about
Role of the C-terminal propeptide in the activity and maturation of gamma -interferon-inducible lysosomal thiol reductase (GILT).Absence of gamma-interferon-inducible lysosomal thiol reductase in melanomas disrupts T cell recognition of select immunodominant epitopesA step-by-step overview of the dynamic process of epitope selection by major histocompatibility complex class II for presentation to helper T cellsShaping T cell - B cell collaboration in the response to human immunodeficiency virus type 1 envelope glycoprotein gp120 by peptide primingRedirection of B cell responsiveness by transforming growth factor beta receptorTarget peptide sequence within infectious human immunodeficiency virus type 1 does not ensure envelope-specific T-helper cell reactivation: influences of cysteine protease and gamma interferon-induced thiol reductase activitiesHeterologous Prime-Boost HIV-1 Vaccination Regimens in Pre-Clinical and Clinical TrialsAn unexpected functional link between lysosomal thiol reductase and mitochondrial manganese superoxide dismutaseDefective cross-presentation of viral antigens in GILT-free micePatterns in the assembly of temperate forests around the Northern HemisphereCD4+ T-cell epitope prediction using antigen processing constraintsAnalysis of the Rana catesbeiana tadpole tail fin proteome and phosphoproteome during T3-induced apoptosis: identification of a novel type I keratin.Comparative quantitative mass spectrometry analysis of MHC class II-associated peptides reveals a role of GILT in formation of self-peptide repertoirePathways of antigen processing.Folding of matrix metalloproteinase-2 prevents endogenous generation of MHC class-I restricted epitopeMolecular cloning and expression analysis of the interferon-γ-inducible lysosomal thiol reductase gene from the shrimp Penaeus monodon.Gamma-interferon-inducible lysosomal thiol reductase is upregulated in human melanomaNADPH oxidase activity controls phagosomal proteolysis in macrophages through modulation of the lumenal redox environment of phagosomes.A switch in pathogenic mechanism in myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis in IFN-γ-inducible lysosomal thiol reductase-free mice.Comprehensive analysis of contributions from protein conformational stability and major histocompatibility complex class II-peptide binding affinity to CD4+ epitope immunogenicity in HIV-1 envelope glycoprotein.Distinct recognition by two subsets of T cells of an MHC class II-peptide complex.Oxidizing potential of endosomes and lysosomes limits intracellular cleavage of disulfide-based antibody-drug conjugates.Targeted delivery of antigen processing inhibitors to antigen presenting cells via mannose receptors.Burkitt lymphoma: pathogenesis and immune evasion.Genes involved in systemic and arterial bed dependent atherosclerosis--Tampere Vascular studyAbsence of gamma-interferon-inducible lysosomal thiol reductase (GILT) is associated with poor disease-free survival in breast cancer patients.Characterization of heterogeneity in the molecular pathogenesis of lupus nephritis from transcriptional profiles of laser-captured glomeruliSuppression of IFN-induced transcription underlies IFN defects generated by activated Ras/MEK in human cancer cells.γ-Interferon-inducible lysosomal thiol reductase (GILT) maintains phagosomal proteolysis in alternatively activated macrophagesMHC class II-restricted presentation of the major house dust mite allergen Der p 1 Is GILT-dependent: implications for allergic asthma.Divergent paths for the selection of immunodominant epitopes from distinct antigenic sources.A reductionist cell-free major histocompatibility complex class II antigen processing system identifies immunodominant epitopes.GILT accelerates autoimmunity to the melanoma antigen tyrosinase-related protein 1.Perspective on antigen processing and presentation.Mannose 6 phosphorylation of lysosomal enzymes controls B cell functions.Intracellular regulation of cross-presentation during dendritic cell maturation.Amphipols stabilize the Chlamydia major outer membrane protein and enhance its protective ability as a vaccineElevation of c-MYC disrupts HLA class II-mediated immune recognition of human B cell tumorsHuman BDCA2+CD123+CD56+ dendritic cells (DCs) related to blastic plasmacytoid dendritic cell neoplasm represent a unique myeloid DC subset.An exhaustion-like phenotype constrains the activity of CD4+ T cells specific for a self and melanoma antigen.
P2860
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P2860
description
2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Science
@fr
artículu científicu espublizáu en 2001
@ast
im November 2001 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2001/11/09)
@sk
vědecký článek publikovaný v roce 2001
@cs
wetenschappelijk artikel (gepubliceerd op 2001/11/09)
@nl
наукова стаття, опублікована в листопаді 2001
@uk
name
Defective antigen processing in GILT-free mice
@ast
Defective antigen processing in GILT-free mice
@en
Defective antigen processing in GILT-free mice
@nl
type
label
Defective antigen processing in GILT-free mice
@ast
Defective antigen processing in GILT-free mice
@en
Defective antigen processing in GILT-free mice
@nl
prefLabel
Defective antigen processing in GILT-free mice
@ast
Defective antigen processing in GILT-free mice
@en
Defective antigen processing in GILT-free mice
@nl
P2093
P3181
P356
P1433
P1476
Defective antigen processing in GILT-free mice
@en
P2093
B. Arunachalam
C. Alfonso
K. S. Cannon
L. Karlsson
P. Cresswell
R. A. Flavell
U. T. Phan
W. S. Garrett
P304
P3181
P356
10.1126/SCIENCE.1065500
P407
P577
2001-11-09T00:00:00Z