S100B potently activates p65/c-Rel transcriptional complexes in hippocampal neurons: Clinical implications for the role of S100B in excitotoxic brain injury
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Serotonin 1A Receptors on Astrocytes as a Potential Target for the Treatment of Parkinson's DiseaseTGF-{beta}1 activates two distinct type I receptors in neurons: implications for neuronal NF-{kappa}B signalingReduced cAMP, Akt activation and p65-c-Rel dimerization: mechanisms involved in the protective effects of mGluR3 agonists in cultured astrocytes.S100B Protein, A Damage-Associated Molecular Pattern Protein in the Brain and Heart, and Beyond.S100B protein stimulates microglia migration via RAGE-dependent up-regulation of chemokine expression and releaseThrombin antithrombin complex and IL-18 serum levels in stroke patientsNF-κB in Innate Neuroprotection and Age-Related Neurodegenerative Diseases.CREB-activity and nmnat2 transcription are down-regulated prior to neurodegeneration, while NMNAT2 over-expression is neuroprotective, in a mouse model of human tauopathyThe HMGB1 receptor RAGE mediates ischemic brain damage.S100B-mediated protection against the pro-apoptotic effects of ethanol on fetal rhombencephalic neurons.S100B is increased in Parkinson's disease and ablation protects against MPTP-induced toxicity through the RAGE and TNF-α pathway.Serum S100B represents a new biomarker for mood disorders.Post-ischemic brain damage: NF-kappaB dimer heterogeneity as a molecular determinant of neuron vulnerability.Molecular biomarkers in stroke diagnosis and prognosis.Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammationSerum S100B Levels and Major Depressive Disorder: Its Characteristics and Role in Antidepressant Response.Crosstalk between calcium, amyloid beta and the receptor for advanced glycation endproducts in Alzheimer's disease.Mood disorders are glial disorders: evidence from in vivo studies.RAGE regulation and signaling in inflammation and beyond.S100A6 amyloid fibril formation is calcium-modulated and enhances superoxide dismutase-1 (SOD1) aggregation.Transcriptional regulation mechanisms of hypoxia-induced neuroglobin gene expression.S100 B: A new concept in neurocritical care.A New Technique for Collection of Cerebrospinal Fluid in Rat Pups.The S100B/RAGE Axis in Alzheimer's Disease.S100B alters neuronal survival and dendrite extension via RAGE-mediated NF-κB signaling.Receptor for advanced glycation endproducts (RAGE) deficiency protects against MPTP toxicity.Hypoxia-inducible factor-1 mediates neuronal expression of the receptor for advanced glycation end products following hypoxia/ischemia.S100B protein activates a RAGE-dependent autocrine loop in astrocytes: implications for its role in the propagation of reactive gliosis.Effect of transient cerebral ischemia on the expression of receptor for advanced glycation end products (RAGE) in the gerbil hippocampus proper.
P2860
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P2860
S100B potently activates p65/c-Rel transcriptional complexes in hippocampal neurons: Clinical implications for the role of S100B in excitotoxic brain injury
description
2004 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im Jahr 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/01/01)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/01/01)
@nl
наукова стаття, опублікована в січні 2004
@uk
مقالة علمية نشرت عام 2004 حول موضوع: دماغ
@ar
name
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@ast
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@en
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@nl
type
label
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@ast
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@en
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@nl
prefLabel
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@ast
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@en
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@nl
P2093
P1433
P1476
S100B potently activates p65/c ...... 0B in excitotoxic brain injury
@en
P2093
Hashemi SM
Rothermundt M
P304
P356
10.1016/J.NEUROSCIENCE.2004.06.013
P407
P50
P577
2004-01-01T00:00:00Z