TLR7 recognition is dispensable for influenza virus A infection but important for the induction of hemagglutinin-specific antibodies in response to the 2009 pandemic split vaccine in mice.
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Paradoxical changes in innate immunity in aging: recent progress and new directionsImpact of prior seasonal H3N2 influenza vaccination or infection on protection and transmission of emerging variants of influenza A(H3N2)v virus in ferrets.Innate immune sensing and response to influenza.Recombinant influenza H7 hemagglutinins induce lower neutralizing antibody titers in mice than do seasonal hemagglutinins.Activation of the RIG-I pathway during influenza vaccination enhances the germinal center reaction, promotes T follicular helper cell induction, and provides a dose-sparing effect and protective immunity.Innate immunity to influenza virus infection.Acute clearance of human metapneumovirus occurs independently of natural killer cells.The Split Virus Influenza Vaccine rapidly activates immune cells through Fcγ receptors.Prolonged proinflammatory cytokine production in monocytes modulated by interleukin 10 after influenza vaccination in older adultsDysregulation of Toll-Like Receptor 7 Compromises Innate and Adaptive T Cell Responses and Host Resistance to an Attenuated West Nile Virus Infection in Old MiceHost genetics of severe influenza: from mouse Mx1 to human IRF7The battle between influenza and the innate immune response in the human respiratory tract.TLR3- and MyD88-dependent signaling differentially influences the development of West Nile virus-specific B cell responses in mice following immunization with RepliVAX WN, a single-cycle flavivirus vaccine candidateA West Nile virus NS4B-P38G mutant strain induces adaptive immunity via TLR7-MyD88-dependent and independent signaling pathwaysToll-like receptor 7 agonist imiquimod in combination with influenza vaccine expedites and augments humoral immune responses against influenza A(H1N1)pdm09 virus infection in BALB/c mice.Influenza infection in human host: challenges in making a better influenza vaccine.H1N1 vaccination in Sjögren's syndrome triggers polyclonal B cell activation and promotes autoantibody production.Effect of receptor binding specificity on the immunogenicity and protective efficacy of influenza virus A H1 vaccines.The choice of linker for conjugating R848 to inactivated influenza virus determines the stimulatory capacity for innate immune cells.Respiratory Influenza A Virus Infection Triggers Local and Systemic Natural Killer Cell Activation via Toll-Like Receptor 7.Induction and Subversion of Human Protective Immunity: Contrasting Influenza and Respiratory Syncytial Virus.Innate Immunity Induces the Accumulation of Lung Mast Cells During Influenza Infection
P2860
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P2860
TLR7 recognition is dispensable for influenza virus A infection but important for the induction of hemagglutinin-specific antibodies in response to the 2009 pandemic split vaccine in mice.
description
2012 nî lūn-bûn
@nan
2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@ast
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@en
type
label
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@ast
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@en
prefLabel
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@ast
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@en
P2093
P2860
P921
P356
P1433
P1476
TLR7 recognition is dispensabl ...... andemic split vaccine in mice.
@en
P2093
Jin Hyang Kim
Suryaprakash Sambhara
Victoria Jeisy-Scott
Weiping Cao
William G Davis
P2860
P304
10988-10998
P356
10.1128/JVI.01064-12
P577
2012-07-25T00:00:00Z