
P2860
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P2860
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@ast
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@en
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@nl
type
label
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@ast
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@en
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@nl
prefLabel
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@ast
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@en
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@nl
P2093
P2860
P921
P3181
P356
P1476
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
@en
P2093
Christopher L Antos
Eric N Olson
James A Richardson
John M Shelton
John McAnally
Joseph A Hill
Norbert Frey
Timothy A McKinsey
William Kutschke
P2860
P304
P3181
P356
10.1073/PNAS.231619298
P407
P577
2002-01-08T00:00:00Z