Stress-induced neuroinflammatory priming is time of day dependent. - Wikidata - thesis-toulmin - TriplyDB

Stress-induced neuroinflammatory priming is time of day dependent.

Stress-induced neuroinflammatory priming is time of day dependent.

http://www.wikidata.org/entity/Q30360014

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Circadian Clocks, Stress, and Immunity Interaction between circadian rhythms and stress Physical versus psychological social stress in male rats reveals distinct cardiovascular, inflammatory and behavioral consequences Parkinson's disease: Microglial/macrophage-induced immunoexcitotoxicity as a central mechanism of neurodegeneration The protective effects of resveratrol on social stress-induced cytokine release and depressive-like behavior.Chronic unpredictable stress exacerbates surgery-induced sickness behavior and neuroinflammatory responses via glucocorticoids secretion in adult rats.Modeling the Sex Differences and Inter-individual Variability in the Activity of the Hypothalamic-Pituitary-Adrenal Axis.Interactions between psychosocial stress and the circadian endogenous clock.Stress disinhibits microglia via down-regulation of CD200R: A mechanism of neuroinflammatory priming.Circadian control of pain and neuroinflammation.Diminished circadian rhythms in hippocampal microglia may contribute to age-related neuroinflammatory sensitization.Differential effects of acute versus chronic stress on ethanol sensitivity: evidence for interactions on both behavioral and neuroimmune outcomes.Neuroinflammatory priming to stress is differentially regulated in male and female rats.Putative Inflammatory Sensitive Mechanisms Underlying Risk or Resilience to Social Stress

P2860

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P2860

Stress-induced neuroinflammatory priming is time of day dependent.

http://www.wikidata.org/entity/Q30360014

description

2016 nî lūn-bûn

@nan

2016 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2016年の論文

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2016年論文

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2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

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name

Stress-induced neuroinflammatory priming is time of day dependent.

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Stress-induced neuroinflammatory priming is time of day dependent.

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type

label

Stress-induced neuroinflammatory priming is time of day dependent.

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Stress-induced neuroinflammatory priming is time of day dependent.

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prefLabel

Stress-induced neuroinflammatory priming is time of day dependent.

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Stress-induced neuroinflammatory priming is time of day dependent.

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P356

J.PSYNEUEN.2016.01.006

P1433

Psychoneuroendocrinology

P1476

Stress-induced neuroinflammatory priming is time of day dependent.

@en

P2093

Laura K Fonken

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Matthew G Frank

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Meagan M Kitt

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Michael D Weber

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Rachel A Daut

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Steven F Maier

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P2860

Circadian rhythm transcription factor CLOCK regulates the transcriptional activity of the glucocorticoid receptor by acetylating its hinge region lysine cluster: potential physiological implications

From inflammation to sickness and depression: when the immune system subjugates the brain

The permissive role of glucocorticoids in neuroinflammatory priming: mechanisms and insights

Chronic jet-lag increases mortality in aged mice.

Chronic exposure to exogenous glucocorticoids primes microglia to pro-inflammatory stimuli and induces NLRP3 mRNA in the hippocampus

Control of microglial neurotoxicity by the fractalkine receptor

Core circadian protein CLOCK is a positive regulator of NF-κB-mediated transcription

Diurnal fluctuations in HPA and neuropeptide Y-ergic systems underlie differences in vulnerability to traumatic stress responses at different zeitgeber times.

Dysregulation of inflammatory responses by chronic circadian disruption.

Influence of sleep deprivation and circadian misalignment on cortisol, inflammatory markers, and cytokine balance.

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82-90

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scholarly article

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10.1016/J.PSYNEUEN.2016.01.006

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66

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Linda R. Watkins

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2016-01-11T00:00:00Z

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290218712

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26799851

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4788538

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