Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages.
about
Human Dendritic Cell Response Signatures Distinguish 1918, Pandemic, and Seasonal H1N1 Influenza Viruses.Proinflammatory chemokines are major mediators of exuberant immune response associated with Influenza A (H1N1) pdm09 virus infection.Systems-based approach to examine the cytokine responses in primary mouse lung macrophages infected with low pathogenic avian Influenza virus circulating in South East Asia.Prognoses of patients with acute-on-chronic hepatitis B liver failure are closely associated with altered SOCS1 mRNA expression and cytokine production following glucocorticoid treatment.Serum surfactant protein D (SP-D) is a prognostic marker of poor outcome in patients with A/H1N1 virus infection.Suppressor of cytokine signaling 1 (SOCS1) mitigates anterior uveitis and confers protection against ocular HSV-1 infectionSuppressor of cytokine signaling 4 (SOCS4) protects against severe cytokine storm and enhances viral clearance during influenza infection.The effects of H3N2 swine influenza virus infection on TLRs and RLRs signaling pathways in porcine alveolar macrophages.Galectins regulate the inflammatory response in airway epithelial cells exposed to microbial neuraminidase by modulating the expression of SOCS1 and RIG1Targeting CCL5 in inflammation.G45R on nonstructural protein 1 of influenza A virus contributes to virulence by increasing the expression of proinflammatory cytokines in mice.Antimicrobial peptides alter early immune response to influenza A virus infection in C57BL/6 mice.Orally Administered Salacia reticulata Extract Reduces H1N1 Influenza Clinical Symptoms in Murine Lung Tissues Putatively Due to Enhanced Natural Killer Cell Activity.MCPIP1 attenuates the innate immune response to influenza A viruse by suppressing RIG-I expression in lung epithelial cells.SOCS4 is dispensable for an efficient recall response to influenza despite being required for primary immunity.The Streptococcus pyogenes fibronectin/tenascin-binding protein PrtF.2 contributes to virulence in an influenza superinfection
P2860
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P2860
Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages.
description
2013 nî lūn-bûn
@nan
2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Seasonal and pandemic influenz ...... ine production in macrophages.
@ast
Seasonal and pandemic influenz ...... ine production in macrophages.
@en
type
label
Seasonal and pandemic influenz ...... ine production in macrophages.
@ast
Seasonal and pandemic influenz ...... ine production in macrophages.
@en
prefLabel
Seasonal and pandemic influenz ...... ine production in macrophages.
@ast
Seasonal and pandemic influenz ...... ine production in macrophages.
@en
P2093
P2860
P50
P1433
P1476
Seasonal and pandemic influenz ...... kine production in macrophages
@en
P2093
Benjamin T Suratt
Blanca Ortíz-Quintero
Carlos Cabello
Criselda Mendoza
Elsy Canché-Pool
Gustavo Ramírez-Martínez
Irma Aguilar-Delfín
José L Bañales
Juan Morán
Lorena Orozco
P2860
P304
P356
10.1016/J.CYTO.2013.01.018
P50
P577
2013-02-21T00:00:00Z