The PB2-E627K mutation attenuates viruses containing the 2009 H1N1 influenza pandemic polymerase
about
Connecting the study of wild influenza with the potential for pandemic diseaseThe role of viral, host, and secondary bacterial factors in influenza pathogenesisEvaluation of the human adaptation of influenza A/H7N9 virus in PB2 protein using human and swine respiratory tract explant culturesImproving pandemic influenza risk assessment.Influenza A virus polymerase is a site for adaptive changes during experimental evolution in bat cellsAnalysis by single-gene reassortment demonstrates that the 1918 influenza virus is functionally compatible with a low-pathogenicity avian influenza virus in mice.Two years after pandemic influenza A/2009/H1N1: what have we learned?Viral factors in influenza pandemic risk assessment.Virus-specific factors associated with zoonotic and pandemic potential.PB2-588I enhances 2009 H1N1 pandemic influenza virus virulence by increasing viral replication and exacerbating PB2 inhibition of beta interferon expression.Serological and virological surveillance of avian influenza A virus H9N2 subtype in humans and poultry in Shanghai, China, between 2008 and 2010Analysis of recombinant H7N9 wild-type and mutant viruses in pigs shows that the Q226L mutation in HA is important for transmissionMutations associated with severity of the pandemic influenza A(H1N1)pdm09 in humans: a systematic review and meta-analysis of epidemiological evidence.Predicting host tropism of influenza A virus proteins using random forest.Validation of normal human bronchial epithelial cells as a model for influenza A infections in human distal tracheaA point mutation in the polymerase protein PB2 allows a reassortant H9N2 influenza isolate of wild-bird origin to replicate in human cellsInfluenza virus evolution, host adaptation, and pandemic formation.PB2 residue 158 is a pathogenic determinant of pandemic H1N1 and H5 influenza a viruses in mice.The ability of pandemic influenza virus hemagglutinins to induce lower respiratory pathology is associated with decreased surfactant protein D bindingPandemic (H1N1) 2009 virus revisited: an evolutionary retrospectiveGenomic polymorphism of the pandemic A (H1N1) influenza viruses correlates with viral replication, virulence, and pathogenicity in vitro and in vivoThe M segment of the 2009 new pandemic H1N1 influenza virus is critical for its high transmission efficiency in the guinea pig modelInsights on influenza pathogenesis from the grave.Combination of PB2 271A and SR polymorphism at positions 590/591 is critical for viral replication and virulence of swine influenza virus in cultured cells and in vivoRethinking approaches to improve the utilization of nucleic acid amplification tests for detection and characterization of influenza A in diagnostic and reference laboratories.Reconstruction of the 1918 influenza virus: unexpected rewards from the pastClinical and molecular characteristics of the 2009 pandemic influenza H1N1 infection with severe or fatal disease from 2009 to 2011 in Shenzhen, China.Asparagine substitution at PB2 residue 701 enhances the replication, pathogenicity, and transmission of the 2009 pandemic H1N1 influenza A virus.Rapid sequencing of influenza A virus vRNA, cRNA and mRNA non-coding regions.The K526R substitution in viral protein PB2 enhances the effects of E627K on influenza virus replication.Oral Delivery of a Novel Attenuated Salmonella Vaccine Expressing Influenza A Virus Proteins Protects Mice against H5N1 and H1N1 Viral InfectionNaturally occurring swine influenza A virus PB1-F2 phenotypes that contribute to superinfection with Gram-positive respiratory pathogensIdentification of Rare PB2-D701N Mutation from a Patient with Severe Influenza: Contribution of the PB2-D701N Mutation to the Pathogenicity of Human Influenza.The contribution of animal models to the understanding of the host range and virulence of influenza A viruses.Influence of PB2 host-range determinants on the intranuclear mobility of the influenza A virus polymerase.Avian influenza virus H9N2 seroprevalence and risk factors for infection in occupational poultry-exposed workers in Tai'an of China.Back to the Future: Lessons Learned From the 1918 Influenza Pandemic
P2860
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P2860
The PB2-E627K mutation attenuates viruses containing the 2009 H1N1 influenza pandemic polymerase
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@ast
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@en
type
label
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@ast
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@en
prefLabel
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@ast
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@en
P2093
P2860
P50
P356
P1433
P1476
The PB2-E627K mutation attenua ...... influenza pandemic polymerase
@en
P2093
Brett W Jagger
Genevieve L Allen
John C Kash
Ruixue Wang
Zong-Mei Sheng
P2860
P304
P356
10.1128/MBIO.00067-10
P577
2010-04-01T00:00:00Z