BDNF activates mTOR to regulate GluR1 expression required for memory formation
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mTOR signaling: at the crossroads of plasticity, memory and diseaseMammalian Target of Rapamycin: Its Role in Early Neural Development and in Adult and Aged Brain FunctionStem cell guidance through the mechanistic target of rapamycinNeural and cellular mechanisms of fear and extinction memory formationGlutamatergic autoencephalitides: an emerging fieldTargeting the glutamatergic system to treat major depressive disorder: rationale and progress to dateShedding new light on neurodegenerative diseases through the mammalian target of rapamycinIn vivo AAV1 transduction with hRheb(S16H) protects hippocampal neurons by BDNF productionBDNF mediates adaptive brain and body responses to energetic challengesβ-Hydroxy-β-Methylbutyrate (HMB) Promotes Neurite Outgrowth in Neuro2a CellsA critical role for IGF-II in memory consolidation and enhancement.The proinflammatory RAGE/NF-κB pathway is involved in neuronal damage and reactive gliosis in a model of sleep apnea by intermittent hypoxiaBrain-derived neurotrophic factor activation of CaM-kinase kinase via transient receptor potential canonical channels induces the translation and synaptic incorporation of GluA1-containing calcium-permeable AMPA receptorsTargeted delivery of brain-derived neurotrophic factor for the treatment of blindness and deafness.Contextual learning requires synaptic AMPA receptor delivery in the hippocampus.Elimination of the vesicular acetylcholine transporter in the forebrain causes hyperactivity and deficits in spatial memory and long-term potentiationSustained, neuron-specific IKK/NF-κB activation generates a selective neuroinflammatory response promoting local neurodegeneration with aging.AMPA receptor antagonist NBQX attenuates later-life epileptic seizures and autistic-like social deficits following neonatal seizuresmTORC1 inhibition in the nucleus accumbens 'protects' against the expression of drug seeking and 'relapse' and is associated with reductions in GluA1 AMPAR and CAMKIIα levels.Influence of catch up growth on spatial learning and memory in a mouse model of intrauterine growth restrictionExercise and time-dependent benefits to learning and memory.Hippocampal--prefrontal BDNF and memory for fear extinctionAMPA receptor synaptic plasticity induced by psychostimulants: the past, present, and therapeutic futureBrain-derived neurotrophic factor Val66Met allele impairs basal and ketamine-stimulated synaptogenesis in prefrontal cortex.Persistent inflammation-induced up-regulation of brain-derived neurotrophic factor (BDNF) promotes synaptic delivery of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor GluA1 subunits in descending pain modulatory circuits.A simple role for BDNF in learning and memory?A positive autoregulatory BDNF feedback loop via C/EBPβ mediates hippocampal memory consolidation.The interaction between early life epilepsy and autistic-like behavioral consequences: a role for the mammalian target of rapamycin (mTOR) pathway.Role for mammalian target of rapamycin complex 1 signaling in neuroadaptations underlying alcohol-related disordersConsolidation and reconsolidation of contextual fear memory requires mammalian target of rapamycin-dependent translation in the dorsal hippocampus.Frontier of epilepsy research - mTOR signaling pathway.SynGAP regulates protein synthesis and homeostatic synaptic plasticity in developing cortical networks.HuD interacts with Bdnf mRNA and is essential for activity-induced BDNF synthesis in dendrites.Mammalian target of rapamycin: hitting the bull's-eye for neurological disorders.BDNF release is required for the behavioral actions of ketamine.Contrasting hippocampal and amygdalar expression of genes related to neural plasticity during escape from social aggressionSignaling pathways underlying the rapid antidepressant actions of ketamineBDNF contributes to both rapid and homeostatic alterations in AMPA receptor surface expression in nucleus accumbens medium spiny neurons.Neuronal Nogo-A regulates glutamate receptor subunit expression in hippocampal neurons.GABA(A) receptor blockade enhances memory consolidation by increasing hippocampal BDNF levels.
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P2860
BDNF activates mTOR to regulate GluR1 expression required for memory formation
description
2009 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունիսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique PLoS ONE
@fr
artículu científicu espublizáu en 2009
@ast
im Juni 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/06/23)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/06/23)
@nl
наукова стаття, опублікована в червні 2009
@uk
name
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@ast
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@en
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@nl
type
label
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@ast
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@en
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@nl
prefLabel
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@ast
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@en
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@nl
P2093
P2860
P50
P3181
P1433
P1476
BDNF activates mTOR to regulate GluR1 expression required for memory formation
@en
P2093
Cynthia Katche
Jorge H Medina
Leandro Slipczuk
P2860
P3181
P356
10.1371/JOURNAL.PONE.0006007
P407
P577
2009-06-23T00:00:00Z